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the adjuvantsettingtamoxifen treatment
Patients with imatinib - resistant CMLresultednilotinib treatment
to preserve vision and relieve pain and inflammation(passive) is designedIritis treatment
a great wayto promptandrogen treatment
to provide posterior occlusal support and to minimize occlusal pressures in the anterior maxilla(passive) is designedprosthodontic treatment
ERK activation but not by SAPK / JNK activation in adrenal cancer cells(passive) can be influenced bynilotinib treatment
brain metastasespromptingcorticosteroid treatment
factors other than intractable diseases(passive) caused byCKD treatment
Fox , HS 1992preventsAndrogen treatment
Sarcina , C , Tinelli , C , Ferrario , F , Visciano , B , Pani , A , De Silvestri , A , De Simone , I , Vecchio , LD , Terraneo , V , Furiani , S , Santagostino , G , Corghi , E & Pozzi , C 2016influencesCorticosteroid treatment
to functional improvement in AD patients ... 133leadsto functional improvement in AD patients ... 133
ZIKV - induced neurodegeneration ( FigpreventedZIKV - induced neurodegeneration ( Fig
to significant decreases in scores of centrilobular necrosis , apoptosis , inflammatory cell infiltration , vacuolization , and liver necrosis ( P < 0.05may leadto significant decreases in scores of centrilobular necrosis , apoptosis , inflammatory cell infiltration , vacuolization , and liver necrosis ( P < 0.05
the increase in intraocular pressure ( IOP ) and brain damage induced by ZIKVpreventsthe increase in intraocular pressure ( IOP ) and brain damage induced by ZIKV
neurodegeneration and microgliosis induced by ZIKV infection in the cortex and hippocampus of IFN - α / βR−/− micepreventsneurodegeneration and microgliosis induced by ZIKV infection in the cortex and hippocampus of IFN - α / βR−/− mice
in significant benefitsresultedin significant benefits
to functional improvement and reduces care dependence in severely demented patients ( Winbald , 1999leadsto functional improvement and reduces care dependence in severely demented patients ( Winbald , 1999
in statistically significantly more patients improving on each of the three clusters , language , memory , and praxis ... compared with placebo , and a lower percentage of patients treated with memantine showed any worsening on any of the three clusters compared with patients receiving placeboresultedin statistically significantly more patients improving on each of the three clusters , language , memory , and praxis ... compared with placebo , and a lower percentage of patients treated with memantine showed any worsening on any of the three clusters compared with patients receiving placebo
in a statistically significant smaller loss of the number of astrocytesresultedin a statistically significant smaller loss of the number of astrocytes
in significant improvement in hot flashesresultedin significant improvement in hot flashes
stabilization of the disease progress and reduced a risk of stroke and transitory ischemic attacks in the disease courseresultedstabilization of the disease progress and reduced a risk of stroke and transitory ischemic attacks in the disease course
to a more interactive and dignified life for patients with moderate - to - severe ADmay leadto a more interactive and dignified life for patients with moderate - to - severe AD
deviations from this normal pattern of fine movements whereby there was a suppression of fine movements at 10 min after treatment in animals treated with the three highest doses of memantine ( 5 , 10 , and 20 mg / kg ) compared with saline controlscauseddeviations from this normal pattern of fine movements whereby there was a suppression of fine movements at 10 min after treatment in animals treated with the three highest doses of memantine ( 5 , 10 , and 20 mg / kg ) compared with saline controls
in significant clinical improvement , stabilization of the disease progressresultedin significant clinical improvement , stabilization of the disease progress
apoptotic cell deathpreventedapoptotic cell death
the CMS - induced increase in corticotropin - releasing factor ( CRF ) mRNA levels in the dBNSTpreventsthe CMS - induced increase in corticotropin - releasing factor ( CRF ) mRNA levels in the dBNST
decreases in the levels of NR1causeddecreases in the levels of NR1
to improve microcirculation and eliminate toxinsdesignedto improve microcirculation and eliminate toxins
in improvements among patient subgroups defined by HIV RNA concentration in the CSF or plasma at baselinemay resultin improvements among patient subgroups defined by HIV RNA concentration in the CSF or plasma at baseline
Stress - Induced Change in Hippocampal Volume(passive) Is Prevented byStress - Induced Change in Hippocampal Volume
in significant improvements in cardiac output and stroke volumeresultedin significant improvements in cardiac output and stroke volume
a 13.8 % increase in running distance of the atorvastatin - treated mice at 16 wk of agecauseda 13.8 % increase in running distance of the atorvastatin - treated mice at 16 wk of age
the increase in nicotine self - administration shown by controlspreventedthe increase in nicotine self - administration shown by controls
neuronal necrosis induced by excitotoxicity in Alzheimer ’s disease , but fails to reduce apoptosis from chronically elevated levels of oxidative stress [ 116can preventneuronal necrosis induced by excitotoxicity in Alzheimer ’s disease , but fails to reduce apoptosis from chronically elevated levels of oxidative stress [ 116
in a significant reduction in cortical levels of soluble Aβ1 - 42 peptide ( Figresultedin a significant reduction in cortical levels of soluble Aβ1 - 42 peptide ( Fig
the suppression of neurogenesis caused by psychosocial stress , a process in which excitatory amino acids are known to mediate suppression ( 9preventedthe suppression of neurogenesis caused by psychosocial stress , a process in which excitatory amino acids are known to mediate suppression ( 9
neuronal cell death induced by ZIKV infection without interfering with the ability of the virus to replicate in the hostpreventsneuronal cell death induced by ZIKV infection without interfering with the ability of the virus to replicate in the host
the stress - induced impairment of radial maze learningalso preventedthe stress - induced impairment of radial maze learning
an alteration in cellular substrate metabolism with statins(passive) may be influenced byan alteration in cellular substrate metabolism with statins
in decreased plaque area and number in hippocampus and in overlying cortex immunostained with an Ab42-specific antibodyresultedin decreased plaque area and number in hippocampus and in overlying cortex immunostained with an Ab42-specific antibody
in decreased plaque area and number in hippocampus and in overlying cortex immunostained with an Aβ42 -specific antibodyresultedin decreased plaque area and number in hippocampus and in overlying cortex immunostained with an Aβ42 -specific antibody
Stress - Induced Reduction in Cell Proliferation(passive) Is Prevented byStress - Induced Reduction in Cell Proliferation
hypoalgesia in male db−/db− micepreventshypoalgesia in male db−/db− mice
The antioxidant effect(passive) caused byThe antioxidant effect
significant reductions in the duration of headaches as well as symptoms like nausea and vomitingalso createdsignificant reductions in the duration of headaches as well as symptoms like nausea and vomiting
mitochondrial function and physical performance of statin - treated micecould influencemitochondrial function and physical performance of statin - treated mice
Mitochondrial uncoupling in hyperglycaemic db / db mice(passive) was prevented byMitochondrial uncoupling in hyperglycaemic db / db mice
decreases between 11 and 17 mm Hg and diastolic blood pressure decreasesresulteddecreases between 11 and 17 mm Hg and diastolic blood pressure decreases
inflammatory cells infiltration and reduced fibrosis in ISO administered ratspreventedinflammatory cells infiltration and reduced fibrosis in ISO administered rats
in the extensive coagulative necrosis and fibrinoid vasculitis that surrounds the parenchymal infarct seen in figure 1C. Dresultedin the extensive coagulative necrosis and fibrinoid vasculitis that surrounds the parenchymal infarct seen in figure 1C. D