As a result , replacement fluid is necessaryto preventvolume depletion
that thiazide diuretics could be harmfulcausevolume depletion
diarrhoea , vomiting or excessive bleeding(passive) caused byvolume depletion
The blood cells drawn from the animals ... re - infusedto preventvolume depletion
bleeding , gastrointestinal disease such as vomiting , diarrhea or bleeding , renal losses due to diuretics or glucose osmotic diuresis , skin or respiratory losses from insensible losses , sweat or burns , and third space sequestration due to crush injury or skeletal fracture(passive) is caused byIntravascular volume depletion
gastrointestinal losses from watery diarrhea and not renal losses(passive) was caused byvolume depletion
Forward Time Center Space is first - order accurate , although thia- zide diuretics can reduce lithium - induced polyuriacan ... causeintravascular volume depletion
severe exercise , nausea and vomiting , diuretics , adrenal insufficiency , and extensive burns(passive) may be caused byvolume depletion
blood loss or focus of a national inquiry in Great Britain after the other fluid shifts(passive) caused byvolume depletion
by giving the animals a drinking solution containing 0.8 % NaCl and 0.1 % KCl(passive) was preventedVolume depletion
When diuretics are initiated for an acute exacerbation of heart failure , the goal of therapy should be a maximum net loss of 0.5 - 1.0 L of fluid / day ( 0.5 - 1.0 kg body weightto preventintravascular volume depletion
due to penetration of osmotically active particles into the brain , their accumulation creating an osmotic gradient favouring movement of water into the brain , and oedema formation rapid volume depletion stemming from overzealous administration of osmotic diuretics overly rapid administration of hypotonic fluids to patients after a prolonged period of hyperosmolar in the former casesetsvolume depletion
Increase fluid intake , if feasibleto preventvolume depletion
loss of pharmacology(passive) can be caused byVolume depletion Volume depletion
then being givento preventvolume depletion
The leakage of fluid in the lungscausesvolume depletion
chronic vomiting(passive) could be caused byVolume depletion
the false hypervolemiacausingvolume depletion
a lack of Oxygen rather then the excess CO2(passive) is caused byvolume " - asphyxia
acute renal failure in patients taking ACE inhibitorscausingacute renal failure in patients taking ACE inhibitors
to dehydration , hypovolemia , hypotension and pre - renal failurecan contributeto dehydration , hypovolemia , hypotension and pre - renal failure
the vasomotor nephropathy in this disease has been postulated to occur from capillary leakage with associated loss of fluid and protein , hence the proteinuria [ 10causingthe vasomotor nephropathy in this disease has been postulated to occur from capillary leakage with associated loss of fluid and protein , hence the proteinuria [ 10
alsocan ... contributealso
from poor fluid intake , fever , and GI disturbances orcan resultfrom poor fluid intake , fever , and GI disturbances or
to failure in other sytems.22 Acute renal failure as a consequence of surgery and anaesthesiacan contributeto failure in other sytems.22 Acute renal failure as a consequence of surgery and anaesthesia
to increased aminoglycoside concentration within the nephron tobramycin inhl and furosemide oral tobramycin inhlleadingto increased aminoglycoside concentration within the nephron tobramycin inhl and furosemide oral tobramycin inhl
inadequate systemic per - fusion leads to cholestatic jaundicecausinginadequate systemic per - fusion leads to cholestatic jaundice
to dry mucous membranes , tachycardia , and hypotensioncan leadto dry mucous membranes , tachycardia , and hypotension
to poor organ perfusion and ultimately renal failure and multiorgan failureleadingto poor organ perfusion and ultimately renal failure and multiorgan failure
in hypotension ( mildresultingin hypotension ( mild
hypotension and hypoperfusioncan causehypotension and hypoperfusion
in hypotension contraindicatorsresultingin hypotension contraindicators
to non osmotic release of ADHleadingto non osmotic release of ADH
to pre - renal AKI 3leadingto pre - renal AKI 3
a further fall in blood pressure and cardiac outputcausesa further fall in blood pressure and cardiac output
to metabolic alkalosisleadingto metabolic alkalosis
correction of alkalosispreventscorrection of alkalosis
to increased aminoglycoside concentration within the nephron tobramycin ophtleadingto increased aminoglycoside concentration within the nephron tobramycin opht
to hypotension , lightheadedness , and syncopecan leadto hypotension , lightheadedness , and syncope
to multiorgan dysfunction and deathleadingto multiorgan dysfunction and death
secondary hyperaldosteronism , which further complicates their evaluation and treatmentcausessecondary hyperaldosteronism , which further complicates their evaluation and treatment
to acute kidney injury ... thus resulting in permanent damage.7,8could leadto acute kidney injury ... thus resulting in permanent damage.7,8
to electrolyte imbalance ( see section 4.4may leadto electrolyte imbalance ( see section 4.4
scarring or destruction of the patientwill causescarring or destruction of the patient
further increases in aldosterone secretioncausesfurther increases in aldosterone secretion
in a high aldosterone ststeresultsin a high aldosterone stste
to renal vasoconstriction and failurealso leadsto renal vasoconstriction and failure
from gastrointestinal disease or shockresultingfrom gastrointestinal disease or shock
from shock or gastrointestinal diseaseresultingfrom shock or gastrointestinal disease