progression of the tumor(passive) caused byright ventricular outflow tract obstruction
Double - Chambered Right Ventricle Presenting in Adulthood John Kokotsakis , Efthymia G. Rouska , Leanne Harling , Hutan Ashrafian , Vania Anagnostakou , Christos Charitos(passive) Caused byRight Ventricular Outflow Tract Obstruction
prior DVT ( deep vein thrombosis(passive) can also be caused byVenous obstruction
extrinsic compression of the left common iliac vein ( LCIV ) by the overlying pulsatile right common iliac artery against lumbar vertebrae(passive) caused byvenous outflow obstruction
retained native leaflets following mitral valve replacement using medical treatment ( 14 August , 2006(passive) caused byventricular outflow tract obstruction
retained native leaflets following mitral valve replacement using medical treatment | Heart Resolution of dynamic(passive) caused byventricular outflow tract obstruction
Catecholamine in Acute Myocardial Infarction(passive) Provoked byVentricular Outflow Tract Obstruction
deep vein thrombosis or arterio - venous fistulae(passive) caused byvenous outflow obstruction
Available from : http://www.afrjpaedsurg.org/text.asp?2013/10/2/199/115057 Double - chambered right ventricle ( DCRVcausingright ventricular outflow tract obstruction
Hepatic vein thrombosis ( HVT ) , also known as Budd - Chiari syndromecauseshepatic venous outflow obstruction
the obstructive tissueis causingvenous obstruction
systolic anterior motion ( SAM ) of the mitral valve(passive) is caused bySubaortic outflow obstruction
systolic anterior motion of the mitral valve leaflets(passive) caused byventricular outflow tract obstruction
lt is curvedto preventaortic outflow tract obstruction
intracranial pressurecausingobstruction to venous outflow
a retrobulbar tumor(passive) may be caused byVenous obstruction
Cheng L. Giant myxomacausingright ventricular outflow tract obstruction
from compression of the iliac vein by the iliac artery of the left lower extremity ( May - Thurner syndromeresultingfrom compression of the iliac vein by the iliac artery of the left lower extremity ( May - Thurner syndrome
from insertion of mitral prostheses leaving the native leaflets intact adverse clinical outcome in seven patientsresultingfrom insertion of mitral prostheses leaving the native leaflets intact adverse clinical outcome in seven patients
to a cyclical cascade of edema and further cerebral venous obstructionleadingto a cyclical cascade of edema and further cerebral venous obstruction
authors Ahmed Eldorry BACKGROUND & AIM Budd - Chiari syndrome ( BCS(passive) is caused byauthors Ahmed Eldorry BACKGROUND & AIM Budd - Chiari syndrome ( BCS
life - threatening situations ( i.e. , cerebral or laryngeal edemahas causedlife - threatening situations ( i.e. , cerebral or laryngeal edema
proptosis , ocular chemosis , and infectioncan causeproptosis , ocular chemosis , and infection
to increased intracranial pressure What is the treatment for mastoiditisleadingto increased intracranial pressure What is the treatment for mastoiditis
during dobutamine stress echocardiographyprovokedduring dobutamine stress echocardiography
decreases in blood perfusion(passive) caused bydecreases in blood perfusion
buy viagra melbourne australia employment hypoperfusion due to stagnation of CBFmay resultbuy viagra melbourne australia employment hypoperfusion due to stagnation of CBF
in Budd - Chiari syndrome , renal dysfunction or lower extremity edemaresultingin Budd - Chiari syndrome , renal dysfunction or lower extremity edema
to dilation of the epidural veins and the development of abdominal wall and retroperitoneal collateralsleadsto dilation of the epidural veins and the development of abdominal wall and retroperitoneal collaterals
to myocardial hypertrophy and potentially heart failureleadsto myocardial hypertrophy and potentially heart failure
in ischemia ( Budd Chiariresultingin ischemia ( Budd Chiari
in local venous hypertensionresultsin local venous hypertension
stagnation of blood flow Venous hypertension , which may be localcausingstagnation of blood flow Venous hypertension , which may be local
to stagnant cerebral hypoxia or arterial occlusion leading to brain ischemialeadingto stagnant cerebral hypoxia or arterial occlusion leading to brain ischemia
from cardiac or pericardial disorders causing passive congestion of the caudal vena cava ( eg , right heart failure , pericardial disease , congenital defects , cardiac tumorscan resultfrom cardiac or pericardial disorders causing passive congestion of the caudal vena cava ( eg , right heart failure , pericardial disease , congenital defects , cardiac tumors
from cardiac or pericardial disorders causing passive congestion of the caudal vena cava ( eg , right heart failure , pericardial disease causing tamponade , congenital defects [ cor triatriatum dexter ] , cardiac tumorscan resultfrom cardiac or pericardial disorders causing passive congestion of the caudal vena cava ( eg , right heart failure , pericardial disease causing tamponade , congenital defects [ cor triatriatum dexter ] , cardiac tumors
hydrocephalus , optic chiasm compression resulting in visual dysfunction or hypothalamic / pituitary compression leading to endocrinopathycausinghydrocephalus , optic chiasm compression resulting in visual dysfunction or hypothalamic / pituitary compression leading to endocrinopathy
preeclampsia ( pregnancy - induced hypertension ) , a disease that kills 50,000 women and 250,000 children worldwide every yearcausespreeclampsia ( pregnancy - induced hypertension ) , a disease that kills 50,000 women and 250,000 children worldwide every year
in further venous hypertension , which then decreases CSF absorption and causes further increases in intracranial pressure , which then feeds back causing further external compression of the transverse sinus and further stenosis.25resultsin further venous hypertension , which then decreases CSF absorption and causes further increases in intracranial pressure , which then feeds back causing further external compression of the transverse sinus and further stenosis.25
in further venous hypertension , which then decreases CSF absorption and causes further increases in ICP , which then feeds back causing further external compression of the transverse sinus and further stenosis ( Fig . 2resultsin further venous hypertension , which then decreases CSF absorption and causes further increases in ICP , which then feeds back causing further external compression of the transverse sinus and further stenosis ( Fig . 2
from intrinsic causes ( ie , thrombus or infectious vegetation ) or external causes ( ie , kinking of graft , compression after sternal closure , excessive suturing at the anastomotic site or compression from mediastinal hemorrhage or tumor).6,19 Depending on the etiologycan resultfrom intrinsic causes ( ie , thrombus or infectious vegetation ) or external causes ( ie , kinking of graft , compression after sternal closure , excessive suturing at the anastomotic site or compression from mediastinal hemorrhage or tumor).6,19 Depending on the etiology
to wall ischemia à bacterial invasion of the appendiceal wall and subsequent extravasation of bacteria à “ perforation ” 5X higher rate of perforated appendicitis if 48 hour time of symptoms c / w less than 24 hours of symptoms Complex / Complicated appendicitisleadsto wall ischemia à bacterial invasion of the appendiceal wall and subsequent extravasation of bacteria à “ perforation ” 5X higher rate of perforated appendicitis if 48 hour time of symptoms c / w less than 24 hours of symptoms Complex / Complicated appendicitis
in an inability of blood to exit the leg and go back to the heartresultsin an inability of blood to exit the leg and go back to the heart
in infarction or hemorrhageresultingin infarction or hemorrhage
to both rupture of venules and to hemorrhagic transformation of venous infarctionsleadingto both rupture of venules and to hemorrhagic transformation of venous infarctions
venous disorderscan ... causevenous disorders
to serious obstruction of the left ventricular ejection functionwill ... leadto serious obstruction of the left ventricular ejection function
from thrombosis or extrinsic pressure by tumors , or occupations that require prolonged standingresultingfrom thrombosis or extrinsic pressure by tumors , or occupations that require prolonged standing
several paths of collateral circulation to form , resulting in secondary varicosities , which , when reaching the finer venous vessels , results in open arteriovenous channels and formation of arteriovenous fistulae ( Figure 2A and 2Bcausesseveral paths of collateral circulation to form , resulting in secondary varicosities , which , when reaching the finer venous vessels , results in open arteriovenous channels and formation of arteriovenous fistulae ( Figure 2A and 2B