PMID : 27576135 the chaperone 78-kDa glucose - regulated protein ( GRP78 ) protects the MPD against PDI - dependent disulfide - bond isomerization by binding to this domain and , therebypreventingADAM17 inhibition
pressure on the carotid sinus(passive) created byvagal inhibition
developmental lead exposure in the dentate gyrus of rat Chemical Abstract(passive) caused byneurogenesis inhibition
/ FREE Full Text23. ↵ Herrmann J , Saguner AM , Versari D , Peterson TE , Chade A , Olson M , Lerman LO , Lerman A. Chroniccontributesproteasome inhibition
The qnr genes encode proteins that prevent fluoroquinolones from interacting with DNA / type - II - topoisomerase complexes formed during DNA replicationthus preventingfluoroquinolone inhibition
developmental lead exposure in the dentate gyrus of rat | SpringerLink November 2014(passive) caused byneurogenesis inhibition
Science Spermpreventscalcineurin inhibition
a commercial mixture of cellulases from T. reesei and excess of β - glucosidaseto preventcellobiose inhibition
Perry , DK , Burns , JM , Pollinger , HS , Amiot , BP , Gloor , JM , Gores , GJ & Stegall , MD 2009causesProteasome inhibition
| Molecular Neurodegeneration | Full Text Histoneleadsdeacetylase inhibition
In particular , the gold ( III ) compound AuL12 was reportedto causeproteasome inhibition
retrograde signal from metabotropic glutamate to cannabinoid receptors , NEURON , 31(3 ) , 2001 , pp(passive) caused byPresynaptic inhibition
Publisher Site | Google Scholar M. Lorenz , N. Wilck , S. Meiners et al .preventsProteasome inhibition
Publisher Site | Google Scholar R. Gordon , E. A. Albornoz , D. C. Christie et al .preventsInflammasome inhibition
Publisher Site | Google Scholar D. K. Perry , J. M. Burns , H. S. Pollinger et al .causesProteasome inhibition
43 2000 Pertussis toxin ... Ribeiro , J. A. article openAccess 44 2008 Potential therapeutic interest of adenosine A2A receptors in psychiatric disorders Cunha , Rodrigo A. ; Ferré , Sergi ; Vaugeois , Jean - Marie ; Chen , Jiang - Fan article openAccess 45 2014preventspresynaptic inhibition
Janprevents31.PARP-1 inhibition
GWI Tyrosine ki-preventsnase inhibition
This steppreventsqPCR inhibition
This EE pathwaypreventedLTP inhibition
The tonic current through these channelscan causeshunting inhibition
UVC irradiationtriggerstranscriptional inhibition
Thelitz , S , Oishi , P , Sanchez , LS , Bekker , JM , Ovadia , B , Johengen , MJ , Black , SM & Fineman , JR 2004preventsPhosphodiesterase-3 inhibition
By selectively exciting A - beta nerve fibres in the skin with TENS , the amount of painful stimulation being transmitted by smaller diameter A - delta and C - fibres can be reducedcausingsegmental inhibition
Number to send5102050100200Start from citationCreate File Select itemprevents236902291.Calpain inhibition
another drugcausespresynaptic inhibition
basal PGE2 ... thosecausedTxB2 inhibition
only enough pressureto triggerautogenic inhibition
Granic , I , Nyakas , C , Luiten , PGM , Eisel , ULM , Halmy , LG , Gross , G , Schoemaker , H , Möller , A & Nimmrich , V 2010preventsCalpain inhibition
release of an inhibitory substance onto the outsides of the presy - naptic nerve fibrils before their own endings terminate on the postsynaptic neuron(passive) is caused byPresynaptic inhibition
Long - term systemic vascular endothelial growth factorcan resultVEGF ) inhibition
any nerve or mobility restrictionsmay be causingneurogenic inhibition
author = { Malmgren , Siri and Ahrén , Bo } , pages = { 1091 - -1099 } , titlecontributesDPP-4 inhibition
The PAR-1 inhibitor SCH79797 and PAR-1 antibodypreventedTRPV5 inhibition
exactly howpreventsmTOR inhibition
The potential physiological benefits ofleadDGAT-1 inhibition
blockade of sodium channelscausespresynaptic inhibition
Spontaneous dopamine release in the presence of cocainecausedpresynaptic inhibition
orderto preventqPCR inhibition
α - synuclein pathology and dopaminergic neurodegeneration in mice ... ” Science Translational Medicine ( 2018preventsα - synuclein pathology and dopaminergic neurodegeneration in mice ... ” Science Translational Medicine ( 2018
α - synuclein pathology and dopaminergic neurodegeneration in mice Low - fouling fluoropolymerspreventsα - synuclein pathology and dopaminergic neurodegeneration in mice Low - fouling fluoropolymers
alpha - synuclein pathology and dopaminergic neurodegeneration in micepreventsalpha - synuclein pathology and dopaminergic neurodegeneration in mice
to antitumor activity in ovarian cancer Abstract # 3582leadsto antitumor activity in ovarian cancer Abstract # 3582
to Antitumor Activity in Ovarian Cancer Vivas - MejiaLeadsto Antitumor Activity in Ovarian Cancer Vivas - Mejia
to a reduced spine density [ 36ledto a reduced spine density [ 36
to a failure of mitotic division during the first mitotic division in pig embryos in JOURNAL OF ASSISTED REPRODUCTION AND GENETICS MESHleadsto a failure of mitotic division during the first mitotic division in pig embryos in JOURNAL OF ASSISTED REPRODUCTION AND GENETICS MESH
that ibrutinib enhances NAD+ and ATP depletion(passive) triggered bythat ibrutinib enhances NAD+ and ATP depletion
sudden deathcausessudden death
to a cessation of mRNA export ( Tokunaga et al . , 2006leadsto a cessation of mRNA export ( Tokunaga et al . , 2006
α - synuclein pathology and dopaminergic neurodegeneration in mice Authors : Gordon R , Albornoz EA , Christie DC , Langley MR , Kumar V , Mantovani S , Robertson AAB , Butler MS , Rowe DB , O’Neill LA , Kanthasamy AG , Schroder K , Cooper MA , Woodruff TMpreventsα - synuclein pathology and dopaminergic neurodegeneration in mice Authors : Gordon R , Albornoz EA , Christie DC , Langley MR , Kumar V , Mantovani S , Robertson AAB , Butler MS , Rowe DB , O’Neill LA , Kanthasamy AG , Schroder K , Cooper MA , Woodruff TM
to Antitumor Activity in Ovarian CancerPoster SessionLeadsto Antitumor Activity in Ovarian CancerPoster Session
to cell death in MM cell lines and patient myeloma samplesleadsto cell death in MM cell lines and patient myeloma samples
to decreased methylation and increased synaptic activity.[2leadsto decreased methylation and increased synaptic activity.[2
α - synuclein pathology and dopaminergic neurodegeneration in mice — Macquarie University Richard Gordon , Eduardo A. Albornoz , Daniel C. Christie , Monica R. Langley , Vinod Kumar , Susanna Mantovani , Avril A. B. Robertson , Mark S. Butler , Dominic B. Rowe , Luke A. O’Neill , Anumantha G. Kanthasamy , Kate Schroder , Matthew A. Cooperpreventsα - synuclein pathology and dopaminergic neurodegeneration in mice — Macquarie University Richard Gordon , Eduardo A. Albornoz , Daniel C. Christie , Monica R. Langley , Vinod Kumar , Susanna Mantovani , Avril A. B. Robertson , Mark S. Butler , Dominic B. Rowe , Luke A. O’Neill , Anumantha G. Kanthasamy , Kate Schroder , Matthew A. Cooper
accumulation of GCP - WD and γ - tubulin at centrosomes ... but also reduces their spindle localizationpreventsaccumulation of GCP - WD and γ - tubulin at centrosomes ... but also reduces their spindle localization
G2 / M arrest , which is the cell cycle phase that is known to be more sensitive to ionizing radiationcausesG2 / M arrest , which is the cell cycle phase that is known to be more sensitive to ionizing radiation
rapid death from cardiac arrest(passive) caused byrapid death from cardiac arrest
metabolic dysfunction and photoreceptor deathcausesmetabolic dysfunction and photoreceptor death
to defective polyploidization and cell death in megakaryocytes ... a result with implications in the design of therapeutic protocols targeting this mitotic kinaseleadsto defective polyploidization and cell death in megakaryocytes ... a result with implications in the design of therapeutic protocols targeting this mitotic kinase
to pharmacodynamic pHisH3 modulation and significant antitumor activity in multiple DLBCL modelsleadsto pharmacodynamic pHisH3 modulation and significant antitumor activity in multiple DLBCL models
the degradation of a number of substrates , some of which may also impact myocardial signaling ( reviewed extensively elsewhere 4,9preventsthe degradation of a number of substrates , some of which may also impact myocardial signaling ( reviewed extensively elsewhere 4,9
in apoptosis of early progenitors and maturation of differentiated neutrophilsresultsin apoptosis of early progenitors and maturation of differentiated neutrophils
the NAD formation mechanism during ES - induced muscle contractioninfluencedthe NAD formation mechanism during ES - induced muscle contraction
apoptosis and suppressed the growth of PDK1- and MYC - driven xenograft tumorstriggeredapoptosis and suppressed the growth of PDK1- and MYC - driven xenograft tumors
to alteredexpression of NF - κB - dependent genes resulting in reduced apoptosisleadsto alteredexpression of NF - κB - dependent genes resulting in reduced apoptosis
to induction of differentiation , cell cycle arrest and apoptosis in AML cellsleadsto induction of differentiation , cell cycle arrest and apoptosis in AML cells
to sensitize TRAIL - induced apoptosis in human lung cancer H1299 cellscontributesto sensitize TRAIL - induced apoptosis in human lung cancer H1299 cells
AKT1 / FOXO3 / NOXA – dependent apoptosis in both EBV - positive and -negative lymphoma cellstriggeredAKT1 / FOXO3 / NOXA – dependent apoptosis in both EBV - positive and -negative lymphoma cells
/ delay enzalutamide resistance and restore AR function , leading to reductions in tumor burdencan prevent/ delay enzalutamide resistance and restore AR function , leading to reductions in tumor burden
to increased levels of ROS and apoptosis in cells exposed to bronchoalveolar lavage fluids from infected animalsalso ledto increased levels of ROS and apoptosis in cells exposed to bronchoalveolar lavage fluids from infected animals
AKT1 / FOXO3 / NOXA – dependent apoptosis in EBV - positive and -negative Burkitt 's lymphoma cellstriggeredAKT1 / FOXO3 / NOXA – dependent apoptosis in EBV - positive and -negative Burkitt 's lymphoma cells
Epstein - Barr virus lytic cycle and apoptosis in immortalized and transformed B cells : a basis for new therapiestriggersEpstein - Barr virus lytic cycle and apoptosis in immortalized and transformed B cells : a basis for new therapies
in a reduction in CV events as captured by the primary end point,5 and the mechanism of benefit is the subject of speculationresultedin a reduction in CV events as captured by the primary end point,5 and the mechanism of benefit is the subject of speculation
access of the gonadotropins to the folliclespreventsaccess of the gonadotropins to the follicles
to sudden stoppage of the heart musclleadingto sudden stoppage of the heart muscl
the early onset of OHSS and decreases OHSS 's severity in ratspreventsthe early onset of OHSS and decreases OHSS 's severity in rats