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Qaagi - Book of Why

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PMID : 27576135 the chaperone 78-kDa glucose - regulated protein ( GRP78 ) protects the MPD against PDI - dependent disulfide - bond isomerization by binding to this domain and , therebypreventingADAM17 inhibition

pressure on the carotid sinus(passive) created byvagal inhibition

developmental lead exposure in the dentate gyrus of rat Chemical Abstract(passive) caused byneurogenesis inhibition

/ FREE Full Text23. ↵ Herrmann J , Saguner AM , Versari D , Peterson TE , Chade A , Olson M , Lerman LO , Lerman A. Chroniccontributesproteasome inhibition

The qnr genes encode proteins that prevent fluoroquinolones from interacting with DNA / type - II - topoisomerase complexes formed during DNA replicationthus preventingfluoroquinolone inhibition

developmental lead exposure in the dentate gyrus of rat | SpringerLink November 2014(passive) caused byneurogenesis inhibition

Science Spermpreventscalcineurin inhibition

a commercial mixture of cellulases from T. reesei and excess of β - glucosidaseto preventcellobiose inhibition

Perry , DK , Burns , JM , Pollinger , HS , Amiot , BP , Gloor , JM , Gores , GJ & Stegall , MD 2009causesProteasome inhibition

| Molecular Neurodegeneration | Full Text Histoneleadsdeacetylase inhibition

In particular , the gold ( III ) compound AuL12 was reportedto causeproteasome inhibition

retrograde signal from metabotropic glutamate to cannabinoid receptors , NEURON , 31(3 ) , 2001 , pp(passive) caused byPresynaptic inhibition

Publisher Site | Google Scholar M. Lorenz , N. Wilck , S. Meiners et al .preventsProteasome inhibition

Publisher Site | Google Scholar R. Gordon , E. A. Albornoz , D. C. Christie et al .preventsInflammasome inhibition

Publisher Site | Google Scholar D. K. Perry , J. M. Burns , H. S. Pollinger et al .causesProteasome inhibition

43 2000 Pertussis toxin ... Ribeiro , J. A. article openAccess 44 2008 Potential therapeutic interest of adenosine A2A receptors in psychiatric disorders Cunha , Rodrigo A. ; Ferré , Sergi ; Vaugeois , Jean - Marie ; Chen , Jiang - Fan article openAccess 45 2014preventspresynaptic inhibition

Janprevents31.PARP-1 inhibition

GWI Tyrosine ki-preventsnase inhibition

This steppreventsqPCR inhibition

This EE pathwaypreventedLTP inhibition

The tonic current through these channelscan causeshunting inhibition

UVC irradiationtriggerstranscriptional inhibition

Thelitz , S , Oishi , P , Sanchez , LS , Bekker , JM , Ovadia , B , Johengen , MJ , Black , SM & Fineman , JR 2004preventsPhosphodiesterase-3 inhibition

By selectively exciting A - beta nerve fibres in the skin with TENS , the amount of painful stimulation being transmitted by smaller diameter A - delta and C - fibres can be reducedcausingsegmental inhibition

D'Uscio , LV , Quaschning , T , Burnett , JCJ & Lüscher , TF 2001preventsVasopeptidase inhibition

Number to send5102050100200Start from citationCreate File Select itemprevents236902291.Calpain inhibition

another drugcausespresynaptic inhibition

basal PGE2 ... thosecausedTxB2 inhibition

only enough pressureto triggerautogenic inhibition

Granic , I , Nyakas , C , Luiten , PGM , Eisel , ULM , Halmy , LG , Gross , G , Schoemaker , H , Möller , A & Nimmrich , V 2010preventsCalpain inhibition

release of an inhibitory substance onto the outsides of the presy - naptic nerve fibrils before their own endings terminate on the postsynaptic neuron(passive) is caused byPresynaptic inhibition

Long - term systemic vascular endothelial growth factorcan resultVEGF ) inhibition

any nerve or mobility restrictionsmay be causingneurogenic inhibition

author = { Malmgren , Siri and Ahrén , Bo } , pages = { 1091 - -1099 } , titlecontributesDPP-4 inhibition

The PAR-1 inhibitor SCH79797 and PAR-1 antibodypreventedTRPV5 inhibition

exactly howpreventsmTOR inhibition

The potential physiological benefits ofleadDGAT-1 inhibition

blockade of sodium channelscausespresynaptic inhibition

Spontaneous dopamine release in the presence of cocainecausedpresynaptic inhibition

orderto preventqPCR inhibition

α - synuclein pathology and dopaminergic neurodegeneration in mice ... ” Science Translational Medicine ( 2018preventsα - synuclein pathology and dopaminergic neurodegeneration in mice ... ” Science Translational Medicine ( 2018

α - synuclein pathology and dopaminergic neurodegeneration in mice Low - fouling fluoropolymerspreventsα - synuclein pathology and dopaminergic neurodegeneration in mice Low - fouling fluoropolymers

alpha - synuclein pathology and dopaminergic neurodegeneration in micepreventsalpha - synuclein pathology and dopaminergic neurodegeneration in mice

to antitumor activity in ovarian cancer Abstract # 3582leadsto antitumor activity in ovarian cancer Abstract # 3582

to Antitumor Activity in Ovarian Cancer Vivas - MejiaLeadsto Antitumor Activity in Ovarian Cancer Vivas - Mejia

medulloblastoma cell proliferation , self - renewal , cell - cycle progression , and induced apoptosispreventedmedulloblastoma cell proliferation , self - renewal , cell - cycle progression , and induced apoptosis

to a reduced spine density [ 36ledto a reduced spine density [ 36

to a failure of mitotic division during the first mitotic division in pig embryos in JOURNAL OF ASSISTED REPRODUCTION AND GENETICS MESHleadsto a failure of mitotic division during the first mitotic division in pig embryos in JOURNAL OF ASSISTED REPRODUCTION AND GENETICS MESH

neurodegeneration [ 13causesneurodegeneration [ 13

that ibrutinib enhances NAD+ and ATP depletion(passive) triggered bythat ibrutinib enhances NAD+ and ATP depletion

sudden deathcausessudden death

to a cessation of mRNA export ( Tokunaga et al . , 2006leadsto a cessation of mRNA export ( Tokunaga et al . , 2006

α - synuclein pathology and dopaminergic neurodegeneration in mice Authors : Gordon R , Albornoz EA , Christie DC , Langley MR , Kumar V , Mantovani S , Robertson AAB , Butler MS , Rowe DB , O’Neill LA , Kanthasamy AG , Schroder K , Cooper MA , Woodruff TMpreventsα - synuclein pathology and dopaminergic neurodegeneration in mice Authors : Gordon R , Albornoz EA , Christie DC , Langley MR , Kumar V , Mantovani S , Robertson AAB , Butler MS , Rowe DB , O’Neill LA , Kanthasamy AG , Schroder K , Cooper MA , Woodruff TM

to Antitumor Activity in Ovarian CancerPoster SessionLeadsto Antitumor Activity in Ovarian CancerPoster Session

to cell death in MM cell lines and patient myeloma samplesleadsto cell death in MM cell lines and patient myeloma samples

to decreased methylation and increased synaptic activity.[2leadsto decreased methylation and increased synaptic activity.[2

α - synuclein pathology and dopaminergic neurodegeneration in mice — Macquarie University Richard Gordon , Eduardo A. Albornoz , Daniel C. Christie , Monica R. Langley , Vinod Kumar , Susanna Mantovani , Avril A. B. Robertson , Mark S. Butler , Dominic B. Rowe , Luke A. O’Neill , Anumantha G. Kanthasamy , Kate Schroder , Matthew A. Cooperpreventsα - synuclein pathology and dopaminergic neurodegeneration in mice — Macquarie University Richard Gordon , Eduardo A. Albornoz , Daniel C. Christie , Monica R. Langley , Vinod Kumar , Susanna Mantovani , Avril A. B. Robertson , Mark S. Butler , Dominic B. Rowe , Luke A. O’Neill , Anumantha G. Kanthasamy , Kate Schroder , Matthew A. Cooper

accumulation of GCP - WD and γ - tubulin at centrosomes ... but also reduces their spindle localizationpreventsaccumulation of GCP - WD and γ - tubulin at centrosomes ... but also reduces their spindle localization

G2 / M arrest , which is the cell cycle phase that is known to be more sensitive to ionizing radiationcausesG2 / M arrest , which is the cell cycle phase that is known to be more sensitive to ionizing radiation

rapid death from cardiac arrest(passive) caused byrapid death from cardiac arrest

metabolic dysfunction and photoreceptor deathcausesmetabolic dysfunction and photoreceptor death

to defective polyploidization and cell death in megakaryocytes ... a result with implications in the design of therapeutic protocols targeting this mitotic kinaseleadsto defective polyploidization and cell death in megakaryocytes ... a result with implications in the design of therapeutic protocols targeting this mitotic kinase

to pharmacodynamic pHisH3 modulation and significant antitumor activity in multiple DLBCL modelsleadsto pharmacodynamic pHisH3 modulation and significant antitumor activity in multiple DLBCL models

the degradation of a number of substrates , some of which may also impact myocardial signaling ( reviewed extensively elsewhere 4,9preventsthe degradation of a number of substrates , some of which may also impact myocardial signaling ( reviewed extensively elsewhere 4,9

in apoptosis of early progenitors and maturation of differentiated neutrophilsresultsin apoptosis of early progenitors and maturation of differentiated neutrophils

the NAD formation mechanism during ES - induced muscle contractioninfluencedthe NAD formation mechanism during ES - induced muscle contraction

apoptosis and suppressed the growth of PDK1- and MYC - driven xenograft tumorstriggeredapoptosis and suppressed the growth of PDK1- and MYC - driven xenograft tumors

to alteredexpression of NF - κB - dependent genes resulting in reduced apoptosisleadsto alteredexpression of NF - κB - dependent genes resulting in reduced apoptosis

to induction of differentiation , cell cycle arrest and apoptosis in AML cellsleadsto induction of differentiation , cell cycle arrest and apoptosis in AML cells

to sensitize TRAIL - induced apoptosis in human lung cancer H1299 cellscontributesto sensitize TRAIL - induced apoptosis in human lung cancer H1299 cells

MB cell proliferation , self - renewal , cell cycle progression , and induced apoptosispreventedMB cell proliferation , self - renewal , cell cycle progression , and induced apoptosis

AKT1 / FOXO3 / NOXA – dependent apoptosis in both EBV - positive and -negative lymphoma cellstriggeredAKT1 / FOXO3 / NOXA – dependent apoptosis in both EBV - positive and -negative lymphoma cells

/ delay enzalutamide resistance and restore AR function , leading to reductions in tumor burdencan prevent/ delay enzalutamide resistance and restore AR function , leading to reductions in tumor burden

to increased levels of ROS and apoptosis in cells exposed to bronchoalveolar lavage fluids from infected animalsalso ledto increased levels of ROS and apoptosis in cells exposed to bronchoalveolar lavage fluids from infected animals

AKT1 / FOXO3 / NOXA – dependent apoptosis in EBV - positive and -negative Burkitt 's lymphoma cellstriggeredAKT1 / FOXO3 / NOXA – dependent apoptosis in EBV - positive and -negative Burkitt 's lymphoma cells

Epstein - Barr virus lytic cycle and apoptosis in immortalized and transformed B cells : a basis for new therapiestriggersEpstein - Barr virus lytic cycle and apoptosis in immortalized and transformed B cells : a basis for new therapies

in a reduction in CV events as captured by the primary end point,5 and the mechanism of benefit is the subject of speculationresultedin a reduction in CV events as captured by the primary end point,5 and the mechanism of benefit is the subject of speculation

access of the gonadotropins to the folliclespreventsaccess of the gonadotropins to the follicles

to sudden stoppage of the heart musclleadingto sudden stoppage of the heart muscl

the early onset of OHSS and decreases OHSS 's severity in ratspreventsthe early onset of OHSS and decreases OHSS 's severity in rats

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