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Qaagi - Book of Why

Causes

Effects

an abilityto influencetranscription factors

of two subunits : c - fos and c - jun(passive) is composedAP-1 transcription factor

Discovery and characterization of RUNX1 Nusslein - Volhard and Wieschausdiscoveredthe transcription factor RUNX

Rel family of genes(passive) is composedThe transcription factor

Antioxidant responsive element , Quinone reductase , Oxidative stresscontributesThe Nrf2 transcription factor

UW researchers ( gray and red(passive) designed bya transcription factor

microRNA and RNAi Cell signalingcan triggertranscription factors

forms a complex with HIF-1βcreatingHIF-1 transcription factor

Activated MAPK can enter the nucleusto triggertranscription factors

McMahon , M. , Itoh , K. , Yamamoto , M. & Hayes , J. D. Keap1-dependent proteasomal degradation ofcontributestranscription factor Nrf2

Calcineurin inhibitors , including FK506 , serveto preventtranscription of the autocrine factor

to regulate the expression of genes coding for 5S RNA(passive) was discoveredThe transcription factor

Ca2 + -mediated calcineurin activity ( 81 , 124(passive) triggered bya transcription factor

of five open reading frames ( ORFs ) that contains genes that respond to biotin - mediated feedback repression(passive) is composedThe transcription factor

as a way neurons activity could change its structure  Signal passing between post and pre synaptic neurons= nitric oxide(passive) were discovered Transcription factor

of an alpha subunit , encoded by the RUNX1 , RUNX2 , and RUNX3 genes , which binds to DNA via a Runt domain , and a beta subunit , encoded by the CBFB gene ( ) , which increases the affinity of the alpha subunit for(passive) are composedRUNX transcription factors

of proteins , which are coded for in our(passive) are composedTranscription factors

the nucleotide sequence variations of the HLA - DR promoter(passive) influenced bytranscription factors

of two essential functional regions : a DNA - binding domain and an activator domain(passive) are composedTranscription factors

a transcriptional regulatory axis ,composedof transcription factor

of two modules : the DNA‐binding domain and the transcription activation domain(passive) are generally composedTranscription factors

in prostate cancer(passive) were discoveredtranscription factors

Urnov , F.D. and Rebar , E.J. ( 2002Designedtranscription factors

of at least four discrete domains : a DNA - binding domain , a nuclear - localization signal , a transcription - activation domain , and an oligomerization site(passive) are usually composedTranscription factors

of more than one polypeptide(passive) can be composedTranscription factors

the transversion(passive) created bytranscription factors

epigenetic mechanisms(passive) are triggered byTranscription factors

to target DNA sequences contained within the DNase I - hypersensitive regions [ 10(passive) were designedZFP ) transcription factors

of several functional domains , one of which must be a DNA - binding domain(passive) may be composedTranscription factors

of distinct functional modules(passive) are composedTranscription factors

a specific DNA sequencehas createdZFP transcription factors

Takahashi in 2006(passive) discovered bypluripotent transcription factors

The kinase activity of both AKT and SGK1 toward the forkhead box protein O1 ( FOXO1 ) andcausestranscription factors

reactive species and proinflammmatory(passive) are ... influenced byTranscription factors

The first oneis designingtranscription factors

gene fusions as the result of chromosomal translocations(passive) created byUSA.AbstractChimeric transcription factors

CDK8influencetranscription factors

upon transfer of Arabidopsis(passive) is triggeredtranscription factors

hypoxia in advanced bulky HCC tumors(passive) triggered bya transcription factor

post - translational modification , expression levels , and protein stability ... knownto influencetranscription factor activity

to the regulation of ABA homeostasis and drought stress tolerance in Arabidopsis thaliana 12contributesto the regulation of ABA homeostasis and drought stress tolerance in Arabidopsis thaliana 12

the DNA to loop so that the regulatory sequence ( and bound transcription factor ) become close to the RNA polymerase binding site.[25causethe DNA to loop so that the regulatory sequence ( and bound transcription factor ) become close to the RNA polymerase binding site.[25

the DNA to loop so that the regulatory sequence ( and bound transcription factor ) become close to the RNA polymerase binding site.[33causethe DNA to loop so that the regulatory sequence ( and bound transcription factor ) become close to the RNA polymerase binding site.[33

the DNA to loop so that the regulatory sequence ( and bound transcription factor ) become close to the RNA polymerase binding site.[62causethe DNA to loop so that the regulatory sequence ( and bound transcription factor ) become close to the RNA polymerase binding site.[62

to the regulation of ABA homeostasis and drought stress tolerance in Arabidopsis thaliana Moritz , Thomascontributesto the regulation of ABA homeostasis and drought stress tolerance in Arabidopsis thaliana Moritz , Thomas

to cell differentiation and proliferationleadsto cell differentiation and proliferation

to cellular proliferation or differentiation ( 30leadingto cellular proliferation or differentiation ( 30

changes in gene regulationcausingchanges in gene regulation

CD4 + T cell differentiationcould influenceCD4 + T cell differentiation

neural stem / progenitor cell differentiation and maturation in vivoinfluencesneural stem / progenitor cell differentiation and maturation in vivo

dramatic changes in gene expressioncan triggerdramatic changes in gene expression

cell cycle and proliferationinfluencescell cycle and proliferation

to the expression of a gene [ 9leadingto the expression of a gene [ 9

of HIF-1α and HIF-1β subunitscomposedof HIF-1α and HIF-1β subunits

neuronal differentiationinfluenceneuronal differentiation

in altered gene expression and therefore cell proliferationresultingin altered gene expression and therefore cell proliferation

to changes in gene expression < ref namewill leadto changes in gene expression < ref name

cancer through coordinated changes in gene expressioncausecancer through coordinated changes in gene expression

to changes in gene expression and enzyme activitiesleadingto changes in gene expression and enzyme activities

to cell proliferation ( regenerationleadingto cell proliferation ( regeneration

to the induction of GLT-1 expression in astrocytes through an interaction with a distal enhancer elementcontributesto the induction of GLT-1 expression in astrocytes through an interaction with a distal enhancer element

to the expression of the gene products ( growth , angiogenesis , proliferationleadsto the expression of the gene products ( growth , angiogenesis , proliferation

to activation of the genes involved in cell proliferationleadsto activation of the genes involved in cell proliferation

to cell cycle arrest and apoptosisleadingto cell cycle arrest and apoptosis

to changes in gene expression after TGF - β1 treatmentleadto changes in gene expression after TGF - β1 treatment

reprogramming of cardiac gene expressiontriggerreprogramming of cardiac gene expression

to silencing of specific gene expression.[9leadingto silencing of specific gene expression.[9

to silencing of specific gene expression.[5leadingto silencing of specific gene expression.[5

to altered gene expression ( 2leadingto altered gene expression ( 2

to the expression of genes involved in growth and differentiation.65leadsto the expression of genes involved in growth and differentiation.65

to both embryonic and adult neurogenesis as a multifunctional regulatorcontributesto both embryonic and adult neurogenesis as a multifunctional regulator

to improved cell survival and protein synthesisleadto improved cell survival and protein synthesis

Th2 and Th17 differentiation and inflammationinfluenceTh2 and Th17 differentiation and inflammation

to activation or silencing of genes important in cell differentiation and maintenanceleadingto activation or silencing of genes important in cell differentiation and maintenance

to cell proliferation [ 9–11leadingto cell proliferation [ 9–11

the expression of some genes[2causingthe expression of some genes[2

cell type differentiation andinfluencingcell type differentiation and

to the generation of retinal cell typeleadingto the generation of retinal cell type

to changes in gene expression.[http://www.youtube.com /will leadto changes in gene expression.[http://www.youtube.com /

osteoblast differentiation ( Runx2 and Sp7triggeringosteoblast differentiation ( Runx2 and Sp7

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