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the chronic adminstrations of aluminum and sodium fluoride(passive) caused byK. F. Toxin - induced blood vessel inclusions
the chronic administrations of aluminum and sodium fluoride(passive) caused byK. F. Toxin - induced blood vessel inclusions
The application of doxycycline might offer a new therapeutic approachto preventNO - induced cochlea damage through ototoxic substances
This compoundpreventedalmost all the Aβ oligomer - induced neurotoxicity
PAA - mediated blockage of late viral gene expression at this time pointresultedin no suppressive effect on IFN - induced STAT2 phosphorylation
silybinpreventedcisplatin induced nephrotoxicity ... but did not prevent cyclosporine - induced glomerular damage except for lipid peroxidation
treatment withresultedin near - complete abrogation of reovirus - induced c - Jun phosphorylation without concomitant ERK inhibition
Group OC ,composedof DMBA - induced animals that did not consume EtOH
cell culture studies ... serious enoughto causecholestasis drugs known to be virtually no corticospinal axons regenerated
As PMA is knownto preventthe phenylephrine - induced increase in cytoplasmic free Ca2
the potential influence on energy metabolism and reactive oxidative species productioncausingthe potential influence on energy metabolism and reactive oxidative species production
the damage(passive) caused bythe damage
oftenhave ... ledoften
synaptic vesicle depletion(passive) is caused ... bysynaptic vesicle depletion
in the down - regulation of PPAR gamma activityresultingin the down - regulation of PPAR gamma activity
SIK2 intrinsic catalytic activity when isolated from hepatocytes / liverto influenceSIK2 intrinsic catalytic activity when isolated from hepatocytes / liver
to the activation of phosphatidylcholine - specific phospholipase C ( PLC ... to increase intracellular Ca2 + concentration , resulting in Ca2leadsto the activation of phosphatidylcholine - specific phospholipase C ( PLC ... to increase intracellular Ca2 + concentration , resulting in Ca2
to an increase in the level of TMRE stainingledto an increase in the level of TMRE staining
to strong association of Gab1 with tyrosine phosphatase SHP2 and PI3-kinase p85 subunitleadingto strong association of Gab1 with tyrosine phosphatase SHP2 and PI3-kinase p85 subunit
increased morbidity and mortalitycan causeincreased morbidity and mortality