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which is likely due to reduced binding affinity.3would leadto reduced receptor stimulation
as crucial pattern recognition receptors ( PRRs ) involved in the recognition of pathogen - associated molecular patterns ( PAMPs(passive) are discoveredToll - like receptors ( TLRs
First , autoantibodies may act as an agonist or a receptor ligandcausingexcessive receptor stimulation
Mannose - binding lectin ( MBL ) andmay influenceToll - like receptor ( TLR ) polymorphisms
Researchersdiscoveredtoll - like receptors
It is caused by mutation in the EGF(Epithelial growth factor ) gene ,resultinginadequate stimulation of EGF receptor
first(passive) have been ... discoveredToll - like receptors
first(passive) were ... discoveredToll - like receptors
Activation of human macrophages bycausestoll - like receptors
TLR ) family in human(passive) is composedThe Toll - like receptor
TLR ) family(passive) is composedThe Toll - like receptor
in Drosophila(passive) were discoveredToll - like receptors
he and Janewaydiscoveredtoll - like receptors
Mutations of the c - kit receptorleadingto uncontrolled stimulation of the receptor
activationresultingfrom LPA receptor stimulation
the c - kit receptorleadingto uncontrolled stimulation of the receptor
extracellular signalsoriginatedfrom prolonged receptor stimulation
the increase in PRApossibly resultingfrom macula densa receptor stimulation
Both processes of production and degradationeffectively causetransient receptor stimulation
Activation of adenylyl cyclasecausesstimulation of adenosine receptors
Such treatment is knownto resultin CD3 receptor stimulation
firingresultingfrom glutamate receptor stimulation
Dr. Siragy ... the firstto discoverthat stimulation of angiotensin AT2 receptor
Physiological effects ... analgesiaresultfrom cannabinoid receptor stimulation
toxicityresultingfrom AMPA / kainate receptor stimulation
Calcium influxresultingfrom NMDA receptor stimulation
Several modalitiescan resultin AT2 receptor stimulation
Box 2 Possible mechanisms of interaction between agonists and steroids leadsreceptor stimulation
disease that are directed against the TSH receptor located on the thyroid cell surfacecausingstimulation of this receptor
that are directed against the TSH receptorcausingstimulation of this receptor
TSH receptor antibodies often present in the serum of patients with Graves disease that are directed against the TSH receptorcausingstimulation of this receptor
TSH receptor antibodies : antibodies often present in the serum of patients with Graves disease that are directed against the TSH receptorcausingstimulation of this receptor
that are directed against the TSH receptor located on the thyroid cell surfacecausingstimulation of this receptor
by stimulating antibodies(passive) caused bythe receptor stimulation
all the physiological responsesresultfrom the stimulation of receptors
a medicationcausesstimulation of receptors
to the changes produced by brain deathcontributesto the changes produced by brain death
in increased production for all six cytokinesresultedin increased production for all six cytokines
in inflammatory cytokine expressionresultingin inflammatory cytokine expression
in the activation of innate immune cells such as macrophages and dendritic cellsresultsin the activation of innate immune cells such as macrophages and dendritic cells
to innate immune responsescontributesto innate immune responses
to muscle protein degradationcontributesto muscle protein degradation
to activationleadsto activation
in an increased cardiac myocyte apoptosis via cAMP - dependent mechanism ( Communal et al .resultsin an increased cardiac myocyte apoptosis via cAMP - dependent mechanism ( Communal et al .
alsocan ... leadalso
neuroprotection in a conscious rat model of strokecausesneuroprotection in a conscious rat model of stroke
the release of various neurotransmitters including serotonin , dopamine , norepinephrine , and acetylcholineinfluencesthe release of various neurotransmitters including serotonin , dopamine , norepinephrine , and acetylcholine
in increases production of astroglial Norrinalso resultedin increases production of astroglial Norrin
to the development of motor complicationsleadsto the development of motor complications
the release of D - serinecausedthe release of D - serine
to the decreased expression of P2Y12 and P2Y13 receptorswould leadto the decreased expression of P2Y12 and P2Y13 receptors
activation of the G protein and the formation of an intracellular chemical , the second messengercausesactivation of the G protein and the formation of an intracellular chemical , the second messenger
in activation of transcriptionresultsin activation of transcription
in a loss of SERT activityresultingin a loss of SERT activity
in activation ofalso resultsin activation of
relaxation of vascular smooth muscle and airway smooth muscle resulting in vasodilation and bronchodilation , respectivelycausesrelaxation of vascular smooth muscle and airway smooth muscle resulting in vasodilation and bronchodilation , respectively
in a change in cell activity , which may send feedback to the original hormone - producing cellresultsin a change in cell activity , which may send feedback to the original hormone - producing cell
cardiac hypertrophycausescardiac hypertrophy
a buildup of intracellular inositol triphosphate and thereby an increase of cytosolic Ca2causesa buildup of intracellular inositol triphosphate and thereby an increase of cytosolic Ca2
in vasoconstrictionresultsin vasoconstriction
to a further recruitment of the kinasesledto a further recruitment of the kinases
in relaxationresultsin relaxation
contraction and inositol phosphate increase in medium - sizecausescontraction and inositol phosphate increase in medium - size
activation of PLA2(passive) caused byactivation of PLA2
cardiac hypertrophycausescardiac hypertrophy
in the rapid internalization and sequestration of the receptors at the perinuclear arearesultsin the rapid internalization and sequestration of the receptors at the perinuclear area
in many pathophysiological eventsmay resultin many pathophysiological events
The analgesic , rewarding , and stimulant effects(passive) are probably caused byThe analgesic , rewarding , and stimulant effects
increased heart rate and myocardial contractilitycausesincreased heart rate and myocardial contractility
to increased vascular smooth muscle contraction , pupillary dilator muscle contraction , and intestinal sphincter muscle contractionleadsto increased vascular smooth muscle contraction , pupillary dilator muscle contraction , and intestinal sphincter muscle contraction
contraction and inositol phosphate increase in medium - size human isolated bronchicausescontraction and inositol phosphate increase in medium - size human isolated bronchi
decrease in ophthalmic tonuscausesdecrease in ophthalmic tonus
to fasciculations and muscle weakness , which is most pronounced in the respiratory muscles , which leads to worsening of pulmonary dysfunction caused by bronchorrhea from muscarinic effectsleadsto fasciculations and muscle weakness , which is most pronounced in the respiratory muscles , which leads to worsening of pulmonary dysfunction caused by bronchorrhea from muscarinic effects
slow inhibition of IRK1 inwardly rectifying K+ channels by direct protein kinase A - mediated phosphorylationcausesslow inhibition of IRK1 inwardly rectifying K+ channels by direct protein kinase A - mediated phosphorylation
in the release of both IP 3 and DAG , each of which then goes on to elicit a separate cellular responseresultsin the release of both IP 3 and DAG , each of which then goes on to elicit a separate cellular response
in the postsynaptic membrane becoming more electrically positive ( depolarizedresultsin the postsynaptic membrane becoming more electrically positive ( depolarized