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the decrease in blood flow(passive) caused bytissue hypoxia
adequate blood flowcausingTissue Hypoxia
forward blood flowresultingin tissue hypoxia
Poor blood flowmay resultin tissue hypoxia
impaired blood flow(passive) caused bytissue hypoxia
microvascular blood flowleadingto tissue hypoxia
reduction in blood flow , arterial hypoxemia , or both(passive) caused bytissue hypoxia
Formation of carboxyhemoglobin reduces oxygen - carrying capacity of the bloodcausingtissue hypoxia
extended constriction of blood flow to any arealeadsto tissue hypoxia
hypoxemia or ischemiaresultingin tissue hypoxia
decreased blood flow and thrombosis of microvessels(passive) caused byTissue hypoxia
while also increasing blood flow by vasodilationthus preventingtissue hypoxia
the blood flow known as " vaso - occlusion eventleadto tissue hypoxia
The fundamental physiologic manifestation of anemia is a reduced oxygen - carrying capacity of the bloodresultingin tissue hypoxia
small blood vesselsleadingto tissue hypoxia
This decreases the oxygen carrying capacity of the bloodresultingin tissue hypoxia
More specifically , it is a hyperactivation of the sympathetic system causing extreme vasoconstriction of the peripheral blood vesselsleadingto tissue hypoxia
Small , pale red blood cells ( erythrocytescausetissue hypoxia
Mechanism Its pathophysiology includes hyperactivation of the sympathetic nervous system causing extreme vasoconstriction of the peripheral blood vesselsleadingto tissue hypoxia
that is characterized by formation of small , pale red blood cellscausingtissue hypoxia
to deliver it to peripheral tissuesthus contributingto tissue hypoxia
reaching tissuesthus causingtissue hypoxia
cadmium in most tissues(passive) caused bytissue histotoxic hypoxia
AB - Disruption of blood vessels caused by a spinal cord injuryleadsto tissue hypoxia
Anemia is a deficiency of red blood cells ( RBCs ) and/or hemoglobin , which results in a reduced ability of blood to transfer oxygen to the tissuescausingtissue hypoxia
lung disease(passive) caused bytissue hypoxia
vascular diseaseleadingto tissue hypoxia
the disease processcausingtissue hypoxia
cardiac diseaseresultingfrom tissue hypoxia
circulating bloodresultinginto tissue hypoxia
According to Wikipedia , Raynaud ’s syndrome is a hyperactivation of the sympathetic system causing extreme vasoconstriction of the peripheral blood vesselsleadingto tissue hypoxia
Ischemia , however , results from a rate of blood flow that is insufficient to satisfy metabolic demandsthereby leadingto tissue hypoxia
the oxygen carrying capacity of the blood decreasesmay causetissue hypoxia
Carbon monoxide ( CO ) poisoningcan causetissue hypoxia
The phenomenon causes extreme vasoconstriction of the peripheral blood vessels in the fingers and toesleadingto tissue hypoxia
a reduction in both insulin - independent and insulin - dependent Class 1 glucose transporters , a reduction in cell membrane functionality , and a decreased microcirculatory blood flowresultsin tissue hypoxia
Seong Rok N2 - Disruption of blood vessels caused by a spinal cord injuryleadsto tissue hypoxia
will keep the tissue around the blood vessel from swelling and constricting blood flow to your extremitiesleadingto tissue hypoxia
hypoxemia , anemia , or hemorrhage ( 21(passive) caused bytissue hypoxia
blood carrying oxygen capacityresultingin tissue hypoxia
most cases of lactic acidosis(passive) are caused bymost cases of lactic acidosis
to anaerobic metabolism , which causes metabolic acidosis.[ems1.comleadsto anaerobic metabolism , which causes metabolic acidosis.[ems1.com
to anaerobic metabolism and metabolic acidosisleadsto anaerobic metabolism and metabolic acidosis
to severe metabolic acidosisthen leadsto severe metabolic acidosis
to high anion - gap metabolic acidosismay leadto high anion - gap metabolic acidosis
in premature lactate mediated metabolic acidosisresultsin premature lactate mediated metabolic acidosis
to neuronal damagecan leadto neuronal damage
permanent damagecausespermanent damage
to cellular dysfunctionleadsto cellular dysfunction
to gastric injury and a slow healing process [ 18leadsto gastric injury and a slow healing process [ 18
cellular mechanisms that lead to the development of insulin resistance , to a state of chronic inflammation with infiltration of macrophages ... the reduction of adiponectin and increased of leptin , adipocyte death , ER stress and mitochondrial dysfunction [ 30 - 34may causecellular mechanisms that lead to the development of insulin resistance , to a state of chronic inflammation with infiltration of macrophages ... the reduction of adiponectin and increased of leptin , adipocyte death , ER stress and mitochondrial dysfunction [ 30 - 34
ischemic injury(passive) caused byischemic injury
anaerobic metabolism , which increases lactate productioncausesanaerobic metabolism , which increases lactate production
asphyxiation or brain damagecan causeasphyxiation or brain damage
an increased anion gap metabolic acidosis secondary to increased lactic acid productionmay causean increased anion gap metabolic acidosis secondary to increased lactic acid production
from hypocapnic vasoconstriction and inhibition of oxygen - hemoglobin dissociation following the elevation in pH ( the Bohr Effectresultingfrom hypocapnic vasoconstriction and inhibition of oxygen - hemoglobin dissociation following the elevation in pH ( the Bohr Effect
to the generation of hypoxia - inducible growth factors and the recruitment of inflammatory cellsleadsto the generation of hypoxia - inducible growth factors and the recruitment of inflammatory cells
to organ dysfunction and cellular death.7 Data analysismay leadto organ dysfunction and cellular death.7 Data analysis
to contractile dysfunction of the heart and the development of heart failure.1contributeto contractile dysfunction of the heart and the development of heart failure.1
to tissue damage or cell deathleadsto tissue damage or cell death
to cell and organ damage 3may leadto cell and organ damage 3
lung damage in the sepsis modelcausinglung damage in the sepsis model
angiogenesis and sFlt-1 productioncould influenceangiogenesis and sFlt-1 production
to production of hypoxia inducible factor 2αleadsto production of hypoxia inducible factor 2α
to increased [ lactate ] concentrationsdoes ... leadto increased [ lactate ] concentrations
tissue acidosis and pain , which then causes blood alkalosis , which lowers 2.3 DPG even furthercausestissue acidosis and pain , which then causes blood alkalosis , which lowers 2.3 DPG even further
to organ failure [ 1could leadto organ failure [ 1
severe ischemic pain or even strokecan causesevere ischemic pain or even stroke
to death or permanent neurologic damage if untreatedcan leadto death or permanent neurologic damage if untreated
to upregulation of HIF-1α [ 51 , 52leadsto upregulation of HIF-1α [ 51 , 52
in lactic acid accumulation , ketoacidosis , drugs , toxins , and renal retention Metabolic Acidosis ( Manifestations ) Manifestations•Appear as regulatory systems fail to maintainresultingin lactic acid accumulation , ketoacidosis , drugs , toxins , and renal retention Metabolic Acidosis ( Manifestations ) Manifestations•Appear as regulatory systems fail to maintain
to disease progression ( Girouard and Iadecola , 2006could contributeto disease progression ( Girouard and Iadecola , 2006
from cerebrovascular compressionresultingfrom cerebrovascular compression
an increase in intermediates of the citric acid cycle [ 33 , 34can causean increase in intermediates of the citric acid cycle [ 33 , 34
to anaerobic metabolism and lactic acid production that may be aggravated by abundant glucose supply andleadsto anaerobic metabolism and lactic acid production that may be aggravated by abundant glucose supply and
to contractile dysfunction of the heart and the development of heart failure.1Transforming growth factor β ( TGFβcontributeto contractile dysfunction of the heart and the development of heart failure.1Transforming growth factor β ( TGFβ
hyperlactemia which then caused acidosis , cell death and generalised physiological Armageddoncausedhyperlactemia which then caused acidosis , cell death and generalised physiological Armageddon
to a progressive depression of the cellular purine nucleotide pool creating an energy deficitleadsto a progressive depression of the cellular purine nucleotide pool creating an energy deficit
inflammatory changes ( left ) and inflammatory diseases leadingcausesinflammatory changes ( left ) and inflammatory diseases leading
to production of hypoxia inducible factor 1 ( HIF‑1 ) which upregulates production of VEGFleadsto production of hypoxia inducible factor 1 ( HIF‑1 ) which upregulates production of VEGF