to AML cell deathleadsto AML cell death

to cell death ( 5 , 6leadsto cell death ( 5 , 6

cancer cell death by depleting mono - unsaturated fatty acidscausescancer cell death by depleting mono - unsaturated fatty acids

to inhibition of tumor cell interactions with E - selectin , VCAM1 , and MADCAM1leadsto inhibition of tumor cell interactions with E - selectin , VCAM1 , and MADCAM1

that ibrutinib enhances NAD+ and ATP depletion(passive) triggered bythat ibrutinib enhances NAD+ and ATP depletion

Expression of membrane - targeted Gα13 rescues spine enlargement(passive) caused byExpression of membrane - targeted Gα13 rescues spine enlargement

diet - induced obesity and insulin resistance in LDLr-/- Apob100/100 micepreventsdiet - induced obesity and insulin resistance in LDLr-/- Apob100/100 mice

both suppress spine enlargement(passive) caused byboth suppress spine enlargement

the cytokinesis defects(passive) caused bythe cytokinesis defects

Spontaneous Ventricular Fibrillation in Aged Hearts | Heart and Circulatory Physiology SkipCausesSpontaneous Ventricular Fibrillation in Aged Hearts | Heart and Circulatory Physiology Skip

mitochondrial dysfunction and free radical damage Cleeter , Michael W.J.causesmitochondrial dysfunction and free radical damage Cleeter , Michael W.J.

spontaneous ventricular fibrillation in aged hearts ' ... American Journal of Physiology - Heart and Circulatory Physiology , vol .causesspontaneous ventricular fibrillation in aged hearts ' ... American Journal of Physiology - Heart and Circulatory Physiology , vol .

ligands PD - L1 and PD - L2 on CD11b+ monocytes / macrophages in the leukemia microenvironmentledligands PD - L1 and PD - L2 on CD11b+ monocytes / macrophages in the leukemia microenvironment

the presynaptic terminals from being fully activatedpreventsthe presynaptic terminals from being fully activated

spontaneous ventricular fibrillation in aged hearts Norishige Morita Translational Arrhythmia Research Section , Cardiovascular Research Laboratory , David Geffen School of Medicine at UCLA , 675 Charles E Young Dr South , MRL 3645 Mail Code 176022 , Los Angeles , CA 90095causesspontaneous ventricular fibrillation in aged hearts Norishige Morita Translational Arrhythmia Research Section , Cardiovascular Research Laboratory , David Geffen School of Medicine at UCLA , 675 Charles E Young Dr South , MRL 3645 Mail Code 176022 , Los Angeles , CA 90095

a significant reduction in the fluorescence transient ( average 20±1 % ) in excitor terminalscauseda significant reduction in the fluorescence transient ( average 20±1 % ) in excitor terminals

a smaller decrease in the EPSP induced by the mechanical stimulus than in the EPSP induced by the electrical stimuluscauseda smaller decrease in the EPSP induced by the mechanical stimulus than in the EPSP induced by the electrical stimulus

to a diminishment or even complete block of afferent spike propagation into sensory terminals ( El Manira and Clarac , 1994leadsto a diminishment or even complete block of afferent spike propagation into sensory terminals ( El Manira and Clarac , 1994

the development of thrombuspreventsthe development of thrombus

in a prolonged reduction in thrombogenicity of the restenotic plaque through this effect on TF expressionmay resultin a prolonged reduction in thrombogenicity of the restenotic plaque through this effect on TF expression

2011 Catheter thrombosis with fondaparinux(passive) is prevented by2011 Catheter thrombosis with fondaparinux

stress fiber assemblypreventsstress fiber assembly

activation of circulating cells by endogenous “ danger signalspreventsactivation of circulating cells by endogenous “ danger signals

stroke in atrial fibrillationto preventstroke in atrial fibrillation

IFN - γ , and IL-2 expression by T cells throughout the immune system and acute graft rejection after lung transplantationpreventedIFN - γ , and IL-2 expression by T cells throughout the immune system and acute graft rejection after lung transplantation

to even higher replication stress in cancer cells ... making them unable to romsey avoid dna damage bexhill during replication at atr - inhibitor doses tolerable to antrim and newtownabbey the animals or patientleadsto even higher replication stress in cancer cells ... making them unable to romsey avoid dna damage bexhill during replication at atr - inhibitor doses tolerable to antrim and newtownabbey the animals or patient

cytokine secretion by activated PBMCpreventedcytokine secretion by activated PBMC

to reduction of inflammatory cytokinesleadsto reduction of inflammatory cytokines

in a significant decrease of TLR7L antitumoral activity showing the requirement for typeresultedin a significant decrease of TLR7L antitumoral activity showing the requirement for type

TAMs from suppressing the body ’s anti - tumor immune responsepreventedTAMs from suppressing the body ’s anti - tumor immune response

p38 phosphorylationpreventsp38 phosphorylation

to both decreased levels of expression ( extra- and intracellularly ) of Siglec-1 and a lower percentage of cells expressing this receptor as induced by cmMTB treatmentleadsto both decreased levels of expression ( extra- and intracellularly ) of Siglec-1 and a lower percentage of cells expressing this receptor as induced by cmMTB treatment

the NLRP3 inflammasome activation induced by cholesterol crystalspreventsthe NLRP3 inflammasome activation induced by cholesterol crystals

Epstein - Barr virus lytic cycle and apoptosis in immortalized and transformed B cells : a basis for new therapiestriggersEpstein - Barr virus lytic cycle and apoptosis in immortalized and transformed B cells : a basis for new therapies

alsopreventsalso

AKT1 / FOXO3 / NOXA – dependent apoptosis in both EBV - positive and -negative lymphoma cellstriggeredAKT1 / FOXO3 / NOXA – dependent apoptosis in both EBV - positive and -negative lymphoma cells

rupture of established plaques in apolipoprotein E knockout mice S. Karanam , R. Fritsche - Danielson , C. L. Jacksonpreventsrupture of established plaques in apolipoprotein E knockout mice S. Karanam , R. Fritsche - Danielson , C. L. Jackson

rupture of established plaques in apolipoprotein E knockout mice R. Fritsche - Danielson , C. L. Jackson , S. Karanampreventsrupture of established plaques in apolipoprotein E knockout mice R. Fritsche - Danielson , C. L. Jackson , S. Karanam

NAD and ATP levels reduction , ii ) mitochondrial respiration impairment and iii ) decrease of ERK1/2 and NF - kB activationledNAD and ATP levels reduction , ii ) mitochondrial respiration impairment and iii ) decrease of ERK1/2 and NF - kB activation

TAA formation ( Figure 6A–6D ... and can prevent further growth of a small aneurysm after it has formedcan preventTAA formation ( Figure 6A–6D ... and can prevent further growth of a small aneurysm after it has formed