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the increased intake of phosphorus in higher quantitiescausesinhibition of 1?-hydroxylase
Hydrocodone Description : Hydrocodone is a phenanthrene derivative opiate agonist that binds to opioid receptors in the CNScausinginhibition of ascendin
relatedto promptVEGF inhibition
retrograde signal from metabotropic glutamate to cannabinoid receptors(passive) caused byPresynaptic inhibition
Activation of GABAB receptorscausespresynaptic inhibition
MG132 treatment increased levels of high molecular weight polyubiquitinated proteins after 4 and 16 h(passive) caused byproteasome inhibition
the CLAVATA1 receptorultimately causingWUSCHEL inhibition
that has previously been shownto causepresynaptic inhibition
MG132(passive) caused byproteasome inhibition
by retrograde signal from metabotropic glutamate(passive) caused byPresynaptic inhibition
Nerve agents and other organophosphate pesticidescauseacetylcholinesterase inhibition
Exposurecan causecholinesterase inhibition
vaccinecausinginhibition of erythropoiesis
by retrograde signal(passive) caused byPresynaptic inhibition
This primary afferent depolarizationcausespresynaptic inhibition
endogenous adenosine(passive) caused bypresynaptic inhibition
by the agent(passive) caused byacetylcholinesterase inhibition
by noradrenaline(passive) caused bypresynaptic inhibition
Enkephalincausespresynaptic inhibition
the inhibitory effects ... the undissociated form of the acidcausesinhibition
the concentrations of these inhibitors ... shownto causeinhibition
evidencecan influenceGABAergic inhibition
effects of molecules that are a substrate and decrease rates of enzymes(passive) are caused byInhibition
NO binding to Cox , both in isolated mitochondria and in whole cells(passive) caused byinhibition
Damaged cell studies of the concentrationcausedinhibition
GABA binding resulting in hyperpolarization of the synaptic transmembrane potential of the affected neuron(passive) is provoked byInhibition
a decrease in GABA signaling(passive) caused byinhibition
Histoneleadsdeacetylase inhibition
It is highly toxiccausingcholinesterase inhibition
by organophosphates ( OPs(passive) caused byacetylcholinesterase inhibition
carbamates(passive) caused bycholinesterase inhibition
The effect of a small quantity ... likelyto causeinhibition
by an inhibitory compound(passive) to be caused byinhibition
a sudden shockcausingvagal inhibition
a sudden shock ... orcausingvagal inhibition
The sensitivity of N - methyl - d - aspartate receptorsto leadinhibition
by the three inhibitors(passive) caused byinhibition
It binds competitively to the receptors for adenosine A1 and A2Acausinginhibition
several mechanisms(passive) can be caused byInhibition
rich proteinscausinginhibition
in decreased cellular proliferationwill resultin decreased cellular proliferation
in extensive apoptotic changes in the vasculature of neonatal , but not adult , miceresultsin extensive apoptotic changes in the vasculature of neonatal , but not adult , mice
in decreased NLRP3 inflammasome activationresultingin decreased NLRP3 inflammasome activation
to senescenceleadsto senescence
in turnledin turn
alsoleadsalso
to reduction of inflammatory cytokinesleadsto reduction of inflammatory cytokines
cellular stress via the excessive activation of the PLC - IP3 pathwaycausedcellular stress via the excessive activation of the PLC - IP3 pathway
to physiological and behavioral phenotypesleadingto physiological and behavioral phenotypes
to physiological and behavioral phenotypes leadingto physiological and behavioral phenotypes
to paired pulse effects heredoes contributeto paired pulse effects here
in the late 1950sdiscoveredin the late 1950s
cell deathcausescell death
lysosomal dysfunctioncauseslysosomal dysfunction
to changes in lipoprotein metabolismleadsto changes in lipoprotein metabolism
mitochondrial dysfunction and free radical damagecausesmitochondrial dysfunction and free radical damage
cell death(passive) caused bycell death
to inflammation , necrosis , steatosis and fibrosisleadsto inflammation , necrosis , steatosis and fibrosis
cancer cell deathcausescancer cell death
the induction of autophagy(passive) caused bythe induction of autophagy
/ macrophages in the leukemia microenvironmentled/ macrophages in the leukemia microenvironment
the accelerated atherosclerosis(passive) caused bythe accelerated atherosclerosis
to detectable pathological changes within the liver organoidsleadsto detectable pathological changes within the liver organoids
complications such as a hampered neuroplasticity or dysregulated immune reactions ... as reported recently by Bohacek et alcan causecomplications such as a hampered neuroplasticity or dysregulated immune reactions ... as reported recently by Bohacek et al
hyperactivity and impulsivity in mice via dopamine D2 receptor - mediated mechanismcauseshyperactivity and impulsivity in mice via dopamine D2 receptor - mediated mechanism
to an increase in LPL , GLUT4 and FABPs at 4 days post - infection of miR-122 inhibitorleadsto an increase in LPL , GLUT4 and FABPs at 4 days post - infection of miR-122 inhibitor
to significant raises in both free of charge and esterified cholesterolledto significant raises in both free of charge and esterified cholesterol
spontaneous ventricular fibrillation in aged heartscausesspontaneous ventricular fibrillation in aged hearts
to downregulation of the TGFB1 pathway ... and specifically by inhibition of PDGFB and CTGF ... two cardinal regulators of MSC / pericyte proliferation and functionleadsto downregulation of the TGFB1 pathway ... and specifically by inhibition of PDGFB and CTGF ... two cardinal regulators of MSC / pericyte proliferation and function
precocious spermatogonial differentiation in the adult mouse and is able to drive synchronous spermatogenesis in the neonatal mousecausesprecocious spermatogonial differentiation in the adult mouse and is able to drive synchronous spermatogenesis in the neonatal mouse
to the argument that rhomboids use a variation of the classical serine protease catalytic mechanismhas contributedto the argument that rhomboids use a variation of the classical serine protease catalytic mechanism