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the pre - established fibrinogen layercreatingthrombin
Thrombostatinspreventthrombin
cleaving PAR1 or PAR4 at its thrombin cleavage siteto preventthrombin
cleaving PAR1 at or near its thrombin cleavage siteto preventthrombin
Thromboplastin from placenta and decidua activate extrinsic clotting pathwaycausingthrombin
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The pleiotropic cytokine viagra without a doctor prescription walmart tumor necrosis factor - alpha ( TNF - alpha ) andleadthrombin
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The release of ADP and the formation of other activators such as the eicosanoid thromboxane A2 ( TXA2 ) andcreatethrombin
STBMtriggeredthrombin
10 μmol / L Y-27632preventedthrombin
that hTFPI on these cells was actingto preventthrombin
a monoclonal antibodypreventsthrombin
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Ang-1 abilityto preventthrombin
Methyl 2,5-dihydroxycinnamate , a tyrosine kinase inhibitorpreventedthrombin-
L - JNKI1 , a highly specific c - jun N - terminal kinase ( JNK ) inhibitor , and a second specific JNK inhibitor , SP600125preventedthrombin
Hirudincan preventthrombin
extrinsic and intrinsic coagulation cascades in the vascular damage site(passive) is ... triggered byThrombin
Omrix BioPharmaceuticalscreatedthrombin
that contacting the substrate with thrombin before fibrinogen results in a fibrin coating that strongly adheres to the substrate surface(passive) has been discoveredthrombin
because fibrin bound thrombin can continueto triggerthrombin
Treatment of EC with bromo - cAMPpreventedthrombin-
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to formation of a blood cloteventually leadsto formation of a blood clot
additional platelet activation and aggregationcausesadditional platelet activation and aggregation
conversion of fibrinogen to fibrin by polymerizationCa ... causesconversion of fibrinogen to fibrin by polymerization
the conversion of fibrinogen to fibrin ( coagulation(passive) caused bythe conversion of fibrinogen to fibrin ( coagulation
a blood - clotting disordercan causea blood - clotting disorder
to the conversion of fibrinogen to fibrin and a stable clotleadsto the conversion of fibrinogen to fibrin and a stable clot
conversion of fibrinogen into fibrin , which binds blood cells together to form ablood clotalso causesconversion of fibrinogen into fibrin , which binds blood cells together to form ablood clot
further fibrin formation or platelet activationTo preventfurther fibrin formation or platelet activation
an immediate conversion of fibrinogen into fibrin and subsequent clot formationtriggersan immediate conversion of fibrinogen into fibrin and subsequent clot formation
platelet activation and consumption(passive) caused byplatelet activation and consumption
blood to coagulatecausesblood to coagulate
to coagulation of bloodleadingto coagulation of blood
blood plasmaprovokesblood plasma
clotting of fibrinogencausesclotting of fibrinogen
local platelet aggregationcauseslocal platelet aggregation
to protective immunity in pneumonia - derived sepsis via fibrin polymerization and platelet - neutrophil interactions , J Thromb Haemost , 2017contributesto protective immunity in pneumonia - derived sepsis via fibrin polymerization and platelet - neutrophil interactions , J Thromb Haemost , 2017
blood to clot , which is useful for open wounds and other injuries to the body that cause bleedingcausesblood to clot , which is useful for open wounds and other injuries to the body that cause bleeding
The way in which stressed cells boost the production of the key blood clotting factor(passive) has been discoveredThe way in which stressed cells boost the production of the key blood clotting factor
the expected strong platelet aggregation and clot formation(passive) caused bythe expected strong platelet aggregation and clot formation
significant increases in the numbers ( 43 % ) , diameters ( 5 % ) and lengths ( 17 % ) , of both pre- and postcapillaries ( first - order vessels by centripetal ordering ) ... 11888679causedsignificant increases in the numbers ( 43 % ) , diameters ( 5 % ) and lengths ( 17 % ) , of both pre- and postcapillaries ( first - order vessels by centripetal ordering ) ... 11888679
calpain activation but not activation of caspases 3 and 7causedcalpain activation but not activation of caspases 3 and 7
to stable clot formationleadsto stable clot formation
the firm agglutination of aggregated platelets into an irreversible platelet plug and fibrin clotcausesthe firm agglutination of aggregated platelets into an irreversible platelet plug and fibrin clot
The increase in MLC phosphorylation(passive) caused byThe increase in MLC phosphorylation
p70S6 K translocation from the cytosol to the nucleuscausedp70S6 K translocation from the cytosol to the nucleus
a further decrease in the AA , compared with Gap27 alone ... N = 45causeda further decrease in the AA , compared with Gap27 alone ... N = 45
to higher risk of clot formationleadingto higher risk of clot formation
binary option system 964 activation and consumption(passive) caused bybinary option system 964 activation and consumption
fibrin to form a fibrin clotcausingfibrin to form a fibrin clot
the production of fibrin , the netting in blood clotsalso triggersthe production of fibrin , the netting in blood clots
in amounts produced by clotted whole bloodcausesin amounts produced by clotted whole blood
from platelets and endothelium lining of blood vesselsoriginatesfrom platelets and endothelium lining of blood vessels
TF expression on CD14high monocytestriggersTF expression on CD14high monocytes
a striking and time - dependent increase in Akt phosphorylationcauseda striking and time - dependent increase in Akt phosphorylation
the signaling cascade(passive) triggered bythe signaling cascade
increased monocytic - cell adhesion to endothelial cells through a protein - kinase C - dependent pathway , Biochemcausesincreased monocytic - cell adhesion to endothelial cells through a protein - kinase C - dependent pathway , Biochem
a concentration- and time - dependent increase in CTGF expression in WI-38 cells and primary lung fibroblastscauseda concentration- and time - dependent increase in CTGF expression in WI-38 cells and primary lung fibroblasts
clot formation by converting fibrinogen to fibrin , by converting factors V and VIII to their active forms and by activating plateletscan causeclot formation by converting fibrinogen to fibrin , by converting factors V and VIII to their active forms and by activating platelets