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Qaagi - Book of Why

Causes

Effects

10/27/2010 11.08 Annals of neurology Ciprofloxacinpreventsmyelination delay in neonatal rats subjected to E. coli sepsis

if colonizing the immature small intestine with Lactobacillus GG and the use of lactoferrin to promote the growth of LGGwould prevententeroinvasive E Coli ( EIEC ) infection in rat pups

Human Milk OligosaccharidespreventNecrotizing Enterocolitis in Neonatal Rats

Non - neutralizing antibodypreventsSIV infection in neonatal rhesus macaques ( RHMS

Prophylactic intratracheal polymyxin B / surfactantpreventsbacterial growth in neonatal Escherichia coli pneumonia of rabbits

peptides attenuates bacterial penetration through the BBBleadingto reduced E. coli meningitis in neonatal rats

The postbiotic HM0539 fromGGpreventsintestinal infection by The postbiotic HM0539 from Lactobacillus rhamnosus GG

a bacteria belonging to the genus staphylococcus,(passive) is caused byRelated ArticlesInfectious Bacterial Staph Infection in Rats Staphylococcal infection in rats

to exaggerated brain cytokine production to the LPS challenge in adulthoodleadsto exaggerated brain cytokine production to the LPS challenge in adulthood

a common diarrheal illness in childrencausesa common diarrheal illness in children

to persistenceleadsto persistence

in a dose - dependent increase in CSF levels of interleukin-1β , interleukin-6 , tumor necrosis factor α , interleukin-10 , and interferon - γ in acute diseaseresultedin a dose - dependent increase in CSF levels of interleukin-1β , interleukin-6 , tumor necrosis factor α , interleukin-10 , and interferon - γ in acute disease

brain injury(passive) caused bybrain injury

in a dose - dependent increase in CSF levels of interleukin-1ß , interleukin-6 , tumor necrosis factor α , interleukin-10resultedin a dose - dependent increase in CSF levels of interleukin-1ß , interleukin-6 , tumor necrosis factor α , interleukin-10

to marked HP - dependent memory deficits in adulthoodleadsto marked HP - dependent memory deficits in adulthood

PVL - like lesions in periventricular white matter , where the infiltration of inflammatory cells as well as the reduction of oligodendrocytes was observedcausedPVL - like lesions in periventricular white matter , where the infiltration of inflammatory cells as well as the reduction of oligodendrocytes was observed

The subtle signs of disease(passive) caused byThe subtle signs of disease

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