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a codon mutation(passive) caused bythe nonsynonymous substitution
the PLCG2 mutationcausingthe S707Y substitution
an amino acid sequenceresultingfrom the substitution
the mih1 - 1 mutation(passive) caused byThe substitution
n = 4 ) a mutationcausingthe substitution
a different string to useto triggerthe substitution
possible extra costsresultingfrom the substitution
the data wordresultingfrom the substitution
actual prejudiceresultingfrom the substitution
the termsresultfrom the substitution
amino acid sequencesresultingfrom the substitution
the gins2u773 allele as a likely functional null mutation(passive) caused bythe Gins2L52P substitution
the change in value of the instrumentwould ... resultfrom the substitution
either AAC or AAT allele(passive) can be caused byThe K013N substitution
when officers noticed insects eating all the bags of heroin(passive) was only discoveredThe substitution
the permissible judgment ... no prejudicewould resultfrom the substitution
to articulate any prejudice to their defenseresultingfrom the substitution
the OH ion that ’s a nucleophile(passive) is caused byThe substitution
the complicated expressionwill resultfrom the substitution
surcharges and other costsresultingfrom the substitution
any additional costsresultingfrom the substitution
Only the n - th instance of original - stringwill triggerthe substitution
followed by a spaceto triggerthe substitution
the substitution ... undue delay or prejudicewould resultfrom the substitution
Ending characterstriggerthe substitution
Yakima ... the timediscoveredthe substitution
epub the erythrocyte Keefe prompts the Meiniesto influencethe substitution
the retailer ... the damagesresultfrom the substitution
very little errorresultingfrom the substitution
no damage or lossresultingfrom the substitution
Such a " wild - type alleie/1 " wild - type AHASL allele , " or " wild - type AHASL gene ... the mutationcausesthe S653(Af)N substitution
ESR changesresultingfrom the substitution
Use i d myfunc(?a ) = myfunc(?ato triggerthe substitution
Use the EQUATION WRITERto createthe substitution
the use of lexical knowledgeto composethe substitution
the costs mentioned in article 6 concerning amendments and communication and any extra costsresultingfrom the substitution
a C - to - T transition at nucleotide 677 and producing an additional HinfI site(passive) created byThe Ala223Val substitution
Méliès , a prolific innovator in the use of special effectsaccidentally discoveredthe substitution stop
the Mx polymorphismcausingthe S631N substitution
pay a one - time $ 40.00 processing fee to the office via check or cash Additional instructions and an Addendumto createthe substitution
a nonsense variant , which changes a Glutamine to a premature stop codon ( CAA > TAA ) , and is predicted to cause loss of normal protein function through protein truncationcreatesa nonsense variant , which changes a Glutamine to a premature stop codon ( CAA > TAA ) , and is predicted to cause loss of normal protein function through protein truncation
a nonsense variant , which changes a Tryptophan to a premature stop codon ( TGG > TGAcreatesa nonsense variant , which changes a Tryptophan to a premature stop codon ( TGG > TGA
a nonsense variant , which changes a Tryptophan to a premature stop codon(TGG > TAGcreatesa nonsense variant , which changes a Tryptophan to a premature stop codon(TGG > TAG
a nonsense variant , which changes an Arginine to a premature stop codon ( CGA > TGAcreatesa nonsense variant , which changes an Arginine to a premature stop codon ( CGA > TGA
a nonsense variant , which changes an Arginine to a premature stop codon ( AGA > TGAcreatesa nonsense variant , which changes an Arginine to a premature stop codon ( AGA > TGA
a nonsense variant , which changes a Serine to a premature stop codon ( TCA > TAAcreatesa nonsense variant , which changes a Serine to a premature stop codon ( TCA > TAA
a nonsense variant , which changes a Glutamine to a premature stop codon ( CAA > TAA),and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNAdecaycreatesa nonsense variant , which changes a Glutamine to a premature stop codon ( CAA > TAA),and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNAdecay
a nonsense variant , which changes a Glutamine to a premature stop codon ( CAA > TAA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Glutamine to a premature stop codon ( CAA > TAA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Lysine to a premature stop codon ( AAA > TAAcreatesa nonsense variant , which changes a Lysine to a premature stop codon ( AAA > TAA
a nonsense variant , which changes a Glutamine to a premature stop codon ( CAG > TAG ) , and is predicted to cause loss of normal protein function through protein truncationcreatesa nonsense variant , which changes a Glutamine to a premature stop codon ( CAG > TAG ) , and is predicted to cause loss of normal protein function through protein truncation
a nonsense variant , which changes a Leucine to a premature stop codon ( TTG > TAGcreatesa nonsense variant , which changes a Leucine to a premature stop codon ( TTG > TAG
a nonsense variant , which changes a Leucine to a premature stop codon(TTA > TAAcreatesa nonsense variant , which changes a Leucine to a premature stop codon(TTA > TAA
a nonsense variant , which changes a Leucine to a premature stop codon ( TTA > TAGcreatesa nonsense variant , which changes a Leucine to a premature stop codon ( TTA > TAG
a nonsense variant , which changes a Glycine to a premature stop codon ( CAG > TAGcreatesa nonsense variant , which changes a Glycine to a premature stop codon ( CAG > TAG
a nonsense variant , which changes a Glutamic Acid to a premature stop codon ( GAA > TAAcreatesa nonsense variant , which changes a Glutamic Acid to a premature stop codon ( GAA > TAA
a nonsense variant , which changes a Glutamic Acid to a premature stop codon ( GAG > TAGcreatesa nonsense variant , which changes a Glutamic Acid to a premature stop codon ( GAG > TAG
a nonsense variant , which changes a Glutamine to a premature stop codon ( CAG > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Glutamine to a premature stop codon ( CAG > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
in a change to issuer , maturity date , par amount or coupon rate as specified in the previous confirmation ; ( Cresultsin a change to issuer , maturity date , par amount or coupon rate as specified in the previous confirmation ; ( C
in a change to issuer , maturity date , par amount or coupon rate specified in the previous confirmation ; ( iiiresultsin a change to issuer , maturity date , par amount or coupon rate specified in the previous confirmation ; ( iii
a nonsense variant , which changes an Isoleucine to a premature stop codon ( ATA > TAAcreatesa nonsense variant , which changes an Isoleucine to a premature stop codon ( ATA > TAA
a nonsense variant , which changes a Cysteine to a premature stop codon ( TGC > TGA),and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNAdecaycreatesa nonsense variant , which changes a Cysteine to a premature stop codon ( TGC > TGA),and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNAdecay
a nonsense variant , which changes a Leucine to a premature stop codon ( TTA > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Leucine to a premature stop codon ( TTA > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
anonsense variant , which changes a Tryptophan to a premature stop codoncreatesanonsense variant , which changes a Tryptophan to a premature stop codon
a nonsense variant , which changes a Cysteine to a premature stop codon ( TGC > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Cysteine to a premature stop codon ( TGC > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Serine to a premature stop codon ( TCA > TGA),and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNAdecaycreatesa nonsense variant , which changes a Serine to a premature stop codon ( TCA > TGA),and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNAdecay
a nonsense variant , which changes an Aspartic Acid to a premature stop codon ( GAT > TGAcreatesa nonsense variant , which changes an Aspartic Acid to a premature stop codon ( GAT > TGA
a nonsense variant , which changes a Serine to a premature stop codon ( TCA > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Serine to a premature stop codon ( TCA > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAG ) , and is predicted to cause loss of normal protein function through protein truncationcreatesa nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAG ) , and is predicted to cause loss of normal protein function through protein truncation
a nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAA ) , and is predicted to cause loss of normal protein function through protein truncationcreatesa nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAA ) , and is predicted to cause loss of normal protein function through protein truncation
anonsense variant , which changes a Cysteine to a premature stop codon ( TGC > TGAcreatesanonsense variant , which changes a Cysteine to a premature stop codon ( TGC > TGA
anonsense variant , which changes a Glutamine to a premature stop codon ( CAG > TAGcreatesanonsense variant , which changes a Glutamine to a premature stop codon ( CAG > TAG
anonsense variant , which changes a Leucine to a premature stop codon ( TTA > TGAcreatesanonsense variant , which changes a Leucine to a premature stop codon ( TTA > TGA
trigger - happy copy / paste and make it easier to change in the command - line version)Newer editto preventtrigger - happy copy / paste and make it easier to change in the command - line version)Newer edit
a nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Tyrosine to a premature stop codon ( TAT > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Tyrosine to a premature stop codon ( TAT > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Tyrosine to a premature stop codon ( TAT > TAA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Tyrosine to a premature stop codon ( TAT > TAA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Tyrosine to a premature stop codon ( TAC > TAA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
a nonsense variant , which changes a Lysine to a premature stop codon ( AAG > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Lysine to a premature stop codon ( AAG > TAG ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay
in an angle of incident that depends only on the refractive indices of the analyte , the core layer and the outside world : γ ″ = arcsin ( n ′ n ″ arccosresultsin an angle of incident that depends only on the refractive indices of the analyte , the core layer and the outside world : γ ″ = arcsin ( n ′ n ″ arccos
a nonsense variant , which changes a Glycine to a premature stop codon ( GGA > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decaycreatesa nonsense variant , which changes a Glycine to a premature stop codon ( GGA > TGA ) , and is predicted to cause loss of normal protein function through either protein truncation or nonsense - mediated mRNA decay