that in neurons expressing polyQ - huntingtin there was reduced GABA - A receptor traffickingresultingin reduced synaptic inhibition
Furthermore , during CF activation EAAT1 controls the spread of glutamate to nearby post - synaptic NMDA receptors on the PN spine and also to presynaptic NMDA receptors on GABAergic terminalsinfluencinglocal synaptic inhibition
alpha-2 receptorscausingpre - synaptic inhibition
These manipulations ... seizures and deathresultfrom decreased synaptic inhibition
this mechanismcontributesto the homeostasis of synaptic inhibition
by prenatal exposure to moderate levels of ethanol(passive) caused byhippocampal synaptic inhibition
GABA release from presynaptic terminalscontributesto synaptic inhibition
calcium and potassium influxcausesneuronal inhibition
the synapsecausingneuronal inhibition
TMScausesneuronal inhibition
by GABA A(passive) caused bysynaptic inhibition
a reduction in the probability of action potential initiationcausingneuronal inhibition
by GSK-3(passive) caused bypathway inhibition
A reduction in the probability of action potential generationcausesneuronal inhibition
by the tramadol(passive) caused byreuptake inhibition
High dosescan causeCox-1 inhibition
to epilepsyleadsto epilepsy
to a phenotype of increased anxiety - like behaviorcan leadto a phenotype of increased anxiety - like behavior
to a decrease in neurological deficit scoresledto a decrease in neurological deficit scores
to the epileptic , behavioral , and cognitive phenotypes of thedisordercould contributeto the epileptic , behavioral , and cognitive phenotypes of thedisorder
regression of LVH(passive) may be caused byregression of LVH
to an increased neuronal excitationleadsto an increased neuronal excitation
plasticity of excitatory synapses at least indirectly through changes in activityinfluencesplasticity of excitatory synapses at least indirectly through changes in activity
the changes in transmissionmight have causedthe changes in transmission
to the tachycardiacontributesto the tachycardia
from the higher sensitivity of these receptorsresultingfrom the higher sensitivity of these receptors
the sympathetic nervous systemcausesthe sympathetic nervous system
changes in CBF to excitatory input(passive) are ... influenced bychanges in CBF to excitatory input
to disinhibition of pain regulatory pathwaysleadsto disinhibition of pain regulatory pathways
in weakness of the muscleresultsin weakness of the muscle
to hyperexcitation of the central nervous system , convulsions , and deathleadsto hyperexcitation of the central nervous system , convulsions , and death
The pain(passive) can be caused byThe pain
The pain(passive) might be caused byThe pain
alsomay ... contributealso
in weakness of the muscle Once swellingresultsin weakness of the muscle Once swelling
tumor regressioncan causetumor regression
to neuronal hyperexcitability in the spinal cord and the thalamusleadingto neuronal hyperexcitability in the spinal cord and the thalamus
to hyperexcitation of the central nervous systemleadsto hyperexcitation of the central nervous system
accumulation of rMaspincausesaccumulation of rMaspin
Rhythm generation in mature respiratory networks(passive) is influenced strongly byRhythm generation in mature respiratory networks
in muscle weaknessresultingin muscle weakness
to hyperexcitation of the central nervous system , convulsions , andleadsto hyperexcitation of the central nervous system , convulsions , and
Particularly fast rhythmic activity in the beta and gamma range is thought(passive) to be caused byParticularly fast rhythmic activity in the beta and gamma range is thought
The discomfort(passive) can be caused byThe discomfort
to re injury in hamstring strainscan leadto re injury in hamstring strains
the temporal coding properties of medial superior olivary neurons : an in vitro studyinfluencesthe temporal coding properties of medial superior olivary neurons : an in vitro study
a partial or acute loss of kinase activity(passive) caused bya partial or acute loss of kinase activity
in marked accumulation of p62 , contrary to RAPA treatment ( Figure 1Cresultedin marked accumulation of p62 , contrary to RAPA treatment ( Figure 1C
to both apoptosis and differentiationledto both apoptosis and differentiation
to suppression of tumor growth and metastasisleadingto suppression of tumor growth and metastasis
sudden deathcausessudden death
to its analgesic efficacycontributeto its analgesic efficacy
to the same extent as the release of neurotransmittersdoes ... causeto the same extent as the release of neurotransmitters