does not tendto causeperipheral sympathetic stimulation
I do tendto causeperipheral sympathetic stimulation
members of its potentialto causeperipheral sympathetic stimulation
does n't tendto causeperipheral sympathetic stimulation
well MODAFINIL does n't tendto causeperipheral sympathetic stimulation
a tendencyto causeperipheral sympathetic stimulation
by exposure to cool(passive) caused bysympathetic anxious stimulation
his MODAFINIL ... does n't tendto causeperipheral sympathetic stimulation
long - acting MODAFINIL does n't tendto causeperipheral sympathetic stimulation
the intermittent exceeding 4 h ) that provide relatively more constant stimulation of striatal dopamine ( DA ) receptorscontributesdopaminergic stimulation
Normal regulation of the heart Extrinsic Factors * Neural and Hormonal control of the heartcausesSympathetic stimulation
The Bainbridge reflex ... the heartcausessympathetic nervous system stimulation
drugs and eventsmay leadto sympathetic stimulation
by exercise training(passive) caused bysympathetic stimulation
Larger increases in heart rate , especially during exerciseresultfrom sympathetic stimulation
stressleadsto sympathetic stimulation
by the pain(passive) caused bythe sympathetic stimulation
Paincausessympathetic stimulation
by pain(passive) caused bySympathetic stimulation
by an increase in CO2(passive) caused bysympathetic stimulation
The neuronal pathwayleadingto the sympathetic stimulation
conditionscausingsympathetic stimulation
the catecholamines of stress statescausesympathetic stimulation
severe constriction of the blood vesselscausessevere constriction of the blood vessels
severe constrictioncausessevere constriction
to vasoconstriction leading to increase in blood pressureleadingto vasoconstriction leading to increase in blood pressure
release of norepinephrine to alpha - adrenergic receptorscausesrelease of norepinephrine to alpha - adrenergic receptors
severe deficiencycan causesevere deficiency
constipationcausesconstipation
in myocardial dysfunctioncan resultin myocardial dysfunction
to contraction of theleadsto contraction of the
to an increase in systemic vascular resistanceleadingto an increase in systemic vascular resistance
to increased levels of catecholamineleadingto increased levels of catecholamine
to respiratory arrest and clinical signs and symptoms , such as fever , nausea , vomiting , and hemodynamic rhythmsmay leadto respiratory arrest and clinical signs and symptoms , such as fever , nausea , vomiting , and hemodynamic rhythms
secretioncausessecretion
vasoconstrictioncausesvasoconstriction
an increase in cardiac output , heart rate , vasoconstriction , and a decrease in digestive activitycausesan increase in cardiac output , heart rate , vasoconstriction , and a decrease in digestive activity
in an increased flow and parasympathetic stimulation in a reduced flowresultingin an increased flow and parasympathetic stimulation in a reduced flow
an increase in stroke volume , an increase in cardiac output , and systemic vasodilationwill causean increase in stroke volume , an increase in cardiac output , and systemic vasodilation
the muscle to relax , and parasympathetic stimulation causes the muscle to contractcausesthe muscle to relax , and parasympathetic stimulation causes the muscle to contract
an increase in contractions of the heart and an increase in heart ratecausesan increase in contractions of the heart and an increase in heart rate
vasoconstriction of arterioles to increase systemic blood pressurecan causevasoconstriction of arterioles to increase systemic blood pressure
inhibition of gastrointestinal secretion and motor activity ... and contraction of gastrointestinal sphincters and blood vesselscausesinhibition of gastrointestinal secretion and motor activity ... and contraction of gastrointestinal sphincters and blood vessels
vasoconstriction and increases the resulting blood pressurecausesvasoconstriction and increases the resulting blood pressure
to an increase in myocardial contractility and heart rateleadsto an increase in myocardial contractility and heart rate
to increased excitability in all cellsleadsto increased excitability in all cells
the heart rate to increasecausesthe heart rate to increase
the release of the neurotransmitter norepinephrine at the neuromuscular junction of the cardiac nervescausesthe release of the neurotransmitter norepinephrine at the neuromuscular junction of the cardiac nerves
tachycardia , increased cardiac contractility , and renal fluid retentioncausestachycardia , increased cardiac contractility , and renal fluid retention
in an increase in systolic pressure which increases oxygen demandresultin an increase in systolic pressure which increases oxygen demand
cardiac depression and decreasecausescardiac depression and decrease
to a reduction in resting heart rate , cardiac output , systolic and diastolic blood pressure and reflex orthostatic hypotensionleadingto a reduction in resting heart rate , cardiac output , systolic and diastolic blood pressure and reflex orthostatic hypotension
in a reduction in resting heart rate , cardiac output , systolic and diastolic blood pressure , and reflex orthostatic hypotensionresultsin a reduction in resting heart rate , cardiac output , systolic and diastolic blood pressure , and reflex orthostatic hypotension
in reduction in resting heart rate , systolic and diastolic blood pressure ... cardiac output and reflex orthostatic hypotensionresultsin reduction in resting heart rate , systolic and diastolic blood pressure ... cardiac output and reflex orthostatic hypotension
the release of renin which , by stimulating aldosterone secretion , will increase the reabsorption of sodiumalso causesthe release of renin which , by stimulating aldosterone secretion , will increase the reabsorption of sodium