loss of SHANK3leadsto impaired synaptic transmission
a previous study on the developing visual cortex , in which PSA depletion induced precocious maturation of perisomatic innervation by basket interneuronsresultingin enhanced inhibitory synaptic transmission
One or all of these effects on inhibitory GABA - mediated ( GABAergic ) andmay contributeexcitatory glutamatergic synaptic transmission
SHANK3 deficiency is knownto causeimpairments in synaptic transmission
Manipulation of PSCs G - coupled protein receptorsleadto altered synaptic transmission
The subunit composition of synaptic NMDA receptors ( NMDAR ) , such as the relative content of GluN2A- and GluN2B - containing receptorsgreatly influencesthe glutamate synaptic transmission
a series of biochemical eventscontributeto the potentiation of synaptic transmission
a series of eventsleadto propagation of the synaptic transmission
a fearleadsto potentiation of synaptic transmission
2010 Isoflurane - sensitive presynaptic R - type calcium channelscontributeto inhibitory synaptic transmission
the mechanisms by whichcontributesto afferent synaptic transmission
some evidencemay ... influenceglutamatergic synaptic transmission
GABA transporter GAT1 ... where it is thoughtto influenceGABAergic synaptic transmission
these eventsmight contributeto evoked synaptic transmission
The predominant neuronal GABA transporter GAT1 ... where it is thoughtto influenceGABAergic synaptic transmission
the ability of endogenous dopamine , present in slices containing the prefrontal cortexto influenceexcitatory synaptic transmission
In presynaptic terminals , the channels are believedto influencesynaptic transmission
its physiological activation and the mechanisms by whichinfluencessynaptic transmission
by the loss of dopamine(passive) caused bysynaptic transmission
voltage - gated calcium channelscontributingto synaptic transmission
Mechanism for modulation of nicotinic acetylcholine receptorscan influencesynaptic transmission
the synaptic vesicles to fuse and release neurotransmitterscausesthe synaptic vesicles to fuse and release neurotransmitters
in chronic central neuropathic painresultsin chronic central neuropathic pain
in the release of neurotransmittersresultsin the release of neurotransmitters
to the LTP deficit in mutant micemay be contributingto the LTP deficit in mutant mice
to spinal sensitization that at least in part attributes to behavioral hyperalgesia and allodynia during chronic painto contributeto spinal sensitization that at least in part attributes to behavioral hyperalgesia and allodynia during chronic pain
to a better understanding of neuronal communicationshould leadto a better understanding of neuronal communication
from loss of sytresultingfrom loss of syt
the chemical balanceinfluencingthe chemical balance
in learning and memory deficitsresultsin learning and memory deficits
to aberrant firing of globusleadsto aberrant firing of globus
to several neurologic disorderscontributeto several neurologic disorders
muscle contractionscausesmuscle contractions
to aberrant firing of globus pallidus neuronsleadsto aberrant firing of globus pallidus neurons
at least in part ... to the loss of these neuronal populations [ 53 , 54may contributeat least in part ... to the loss of these neuronal populations [ 53 , 54
muscle contractions and is involved in learning and memory causesmuscle contractions and is involved in learning and memory
in the inhibition of the release of nociceptive neurotransmittersto resultin the inhibition of the release of nociceptive neurotransmitters
to seizure activitycontributedto seizure activity
axon - sprouting defects in a subset of GABAergic neuronscausesaxon - sprouting defects in a subset of GABAergic neurons
to these processescan contributeto these processes
to a disinhibition of network activityleadingto a disinhibition of network activity
from fast - spikingoriginatingfrom fast - spiking
from fast - spiking but not from somatostatin - positive interneuronsoriginatingfrom fast - spiking but not from somatostatin - positive interneurons
from the eyeoriginatingfrom the eye
to human ASDsmay contributeto human ASDs
a dendrite(passive) is caused bya dendrite
to aberrant firing of globus pallidus neurons in a rat model of Parkinson 's diseaseleadsto aberrant firing of globus pallidus neurons in a rat model of Parkinson 's disease
a dendrite(passive) is caused bya dendrite
in LTPwould resultin LTP
the predominant phenomenon(passive) caused bythe predominant phenomenon
to human ASDs and that the R451C knockin mice may be a useful model for studying autism - related behaviorsmay contributeto human ASDs and that the R451C knockin mice may be a useful model for studying autism - related behaviors
to AD associated synaptic dysfunctionleadingto AD associated synaptic dysfunction
to aberrant firing of globus pallidus neurons ileadsto aberrant firing of globus pallidus neurons i
from the near - simultaneous activity of multiple pre and postsynaptic neuronsresultingfrom the near - simultaneous activity of multiple pre and postsynaptic neurons
to decreased neuronal excitabilityleadsto decreased neuronal excitability
to enhanced postsynaptic responsesleadingto enhanced postsynaptic responses
postsynaptic cellsto influencepostsynaptic cells
behavioral outputto influencebehavioral output
the postsynaptic potential(passive) caused bythe postsynaptic potential
in an inhibitory postsynaptic potentialresultsin an inhibitory postsynaptic potential
in an excitatory postsynaptic potentialresultsin an excitatory postsynaptic potential