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Qaagi - Book of Why

Causes

Effects

the loss of one copy of the gene Shank3(passive) caused byexcitatory synaptic transmission

presynaptic calcium influx(passive) is triggered by18602451 Synaptic transmission

The predominant neuronal GABA transporter GAT1 ... where it is thoughtto influenceGABAergic synaptic transmission

elevating postsynaptic parkin(passive) caused byexcitatory synaptic transmission

Changes in PSD scaffolding protein contentmay influenceexcitatory synaptic transmission

drugsinfluenceneuronal synaptic transmission

Postsynaptic NMDA receptorscontribute significantlyto retinogeniculate synaptic transmission

/ kainate receptorsinfluenceexcitatory synaptic transmission

endogenous ET-1may ... influencesynaptic transmission in the NTS

the potentialto strongly influenceafferent synaptic transmission

Here we have explored the ability of endogenous dopamine , present in slices containing the prefrontal cortexto influenceexcitatory synaptic transmission

SHANK3 deficiency is knownto causeimpairments in synaptic transmission

drug actionsinfluencingGABAergic synaptic transmission

AD - related presenilin mutation ( 3(passive) caused byexcitatory synaptic transmission

exocytosis of the acidic contents of synaptic vesicles(passive) is caused byexcitatory synaptic transmission

gene mutation or environmental toxicantinfluencessynaptic transmission at all

the loss of proteins at excitatory synaptic inputs(passive) caused byglutamatergic synaptic transmission

nociceptin receptor activation ( Wigström and Gustafsson , 1983(passive) caused byinhibitory synaptic transmission

50 μm ) were included in the extracellular bathing solutionto preventexcitatory synaptic transmission

d - AP-5 ; 10–20 μm ) receptor antagoniststo preventexcitatory synaptic transmission

transmitter release(passive) are triggered byExcitatory synaptic transmission

enhanced sensory input(passive) triggered byexcitatory synaptic transmission

disruptions in a specific synaptic proteinmight influenceaberrant synaptic transmission

Altered ubiquitination of these substrates when Nedd4 - 2 ’s function is compromisedcould contributeto aberrant synaptic transmission

The delayed and deficient maturation of astrocytes might , in parallel , lead to disruption of glutamatergic , potassium , and neuromodulatory homoeostasisresultingin dysregulated synaptic transmission

the APP cleavage product Aβinfluencessynaptic transmission at the postsynapse

the NIS applicants(passive) discovered recently byglutamatergic synaptic transmission

As a result , the decreased synaptic strength due to TRIAD3A knockdown in neurons could not be rescued by the TRIAD3A missense variantsthereby causingaberrant synaptic transmission

The ACSF included picrotoxin ( 50 μm ) during all recordingsto preventinhibitory synaptic transmission

1994 ; Benson and Cohen , 1996 ) , has been shown to result in complete suppression of action potentials ( Craig et al . , 1994thereby preventingevoked synaptic transmission

several factorsinfluencingGABAergic synaptic transmission

these eventsmight contributeto evoked synaptic transmission

CACNA1A ( XP 176influencesglutamatergic synaptic transmission

View at PubMedView at PublisherSeverely impairedcausesneuromuscular synaptic transmission

more inhibition to the second pyramidal cellcausesmore inhibition to the second pyramidal cell

to human ASDs and that the R451C knockin mice may be a useful model for studying autism - related behaviors.[less ] PMID : 17823315 Mesh Headingsmay contributeto human ASDs and that the R451C knockin mice may be a useful model for studying autism - related behaviors.[less ] PMID : 17823315 Mesh Headings

from fast - spiking but not from somatostatin - positive interneurons — University of Texas Southwestern Medical Center Jay R. Gibson , Kimberly M. Huberoriginatingfrom fast - spiking but not from somatostatin - positive interneurons — University of Texas Southwestern Medical Center Jay R. Gibson , Kimberly M. Huber

in LTP.[7would resultin LTP.[7

from fast - spiking but not from somatostatin - positive interneuron 'soriginatingfrom fast - spiking but not from somatostatin - positive interneuron 's

subsequent LTP inductionpreventssubsequent LTP induction

in LTP.[8 ... Timeline of consolidation[editwould resultin LTP.[8 ... Timeline of consolidation[edit

the disordered , synchronous firing of neurons that underlies a seizurecan triggerthe disordered , synchronous firing of neurons that underlies a seizure

long - term potentiation ( LTP ) at C - fiber synapses and hyperalgesia [ 45triggeringlong - term potentiation ( LTP ) at C - fiber synapses and hyperalgesia [ 45

the sensitivity of postsynaptic kainate receptorsinfluencingthe sensitivity of postsynaptic kainate receptors

cellular events over a wide rangeto influencecellular events over a wide range

to increased nociceptive output by facilitating the input of primary binary options on autopilot neurons onto nociceptive otions in the dorsal horn , or between dorsal horn neurons themselvesleadsto increased nociceptive output by facilitating the input of primary binary options on autopilot neurons onto nociceptive otions in the dorsal horn , or between dorsal horn neurons themselves

to an increase in the postsynaptic Ca(2 + ) concentration that is believed to activate the signaling pathways that shifts the rhythmic expression of circadian clock genesleadsto an increase in the postsynaptic Ca(2 + ) concentration that is believed to activate the signaling pathways that shifts the rhythmic expression of circadian clock genes

to an increase in the postsynaptic Ca2 + concentration that is believed to activate the signaling pathways that shifts the rhythmic expression of circadian clock genesleadsto an increase in the postsynaptic Ca2 + concentration that is believed to activate the signaling pathways that shifts the rhythmic expression of circadian clock genes

cortical signalmay influencecortical signal

neuronal output(passive) caused byneuronal output

to excessive neuronal synchronization in the epileptogenic zone ( EZ ) of the braincan leadto excessive neuronal synchronization in the epileptogenic zone ( EZ ) of the brain

File Select item 192891371.Excitatory and(passive) is ... influencedFile Select item 192891371.Excitatory and

Related citations Select item 1928913719.Excitatory and(passive) is ... influencedRelated citations Select item 1928913719.Excitatory and

to these processescan contributeto these processes

to the propagation of the high [ Ca2+]i region ... see Discussioncontributedto the propagation of the high [ Ca2+]i region ... see Discussion

to seizure activitycontributedto seizure activity

four decades agowas discoveredfour decades ago

in increased dopamine releasewill resultin increased dopamine release

to excitotoxicity " Type 2 : Associated with procaine penicillin , sulfonamides , fluoroquinolones , and macrolidesStarts within days of the first antibiotic dose and resolves within a few daysFrequentlyleadingto excitotoxicity " Type 2 : Associated with procaine penicillin , sulfonamides , fluoroquinolones , and macrolidesStarts within days of the first antibiotic dose and resolves within a few daysFrequently

in the development of an ataxic phenotype in mice [ 48resultingin the development of an ataxic phenotype in mice [ 48

to diminished inhibition ( ∼50 % ) in neuronal circuitsleadingto diminished inhibition ( ∼50 % ) in neuronal circuits

in SAresultsin SA

to autonomic failureleadingto autonomic failure

from 7-day corticosterone treatmentresultingfrom 7-day corticosterone treatment

the flow of information through the neural networkinfluencesthe flow of information through the neural network

to granule cell hyperexcitability in slice culturesmay contributeto granule cell hyperexcitability in slice cultures

from electrical stimulation of presumed GP efferentsresultingfrom electrical stimulation of presumed GP efferents

the “ virtual ” absence of activation when it is measured extracraniallymay causethe “ virtual ” absence of activation when it is measured extracranially

autism spectrum disorders ( ASDs ) ( 10may causeautism spectrum disorders ( ASDs ) ( 10

from dopamine depletionresultingfrom dopamine depletion

a higher excitability(passive) caused bya higher excitability

7.6 pA / pF. Spontaneous IPSCs(passive) caused by7.6 pA / pF. Spontaneous IPSCs

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