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> chemicalscausestrand break formation
the well known chemicalscausestrand break formation
key system orderto causea strand break
forces on DNAcan causestrand breakage
the damagecausesstrand breakage
by heat(passive) caused bystrand breakage
which can damage intracellular DNAcausingstrand breakage
the DNAcausesstrand breakage
baseresultingin strand break
in turnwill causestrand breakage
orderto causea strand break
especially after repeated applicationsmight causestrand breakage
by bending(passive) caused bystrand breakage
bending(passive) caused bystrand breakage
sufficientto causebreakage of the strand
It cross - links DNAcausesstrand breakage
shown to crosslink DNAcausingstrand breakage
by X - irradiation(passive) caused bythe strand break
free oxygen radicalscausestrand breakage
It mix - links DNAcausesstrand breakage
by hydroxyl radical and UV(passive) caused bystrand breakage
the DNA synthesis pathwaycausesstrand breakage
by hydroxyl radicals or UV irradiation(passive) caused bystrand breakage
by radical attack or UV radiation(passive) caused bystrand breakage
by hydroxyl radical and UV irradiation(passive) caused bystrand breakage
hydroxyl radical and UV irradiation(passive) caused bystrand breakage
This molecule ... the DNA synthesis pathwaycausesstrand breakage
online legal the DNA synthesis pathwaycausesstrand breakage
Molecular repair mechanisms targeting this damage stretch of the DNA , damage due to mispairing and/or imidazole ring openingmay causestrand breakage
These ROSthen causestrand breaks in DNA
Increased respiration ... oxidative stresscan causestrand breaks in DNA
the DNAcausingsingle strand breaks
ionizing radiationcausesstrand breaks in DNA ...
ROS attack to DNA sugarsleadsto single - strand breaks
has a pleiotropic mechanism of action where it targets topoisomerase IIcausingstrand breaks in DNA
Since tobacco is knownto causesingle strand breaks
Stable covalent BaP DNA adductscan causesingle - strand breaks
peroxynitrite(passive) caused bysingle - strand breaks
the ionizing radiations(passive) provoked bythe single - strand breaks
reactive oxygen species ( ROS ) ( 3(passive) caused primarily bysingle strand breaks
to apoptosis and cell deathleadsto apoptosis and cell death
if not resulting in cell deathmay leadif not resulting in cell death
in apoptosisresultingin apoptosis
double - strand breakscausesdouble - strand breaks
to DSBs during replication and these DSBscan leadto DSBs during replication and these DSBs
to to DSBs during replicationcan contributeto to DSBs during replication
to DSBs during replicationcan leadto DSBs during replication
to DSBscan leadto DSBs
to DSBscan contributeto DSBs
from inhibition of repair enzymes at levels of 5 to 100 mm in human epithelial type ii cellsresultingfrom inhibition of repair enzymes at levels of 5 to 100 mm in human epithelial type ii cells
to gene mutations that cause cancercan ... leadto gene mutations that cause cancer
to ataxia telangiectasia mutated ( ATM ) phosphorylationleadto ataxia telangiectasia mutated ( ATM ) phosphorylation
immediately after IR treatmentresultingimmediately after IR treatment
chromosome breakage , which can contribute to genetic disorders and intergenerational diseasecan causechromosome breakage , which can contribute to genetic disorders and intergenerational disease
, one after the othermay result, one after the other
alsomay ... resultalso
to cell deathleadingto cell death
to carcinogenesis , mutagenesis and cytotoxicityleadingto carcinogenesis , mutagenesis and cytotoxicity
to carcinogenesis , mutagenesis and cytotoxicitycontributeto carcinogenesis , mutagenesis and cytotoxicity
to carcinogenesis , mutagenesis , and cytotoxicitycontributesto carcinogenesis , mutagenesis , and cytotoxicity
to carcinogenesis , mutagenesis and cytotoxicity ... 26].contributesto carcinogenesis , mutagenesis and cytotoxicity ... 26].
in genome instability and tumor formation during agingcan resultin genome instability and tumor formation during aging
to non - homologous endleadingto non - homologous end
to chromosome rearrangementscould leadto chromosome rearrangements
to cell mutationsleadingto cell mutations
to cell deathleadsto cell death
in cell deathwill resultin cell death
in cell deathresultin cell death
to the cell deathleadingto the cell death
to cell deathleadingto cell death
in cell cycle arrest and [ [ apoptosisresultin cell cycle arrest and [ [ apoptosis
to the rearrangement of DNAcan leadto the rearrangement of DNA
to oncogenic translocation and gene amplificationleadingto oncogenic translocation and gene amplification
to cancerleadingto cancer
to cell death , mutation , or carcinogenesiscan leadto cell death , mutation , or carcinogenesis
genome rearrangements , such as deletions , duplications , and translocations , which have been implicated in carcinogenesismay causegenome rearrangements , such as deletions , duplications , and translocations , which have been implicated in carcinogenesis
in mutationsresultingin mutations
to mutationscan leadto mutations
to activation of the immune system either through increased neoantigens or activation of the STING pathwaycan leadto activation of the immune system either through increased neoantigens or activation of the STING pathway
to chromosomalcan leadto chromosomal