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Muñoz García A , Abarca Costalago M , Hierro Martín I , Valdivielso Felices P. Splenic arterial dissection and factor V Leiden mutationcausingmassive spleen infarction
infection(passive) triggered byspleen infarct
aortic dissection , torsion of splenic artery or compression of the artery from a tumor(passive) can be caused byInfarction of spleen
a drugcausesa spleen infarction
vesselscausingischemia of spleen
clot(passive) caused bya spleen infarction
due(passive) can be causedInfarction of spleen
the vascular pedicle torsion(passive) caused bya spleen infarction
Its diagnosis should be made in prompt timeto preventinfarction of the spleen
thrombosis(passive) caused bya spleen infarction
Pbx1 inactivation in Nkx2 - 5-positive mesenchymecausesspleen hypoplasia
still do n’t knowcausedthe spleen infarction
Because of the nature of the disease and its effects on the cells , the spleen becomes engorged multiple timescausinginfarction of the spleen
TH via TRα during early postnatal development(passive) is influenced byspleen erythropoiesis
acute abdominal paincan causeacute abdominal pain
in auto spleenectomy and is associated with immunodeficiency 74resultsin auto spleenectomy and is associated with immunodeficiency 74
internal bleeding ... and if medical intervention is not taken then spleen can ultimately lead to deathcausesinternal bleeding ... and if medical intervention is not taken then spleen can ultimately lead to death
to abnormal stress hematopoiesisleadsto abnormal stress hematopoiesis
from Pbx1 inactivation in spleen mesenchymal progenitorsresultsfrom Pbx1 inactivation in spleen mesenchymal progenitors
hemolytic anemia(passive) caused byhemolytic anemia
in the most controversial red blood cell aggregation findingsresultedin the most controversial red blood cell aggregation findings
erythrocyte aggregation parameterscontroversially influenceserythrocyte aggregation parameters
stomachache(passive) caused bystomachache
from a Tlx1 ( Hox11)-impartial proliferation defectresultingfrom a Tlx1 ( Hox11)-impartial proliferation defect
from a Tlx1 ( Hox11)-independent proliferation defectresultingfrom a Tlx1 ( Hox11)-independent proliferation defect