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by cells(passive) is caused bymajor signaling molecules
its abilityto influencemultiple signaling molecules
depotsdesignedto hold signaling molecules
Botanical physiologistshave discoveredsignalling molecules called turgorins
EBV(passive) caused bycellular signaling molecules
by the dopamine spatial gradient(passive) caused byintracellular signaling molecules
the approach ... ORto setsignaling molecules
possible paincausingsignaling molecules
the SMAD3 geneinfluencessignaling between molecules
impaired generation of MVBs(passive) caused bysignaling molecules
by impaired generation of MVBs(passive) caused bysignaling molecules
to target the transcripts of resistance genes(passive) can be designedOligonucleotide molecules
based on the sequence of the gene of interest ( target nucleic acid(passive) can be designedRNAi molecules
based on the sequence of the gene of interest(passive) can be designedRNAi molecules
Science Fighting cancer with immunotherapycausesSignaling molecule
to be activated by proteases with known overexpression in the tumor environment(passive) are designedProTIA molecules
Mutations in receptors and/ormay causesignal transduction molecules
based on the sequence of(passive) can be designedAntisense molecules
to reduce(passive) are designedRNAi molecules
to target and turn off specific gene products that contribute to various diseases(passive) to be designedRNAi molecules
to reduce elevated lipoprotein(a(passive) are designedRNAi molecules
to target and shut down specific gene products that contribute to various diseases(passive) may be designedRNAi molecules
to target and shut down specific gene products that contribute to several diseases(passive) can be designedRNAi molecules
These cellcan influencesignalling molecules
an evolutionarily conserved neuromodulatory systemcomposedof the endocannabinoid signalling molecules
that prevent molecules such as cytokines from triggering inflammation(passive) can be designedEffector molecules
changes within the cellinfluenceother signalling molecules
by excessive carbohydrates(passive) caused bylipoprotein molecules
to bind to a specific target(passive) are designedCOBRATM molecules
UV radiationcausesriboflavin molecules
of 8 cysteine residues(passive) are composedHepcidin molecules
of 8 cysteine residues(passive) are composed Hepcidin molecules
of two chains of amino acids , one of which is connected to the actin filaments in the cytoskeleton(passive) are composedIntegrin molecules
by artificial intelligence(passive) designed bybioactive molecules
when acting or inhibiting VOCs , cells or sulfatidesdesigningtriphosphatase molecules
to malignant transformationcontributeto malignant transformation
inflammatory damagecauseinflammatory damage
cytokine response of hostmay influencecytokine response of host
in apoptotic eventsresultin apoptotic events
to the regulation of peripheral B - cell development and antibody responsescontributesto the regulation of peripheral B - cell development and antibody responses
the activity of the brain and the body in specific waysinfluencethe activity of the brain and the body in specific ways
to the dysregulation of vascular growth factors and cytokines , such as vascular endothelial growthleadto the dysregulation of vascular growth factors and cytokines , such as vascular endothelial growth
the activity of the braininfluencethe activity of the brain
in an increase in Ca 2 + concentration in the cytosolresultin an increase in Ca 2 + concentration in the cytosol
to differentiation and the development of particular structuresleadto differentiation and the development of particular structures
in inhibition of NF - B activation and proinflammatory cytokine productionresultingin inhibition of NF - B activation and proinflammatory cytokine production
to caspase 1 activation in macrophages in response to infection bycontributeto caspase 1 activation in macrophages in response to infection by
the symptomscausethe symptoms
this processinfluencethis process
to inflammation , proliferative responses , or apoptosismay leadto inflammation , proliferative responses , or apoptosis
in different reactions from the cellto resultin different reactions from the cell
to decreased function and increased cytotoxicityleadto decreased function and increased cytotoxicity
to the stability of a communitycan contributeto the stability of a community
in other bone cell types on target cellsoriginatingin other bone cell types on target cells
cell growth , apoptosis , and inflammationto influencecell growth , apoptosis , and inflammation
off cell growthsetoff cell growth
to induction of stress responsesleadingto induction of stress responses
blood vessels to grow toward the tumorcauseblood vessels to grow toward the tumor
blood vessels to grow toward tumorcauseblood vessels to grow toward tumor
to dangerous reactionscan ... leadto dangerous reactions
gene expression in interacting cellsinfluencegene expression in interacting cells
in differentiation and proliferationultimately resultingin differentiation and proliferation
to activationleadingto activation
to physiological wound repair and the development of tissue fibrosiscontributeto physiological wound repair and the development of tissue fibrosis
of three subunits , alpha , beta , and gammacomposedof three subunits , alpha , beta , and gamma
cell division , apoptosis , and migrationto influencecell division , apoptosis , and migration
to changes in protein expression in the nucleus that stimulates growthleadto changes in protein expression in the nucleus that stimulates growth
the recipient cell phenotypesto influencethe recipient cell phenotypes
to attenuation of ligand - induced responsesleadingto attenuation of ligand - induced responses
immune reactionscauseimmune reactions
the pain signal to increase and fire more frequentlycausingthe pain signal to increase and fire more frequently
the surrounding non - malignant cells that are collectively known as stroma or tumor microenvironmentinfluencethe surrounding non - malignant cells that are collectively known as stroma or tumor microenvironment
to neuronal degenerationcontributeto neuronal degeneration
the apoptosis of DCs in the tumor environmentto causethe apoptosis of DCs in the tumor environment
patterns of gene expressionto influencepatterns of gene expression