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GIV 's abilityto triggerGαi signaling
The ability of bacterial 23S rRNAto triggerTLR13 signaling
Ruf W. Endogenous EPCR / aPC - PAR1preventssignaling
so fusion to DsRedcould causeconstitutive signaling
nonmetabolizable d - amino acidstriggersignaling
a complex with ligandsto preventsignaling
the binding of IL-1α or IL-1β to IL-1R1(passive) is originated bysignaling
shape , which would result in a change in conformation of TLR4to triggersignaling
Jun 2015 Updates ... the three critical cell surface complexes , Wnt - Fzd - LRP6 , Dkk1-Kremen - LRP6 , and R - spondin - LGR5-RNF43influenceWnt signaling
the boundary ... the surrounding cellstriggeredsignaling
Wang J. A direct cross - talk between interferon - gamma and sonic hedgehogleadssignaling
Notch / Rbp - jpreventssignaling
PV - IgG binding to Dsg3(passive) caused bysignaling
Lancet 2005 March 16;365(9464):1054 - 1061 James C , Ugo V , Le Couedic JP , et al : A unique clonal JAK2 mutation leading tocausesconstitutive signaling
ligand interactions ... necessaryto triggersignaling
a model on parafibromin - mediated IFN - γtriggeredsignaling
2020 Absence of GP130 cytokine receptorcausessignaling
the binding of a lectin - likeligand to the CD24 carbohydrates(passive) could be triggered bySignaling
coexpressing the catalytic subunit of PKA ( pCEV , 0.5 μg(passive) was triggered bysignaling
These factors are knownto influencesignaling
Activation of a Few Dendritic SpinesSpecific Trans - Synaptic Interaction with Inhibitory Interneuronal Neurexin Underlies Differential Ability of Neuroligins(passive) Triggered bySignaling
Activation of a Few Dendritic Spines | Science Shenyu Zhai1 , Eugene D. Ark1 , Paula Parra - Bueno2(passive) Triggered bySignaling
ingress routers in the core of the network(passive) is triggered bySignaling
mTORC1 ... mTORC1 componentspreventsmTORC1 signaling
DYRK3 ... mTORC1 componentspreventsmTORC1 signaling
enhanced production of pro - inflammatory cytokines(passive) caused bysignaling
DNA damage or other stresses(passive) caused bysignaling
The modulation of TGF - βpreventssignaling
the binding of a lectin - like ligand to the CD24 carbohydrates(passive) could be triggered bySignaling
The mechanisms of glucosetriggeredsignaling
additional molecules involved in IFN - γtriggeredsignaling
voltage ... ableto influenceGqPCR signaling
terpenes ... ableto influencesignaling
51 : 676 – 681 : The putative ( pro)renin receptor blocker HRP failsto prevent( pro)renin signaling
Eph / ephrincontributessignaling
It is a user friendlyergonomically designedsignaling
the regulatory mechanisms that mediate cGAStriggeredsignaling
even low levels of calciumcan triggersignaling
the interaction of the Notch receptor with its transmembrane ligands expressed on adjacent cells(passive) is triggered bySignaling
This impaired BMPmay resultsignaling
to the induction of apoptosis and/or cell deathleadingto the induction of apoptosis and/or cell death
up and terminate a callto setup and terminate a call
the development of insulin resistancemay preventthe development of insulin resistance
neuron cell deaththereby preventingneuron cell death
cell death by necrosisto preventcell death by necrosis
to neuronal cell deaththus contributingto neuronal cell death
to cell death or neuroprotectionleadingto cell death or neuroprotection
up and manage phone callto setup and manage phone call
to NF - κB activation ( 17 ) andleadingto NF - κB activation ( 17 ) and
a skin cell syncytium and to stop programmed cell deathto causea skin cell syncytium and to stop programmed cell death
to the serum potentiation of GM - CSF releaseleadingto the serum potentiation of GM - CSF release
in increased proliferation and decreased cell death , which wasresultingin increased proliferation and decreased cell death , which was
to B cell activation , proliferation and differentiationleadingto B cell activation , proliferation and differentiation
to inhibition of apoptosisleadingto inhibition of apoptosis
to thymocyte apoptosisleadingto thymocyte apoptosis
to apoptosis in BL cellsleadsto apoptosis in BL cells
to insulin resistance and metabolic dysregulationcontributesto insulin resistance and metabolic dysregulation
in hepatic insulin resistance ( 18resultingin hepatic insulin resistance ( 18
to apoptosis , stress response , and inflammationleadingto apoptosis , stress response , and inflammation
to cell death and removal of apoptotic cellsleadingto cell death and removal of apoptotic cells
in apoptosis of human cancer cellsresultingin apoptosis of human cancer cells
to CDC25A / B / C- mediated dephosphorylation and restoring cell cycle progression In proliferating cellsleadingto CDC25A / B / C- mediated dephosphorylation and restoring cell cycle progression In proliferating cells
to the insulin resistance , which is the hallmark of diabetes mellituscan leadto the insulin resistance , which is the hallmark of diabetes mellitus
in oncogenesisresultingin oncogenesis
either cell survival or cell deathcauseseither cell survival or cell death
up a packet - switched call over the wireless networkto setup a packet - switched call over the wireless network
in cell death and inhibition of iNKT cell activationcan resultin cell death and inhibition of iNKT cell activation
to cAMP response element - binding protein phosphorylation and endothelial cellsleadingto cAMP response element - binding protein phosphorylation and endothelial cells
ACCcan causeACC
to epithelial - mesenchymal transition ( EMT ) andleadingto epithelial - mesenchymal transition ( EMT ) and
to full integrin activationleadingto full integrin activation
the internal switch to turn off the SMPS based at least partly on the signaling received , and consistent with that disclosed hereincausesthe internal switch to turn off the SMPS based at least partly on the signaling received , and consistent with that disclosed herein
to cellular apoptosis.2leadingto cellular apoptosis.2
to increased protection from apoptosisleadingto increased protection from apoptosis
to reduced microglial activation / proliferationcontributedto reduced microglial activation / proliferation
to insulin resistance , which is frequently observed in AD brainsleadingto insulin resistance , which is frequently observed in AD brains
growth arrest and/or apoptosis ( Zhang & Insel 2004could then triggergrowth arrest and/or apoptosis ( Zhang & Insel 2004
to inflammation and cellular stressleadingto inflammation and cellular stress
to altered mechanisms of integrin activation and adhesionleadingto altered mechanisms of integrin activation and adhesion
natural cell death instead of the rampant cell growth that is normal in tumorsto triggernatural cell death instead of the rampant cell growth that is normal in tumors