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Qaagi - Book of Why

Causes

Effects

its deleterious actionsmay resultin better therapy and outcome for patients with HF

Inflammatory activation in chronic HFleadsto proinflammatory and profibrotic processes , which culminate in ventricular remodeling , dysfunction and HF

how the hemodynamic and neurohormonal factors interactto causemaladaptive cardiac remodeling and progression to HF

patho)physiology Novel chimeric natriuretic peptide CD - NPpreventscardiorenal remodeling and dysfunction in experimental myocardial infarction

excessive activation of protein Kinase A ( PKA ) and Ca2+/calmodulin - dependent kinase II ( CaMK IIcausesadverse cardiac remodeling and promotes HF development

Variations in ABCG2 activitymight influencepharmacokinetics and therapeutic outcome of TKIs

the hypothesis ... superoxide - dependnet Cat activation inhibitionpreventsLV remodeling and dysfunction in the development of HF

Stenotic aortic valve disease ( AS ) causes pressure overload of the left ventricle ( LVmay triggeradverse remodeling and precipitate progression towards heart failure ( HF

COX III participates in stress - induced apoptosis andcontributesto myocardial remodeling and dysfunction following MI and HF

chronic anemialeadsto LV remodeling and poorer outcomes in HF

to better survival with this diseasehas ledto better survival with this disease

to a substantial improvement of mortality and rehospitalizationhas ledto a substantial improvement of mortality and rehospitalization

prevalence and type of SAmight influenceprevalence and type of SA

an art project encouraging patients to think about what was working well for themledan art project encouraging patients to think about what was working well for them

in easier management of neonatal sepsisresultingin easier management of neonatal sepsis

urinary weekscausesurinary weeks

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