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a Philadelphia ( Ph0 ) chromosomeresultingfrom the t(9;22)(q34;q11 ) chromosomal translocation
inhibition of p65causingNF-?B translocation
NUP98-HOXA9 chimeric proteinresultingfrom t(7;11)(p15;p15 ) chromosomal translocation
acute ATR inhibitioninfluenceschromosome translocation
double - strand breaks ( DSBs ) in chromosomes and subsequent non - homologous end joining ( NHEJ ) between different chromosome arms2(passive) is caused byChromosomal translocation
non - homologous chromosomecausingtranslocation Chromosomal translocation
A SYT - SSX fusion generesultingfrom the chromosomal translocation
cholesterol(passive) is influenced bySMO translocation
impaired DNA double - strand breakcould causechromosomal translocation
The factorscould influencechromosomal translocation
Aforementioned chronic oxidative stress induced by long - term intake of HFDcould causeNrf2 translocation
TET1 was first identified as a rare fusion partner of the mixed lineage leukemia gene ,resultingfrom the chromosomal translocation
by distorted genetic development that has evolved over time(passive) is caused byChromosomal translocation
distorted genetic development that has evolved over time(passive) is caused byChromosomal translocation
AMP - activated protein kinase activationcausesGLUT4 translocation
AMPK is known ... , resulting in insulin - independent glucose uptaketo causeGLUT4 translocation
VEGFcausestranslocation of Syx
AMPK is knownto causeGLUT4 translocation
in turnwould influencePKC translocation
a signal cascadecan causeGLUT4 translocation
as cAMP is knownto ... influenceGLUT4 translocation
by the loss of both of them(passive) caused byGLUT4 translocation
the loss of both of them(passive) caused byGLUT4 translocation
PMAcausedPKC - translocation
In contrastcausedtranslocation of PKC -
The wide range of stress conditionsprovokeNPM1 translocation
The fragment may incorrectly reattach to another , non - homologous chromosomecausingtranslocation
Incubation of promastigotes with AVLcausedtranslocation of phosphatidylserine
non - homologous chromosomecausingtranslocation
Binding of the ligandcausestranslocation
breakpoints near proto - oncogene leading to inappropriate expression , or even fusion with another protein(passive) caused byTranslocation
when piece of chromosome attaches itself to another in cell(passive) is causedTranslocation
by defective mucosal barrier(passive) caused bytranslocation
Vascular endothelial growth factorcausestranslocation of p47phox
these mutations(passive) are causedtranslocation
Binding to PIM1causestranslocation
when a piece of chromosome 21 attaches itself to another chromosome in a cell(passive) is causedTranslocation
rearranged chromosome material(passive) is caused byTranslocation
by LPS(passive) caused bytranslocation
The diseasecausingtranslocation
to expression of the unique SYT - SSX fusion protein inleadingto expression of the unique SYT - SSX fusion protein in
in expression of an oncogenic kinase fusion protein known as nucleophosmin - anaplastic lymphoma kinase ( NPM - ALKresultingin expression of an oncogenic kinase fusion protein known as nucleophosmin - anaplastic lymphoma kinase ( NPM - ALK
in the formation of the BCR - ABL1 fusion gene ( 1resultsin the formation of the BCR - ABL1 fusion gene ( 1
to a fusion protein , EWS - FLI1leadingto a fusion protein , EWS - FLI1
This ... a mutation in which the abnormal gene BCR - ABL , is turned into a CML cell(passive) is caused byThis ... a mutation in which the abnormal gene BCR - ABL , is turned into a CML cell
in the formation of the BCR - ABL oncogeneresultsin the formation of the BCR - ABL oncogene
in fusion of the genes encoding RNA - binding protein EWS and transcription factor FLI1 ( EWS - FLI1resultsin fusion of the genes encoding RNA - binding protein EWS and transcription factor FLI1 ( EWS - FLI1
in fusion of the genes encoding RNA - binding protein EWS and transcription factorresultsin fusion of the genes encoding RNA - binding protein EWS and transcription factor
in the formation of the Philadelphia chromosomeresultingin the formation of the Philadelphia chromosome
to overexpression of cyclin D1leadingto overexpression of cyclin D1
in the production of fusion oncoproteinresultingin the production of fusion oncoprotein
in over - expression of cyclin D1resultingin over - expression of cyclin D1
in overexpression of the antiapoptotic protein BCL-2resultingin overexpression of the antiapoptotic protein BCL-2
BCL2 protein overexpressioncausingBCL2 protein overexpression
in the juxtaposition of the bcl-2 proto - oncogene with the heavy chainresultsin the juxtaposition of the bcl-2 proto - oncogene with the heavy chain
to change in expression levelleadsto change in expression level
in the fusion of two myogenic transcription factors , Pax3 and FOXO1resultingin the fusion of two myogenic transcription factors , Pax3 and FOXO1
to the ectopic expression of NPM - ALK , a chimeric tyrosine kinaseleadingto the ectopic expression of NPM - ALK , a chimeric tyrosine kinase
to a chimeric transcript EWSR1 / FLI1leadsto a chimeric transcript EWSR1 / FLI1
a myeloproliferative disease ,(passive) caused bya myeloproliferative disease ,
miscarriagecausedmiscarriage
in 3 copies of the critical region for Down syndromeresultsin 3 copies of the critical region for Down syndrome
enhanced glucose uptakecausingenhanced glucose uptake
alsoresultsalso
alsocan ... leadalso
to the EHE - specific fusion oncogene WWTR1-CAMTA1leadsto the EHE - specific fusion oncogene WWTR1-CAMTA1
to tumour cell transformation3leadingto tumour cell transformation3
in insulin - independent glucose uptakeresultingin insulin - independent glucose uptake
to reduced fertilitycan leadto reduced fertility
in a BCR - ABL fusion which is a driver of the diseaseresultsin a BCR - ABL fusion which is a driver of the disease
to be randomdesignedto be random
to lymphomagenesis65leadingto lymphomagenesis65
repeated abortioncausingrepeated abortion
in an oncogenic BCR - ABL gene fusion that can be found on the shorter derivative 22 chromosomeresultsin an oncogenic BCR - ABL gene fusion that can be found on the shorter derivative 22 chromosome
in an BCR - ABL gene fusion that can be found on the shorter derivative 22 chromosomeresultsin an BCR - ABL gene fusion that can be found on the shorter derivative 22 chromosome
to the development of the BCR / ABL fusion gene and the corresponding fusion proteinleadingto the development of the BCR / ABL fusion gene and the corresponding fusion protein
in insulin - independent glucoseresultingin insulin - independent glucose
in overexpression of the anti - apoptotic protein BCL2resultsin overexpression of the anti - apoptotic protein BCL2
to PML - RARa fusion geneleadingto PML - RARa fusion gene
to the BCR - ABL gene fusionleadingto the BCR - ABL gene fusion