persistent hypertensioncausespersistent hypertension

arterial hypertension(passive) caused byarterial hypertension

from prolonged hypertensionresultingfrom prolonged hypertension

t. Abdominal pain(passive) may also be caused byt. Abdominal pain

to renin - mediated posttraumatic hypertensioncan leadto renin - mediated posttraumatic hypertension

in rapid development of stable hypertensionresultedin rapid development of stable hypertension

to renin secretion with resulting hypertension ( 2leadingto renin secretion with resulting hypertension ( 2

abrupt and severe hypertension and less frequently renal failurecan causeabrupt and severe hypertension and less frequently renal failure

to parenchymal atrophy , impaired renal function and hypertensionleadingto parenchymal atrophy , impaired renal function and hypertension

nocturia , refractory hypertension , and severe renal dysfunctionoften causesnocturia , refractory hypertension , and severe renal dysfunction

flank pain and hematuriamay causeflank pain and hematuria

Renal tubular epithelial cell damage(passive) caused byRenal tubular epithelial cell damage

acute kidney injury ( AKIcan causeacute kidney injury ( AKI

an endothelial cell injury and dysfunction followed by endothelial cell edemacausesan endothelial cell injury and dysfunction followed by endothelial cell edema

from traumatic renal artery dissection Korean J Intern Medresultingfrom traumatic renal artery dissection Korean J Intern Med

from traumatic renal artery dissection The Korean Journal of Internal Medicineresultingfrom traumatic renal artery dissection The Korean Journal of Internal Medicine

in vasospasm which presents as complete nonvisualization of the kidney [ 21may also resultin vasospasm which presents as complete nonvisualization of the kidney [ 21

loin pain and hematuriacausesloin pain and hematuria

acute flank pain in patients presenting to the EDcausingacute flank pain in patients presenting to the ED

intense lower back pain or flank paincan causeintense lower back pain or flank pain

in necrosis of tubular cells , which then slough off into the tubules and block them so that filtrate can not flow through the Part 4 Regulations and Maintenance Systems Pathology Acute Renal Failure Figure B A Patient Undergoing Dialysis tubulesresultsin necrosis of tubular cells , which then slough off into the tubules and block them so that filtrate can not flow through the Part 4 Regulations and Maintenance Systems Pathology Acute Renal Failure Figure B A Patient Undergoing Dialysis tubules

Prevent DIC and damage of renal parenchyma(passive) caused byPrevent DIC and damage of renal parenchyma

to the development of an acute renal failure characterized by a massive tubular cell death mainly affecting S3 segments of proximal tubules in Outer stripe of outer medulla ( OSOMleadsto the development of an acute renal failure characterized by a massive tubular cell death mainly affecting S3 segments of proximal tubules in Outer stripe of outer medulla ( OSOM

hematuria , pyuria , flank pain , and decreased urine outputcauseshematuria , pyuria , flank pain , and decreased urine output

He Jianteng GuEmail author Acute lung injury(passive) caused byHe Jianteng GuEmail author Acute lung injury

PI3K / Akt pathway Acute lung injury(passive) caused byPI3K / Akt pathway Acute lung injury

Acute kidney injury , a devastating kidney disease(passive) is often caused byAcute kidney injury , a devastating kidney disease

to sympathetic activationcan leadto sympathetic activation

in a wide spectrum of clinical manifestations depending on the size of the artery involvedresultingin a wide spectrum of clinical manifestations depending on the size of the artery involved

to renal damage.30 31leadingto renal damage.30 31

renal tubular epithelial cell injury / necrosis(passive) caused byrenal tubular epithelial cell injury / necrosis

pp . 1 - 11 Abstract Background Acute lung injury(passive) caused bypp . 1 - 11 Abstract Background Acute lung injury

redistribution of Na(+)-K(+)-adenosinetriphosphatase ( Na(+)-K(+)-ATPase ) to the apical membrane of proximal tubulescausesredistribution of Na(+)-K(+)-adenosinetriphosphatase ( Na(+)-K(+)-ATPase ) to the apical membrane of proximal tubules

an increase in renin - AII - aldosterone , which in turn raises blood pressurecausesan increase in renin - AII - aldosterone , which in turn raises blood pressure

from traumatic renal artery dissection Kyung Pyo Kang , Sik Lee , Won Kim , Gong Yong Jin , Ki Ryang Na , Il Yong Yun , Sung Kwang Park Korean J Intern Medresultingfrom traumatic renal artery dissection Kyung Pyo Kang , Sik Lee , Won Kim , Gong Yong Jin , Ki Ryang Na , Il Yong Yun , Sung Kwang Park Korean J Intern Med

eventually to irreversible renal impairmentleadseventually to irreversible renal impairment

from renal vein thrombosis complaint of urine discoloration like dark yellow or brown urinecan be causedfrom renal vein thrombosis complaint of urine discoloration like dark yellow or brown urine

consistent increase of renin which is helpful in promoting the generation of type Ⅱ angiotensincan causeconsistent increase of renin which is helpful in promoting the generation of type Ⅱ angiotensin

activation of afferent neural communicationmay triggeractivation of afferent neural communication

to the inability of the kidneys to process and excrete the body 's waste products ( kidney failurecan leadto the inability of the kidneys to process and excrete the body 's waste products ( kidney failure