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by lipid composition and melting phase transition temperature ( Tm(passive) to be influenced byrelease of liposomes
lipid composition and melting phase transition temperature ( Tm(passive) to be influenced byrelease of liposomes
myeloid cellscausingrelease of proinflammatory cytokines
Atpcausesrelease of intracellular
Depolarization using a nerve impulse opens voltage gated calcium channelsto causerelease of acetylcholine
a cell signaling responseleadsto the release of cytokines
stimulation of these receptorscausingrelease of acetylcholine
causes binding of vesicles containing acetylcholine to the pre synaptic membranecausingrelease of acetylcholine
Stimulation of the nervecausesrelease of acetylcholine
specific receptorscausingrelease of acetylcholine
immune system cellscausinga release of cytokines
Activationleadsto the release of cytokines
Most likely the activation ( or suppression ) of the genescausesrelease of cytokines
immune inflammatory genes ... in turncausethe release of cytokines
The inflammatory responsecausesrelease of cytokines interleukin-1
T - lymphocytes in the bodycausingthe release of cytokines
inflammationcausesthe release of cytokines
in turncausethe release of cytokines
Motilin receptor activationcausesrelease of acetylcholine
Arrival of nerve impulsecausesrelease of acetylcholine
Macrophagescausethe release of cytokines
fever ... in turncausesthe release of cytokines
potassium channelcausingrelease of acetylcholine
Mechanical ventilation ( MV ) may activate the innate immune systemcausingthe release of cytokines
epithelial differentiation and epithelial inflammationresultingfrom release of cytokines
the binding of Fccausesthe release of cytokines
The biochemical pathwaycausingthe release of cytokines
Stresscan also causerelease of ACTH
by acute(passive) caused bythe release of cytokines
reactionscausingrelease of cytokines
the anterior pituitaryto causerelease of ACTH
an immune responsecausesthe release of cytokines
ROScausesrelease of cytokines
The bacteria proliferate within the macrophagescausingrelease of cytokines
an incorrect responsecausesrelease of acetylcholine
Corticotropin Releasing Factor ( CRFcausesrelease of ACTH
Another theory iscausesrelease of cytokines
the synapsescausesrelease of Acetylcholine
SEB binds to T - lymphocytes in the bodycausingthe release of cytokines
repetitive workcausesthe release of cytokines
in cell deathresultsin cell death
runny nose , eye itching , sneezing and other symptoms of Mold Allergycausesrunny nose , eye itching , sneezing and other symptoms of Mold Allergy
inflammation which may induce sickness behavior such as decreased appetite , increased withdrawal , fatigue , and other symptoms also linked to depressioncausesinflammation which may induce sickness behavior such as decreased appetite , increased withdrawal , fatigue , and other symptoms also linked to depression
to further damage of myofibers and activation and proliferation of satellite cellsleadingto further damage of myofibers and activation and proliferation of satellite cells
This inflammatory state(passive) is caused byThis inflammatory state
to nasal congestion swelling and irritationcausesto nasal congestion swelling and irritation
to inflammation and rapid production of the skin cellsleadto inflammation and rapid production of the skin cells
inflammation and the rapid production of skin cellscauseinflammation and the rapid production of skin cells
to inflammation and rapid production in the skin cellsleadto inflammation and rapid production in the skin cells
inflammation , cell death and tissue damagecauseinflammation , cell death and tissue damage
inflammation of the typical symptoms of illnesswould causeinflammation of the typical symptoms of illness
to inflammation that leads to joint pain and possibly fatigueleadto inflammation that leads to joint pain and possibly fatigue
in turn to retention of sodium and waterleadsin turn to retention of sodium and water
in a general activation of the immune systemresultin a general activation of the immune system
to retention of sodium and waterleadsto retention of sodium and water
inflammationcausesinflammation
to more inflammationleadto more inflammation
inflammationcausesinflammation
in inflammationresultingin inflammation
to inflammationleadto inflammation
inflammationcauseinflammation
inflammationcausesinflammation
to inflammationleadsto inflammation
to more inflammationleadto more inflammation
to the detrimental inflammatory response [ 17 , 18contributesto the detrimental inflammatory response [ 17 , 18
in the proliferation of antigen - specific T - cellsthen resultsin the proliferation of antigen - specific T - cells
to Th1 and Th17 phenotypesleadingto Th1 and Th17 phenotypes
inflammation to developcauseinflammation to develop
inflammation in the sufferers braincauseinflammation in the sufferers brain
many functions , such as airway remodeling , a characteristic of IL-17 responsescausesmany functions , such as airway remodeling , a characteristic of IL-17 responses
to further modulation of cell activityleadingto further modulation of cell activity
to cell death apoptosisleadingto cell death apoptosis
sodium retentioncausessodium retention
activation of the inflammatory cascadescausesactivation of the inflammatory cascades
in symptoms of chills , fever and possibly shockresultingin symptoms of chills , fever and possibly shock
to increased salt and water retentionleadsto increased salt and water retention
to the recruitment of granulocytes , monocytes and mast cells at the site of inflammationcontributesto the recruitment of granulocytes , monocytes and mast cells at the site of inflammation
to macrophage activationleadingto macrophage activation
to Th1 and Th17 polarization and phenotypesthus leadingto Th1 and Th17 polarization and phenotypes
an inflammatory response in the area and the release of neurotransmitters at synapses in the nervous systemcausean inflammatory response in the area and the release of neurotransmitters at synapses in the nervous system