Loading ...
Polynucleotides and other polyanions have been shownto causerelease of cytokines Dompridone
localized at the site of infectionto preventrelease of the cytokines
.ILA drschmoctor Neutrophils ... donor antigen ( e.g. , anti leukocyte IgG ) ,causesrelease of cytokines
Surgical stresscausesrelease of cytokines
Another theory iscausesrelease of cytokines
Indirect involvement of the myocardium can occur as a result of a severe systemic inflammationcausingrelease of cytokines / chemokines
myeloid cellscausingrelease of proinflammatory cytokines
the surgery itselfcausingrelease of cytokines
the osteoblastscausingrelease of cytokines
also(passive) can ... be triggeredRelease of proinflammatory cytokines
the hypothalamic - pituitary - adrenal axiscausingrelease of proinflammatory cytokines
reactionscausingrelease of cytokines
an antigen triggering IgE mediated type 1 eosinophilic reaction or abnormal T - cell activationcausingrelease of eosinophilotrophic cytokines
with fixed Mϕsto preventrelease of cytokines
atelectasis does NOT cause fever , anesthetics and tissue traumacausingrelease of pyrogenic cytokines
Gram - negative bacilli ( mainly Escherichia coli ) and other toxins , ... in turncausesrelease of cytokines
5β - hydroxypalisadin B ... and suppression of the release of cytokines by 5β - hydroxypalisadin B(passive) was ... influenced byRelease of the cytokines
ATPcausesrelease of intracellular
AvrA is a naturally evolved enzyme capable of disrupting inflammatory and apoptotic signaling pathways inside the cellpreventingrelease of cytokines
DMStriggeredrelease of the cytokines
synthetic MALP-2can causerelease of proinflammatory cytokines
local inflammatory conditionresultingrelease of cytokines
> absorption of toxin from local sitecausesrelease of cytokines
superantigenscausingrelease of cytokines
Oral feeding of neonates with immature gut immune systemcausesrelease of proinflammatory cytokines
iron accumulation in microglia alone ... sufficientto triggerrelease of proinflammatory cytokines
in systemic inflammatory response syndrome ( SIRScan resultin systemic inflammatory response syndrome ( SIRS
adverse host responsescauseadverse host responses
adverse host Reviewcauseadverse host Review
local inflammation and specific killing of virus - infected cells ( 1to causelocal inflammation and specific killing of virus - infected cells ( 1
a systemic inflammatory response syndrome , activation of the kinin system , the intrinsic clotting cascade [ doi.orgcausinga systemic inflammatory response syndrome , activation of the kinin system , the intrinsic clotting cascade [ doi.org
hypotension , rigors , fever , shock , and even deathcan causehypotension , rigors , fever , shock , and even death
local inflammation , which activates an immune response1 Immunogenic cell death also releases DAMPs and tumor - derived antigens , which induce inflammation , recruit innate immune cells , and establish innate and adaptivecauseslocal inflammation , which activates an immune response1 Immunogenic cell death also releases DAMPs and tumor - derived antigens , which induce inflammation , recruit innate immune cells , and establish innate and adaptive
clinical symptoms that include fever , hypotension , and shock ( typically seen in toxic shock syndromecausingclinical symptoms that include fever , hypotension , and shock ( typically seen in toxic shock syndrome
Onset(passive) is triggered byOnset
an inflammatory reaction(passive) caused byan inflammatory reaction
increased secretions from the nosecauseincreased secretions from the nose
to feedback inhibition through SOCSleadsto feedback inhibition through SOCS
to chemotaxis Activation of B cellsleadingto chemotaxis Activation of B cells
a syndrome of fevers , rigors , myalgias , arthralgias , nausea , vomiting , diarrhea , and possibly hypotensioncausea syndrome of fevers , rigors , myalgias , arthralgias , nausea , vomiting , diarrhea , and possibly hypotension
to the state of relative immune tolerance in the livercontributeto the state of relative immune tolerance in the liver
activation of immune cellstriggeractivation of immune cells
monocyte differentiationinfluencemonocyte differentiation
the liver to bring forth C - reactive protein ( Vanderploeg . 2008causethe liver to bring forth C - reactive protein ( Vanderploeg . 2008
to apoptosis of hematopoietic progenitor and stem cellsleadingto apoptosis of hematopoietic progenitor and stem cells
in an intense inflammatory responseresultingin an intense inflammatory response
chorioamnionitis , which leads to enhanced uterine contractions , ultimately resulting in placental abruption ( Oyelese and Ananth 2006 ; Tikkanen et al .causingchorioamnionitis , which leads to enhanced uterine contractions , ultimately resulting in placental abruption ( Oyelese and Ananth 2006 ; Tikkanen et al .
up ... regulation of adhesion molecules , mobilization and activation of leukocytes , platelets and endotheliumcauseup ... regulation of adhesion molecules , mobilization and activation of leukocytes , platelets and endothelium
to dysregulation of B cells and subsequent production of autoantibodiesleadingto dysregulation of B cells and subsequent production of autoantibodies
marrow cells to mobilize and home to the injury site ( vasculogenesisto causemarrow cells to mobilize and home to the injury site ( vasculogenesis
a wave of chemotaxiscreatinga wave of chemotaxis
toxins to run in the tissues and blood streamcan causetoxins to run in the tissues and blood stream
to Th1 andthus leadingto Th1 and
upregulation of adhesion molecules , mobilization and activation of leukocytes , platelets , and endotheliumcauseupregulation of adhesion molecules , mobilization and activation of leukocytes , platelets , and endothelium
neutrophils to seek the site of infection by chemotaxistriggerneutrophils to seek the site of infection by chemotaxis
paincausepain
large numbers of immune cells to arrive at the site of infection and destroy the bacteriacauseslarge numbers of immune cells to arrive at the site of infection and destroy the bacteria
in massive proliferation of T lymphocytesresultingin massive proliferation of T lymphocytes
in both autocrine ( cellresultingin both autocrine ( cell
formation of noncaseating granulomas A 16-year - old bird owner presents with a history of 1 year of shortness of breath , particularly on exertion , productive cough , fatigue and weight losscausingformation of noncaseating granulomas A 16-year - old bird owner presents with a history of 1 year of shortness of breath , particularly on exertion , productive cough , fatigue and weight loss
macrophage recruitment and other changes leading to intraocular pressure reductiontriggermacrophage recruitment and other changes leading to intraocular pressure reduction
vasodilation of the synovial capillariescausevasodilation of the synovial capillaries
to the high rates of bone turnover and failure of remodellingwill contributeto the high rates of bone turnover and failure of remodelling
neutrophils and cellular damage(passive) caused byneutrophils and cellular damage
downstream end organ damagecausingdownstream end organ damage
to the detrimental inflammatory response [ 17 , 18contributesto the detrimental inflammatory response [ 17 , 18