to increased lifespanleadsto increased lifespan

a reversible G2 arrest in cancer cellscausesa reversible G2 arrest in cancer cells

in more cellsresultedin more cells

in increased p65 in the nucleuscan ... resultin increased p65 in the nucleus

to the upregulation of the histoneleadsto the upregulation of the histone

to protectionsurprisingly leadsto protection

to protectionleadsto protection

enhanced degradation of p53causesenhanced degradation of p53

to a nearly three - fold increase in reporter activityledto a nearly three - fold increase in reporter activity

6 push button(passive) can be set6 push button

hyperactivity and impulsivity in mice viacauseshyperactivity and impulsivity in mice via

hyperactivity and impulsivity in mice via causeshyperactivity and impulsivity in mice via

to stomach irritationcan leadto stomach irritation

in distribution of attention over the two visual fieldsresultingin distribution of attention over the two visual fields

to PGD2 suppressionleadsto PGD2 suppression

to inflammation , necrosis , steatosis and fibrosisleadsto inflammation , necrosis , steatosis and fibrosis

to loss of the prosurvival protein survivin and that depletion of survivin selectively kills cells that overexpress MYCleadsto loss of the prosurvival protein survivin and that depletion of survivin selectively kills cells that overexpress MYC

clinicallycan causeclinically

delays in the progression of candidate drugs , orcan causedelays in the progression of candidate drugs , or

in a synthetic lethality in mouse lymphoma and hepatoblastoma with MYC hyper - activationresultedin a synthetic lethality in mouse lymphoma and hepatoblastoma with MYC hyper - activation

to this effectis ... contributingto this effect

to a reduced spine densityledto a reduced spine density

poly - ubiquitinated species of all three proteins to accumulatecausedpoly - ubiquitinated species of all three proteins to accumulate

modest changes in the expression of many target genes ( 1.15 to 1.4 fold change for significant genes in our experimentscausedmodest changes in the expression of many target genes ( 1.15 to 1.4 fold change for significant genes in our experiments

to relaxation of its antagonistleadsto relaxation of its antagonist

to synergistic dominanceleadsto synergistic dominance

delays in the progression of candidate drugs ... or even premature closure of projectscan causedelays in the progression of candidate drugs ... or even premature closure of projects

to detectable pathological changes within the liver organoidsleadsto detectable pathological changes within the liver organoids

to a network dynamic of competitionwill ... leadto a network dynamic of competition

in a cogent subduing of the immunosuppressive consequence of M - MDSCs than that of G - MDSCs in an in - vitro studyresultedin a cogent subduing of the immunosuppressive consequence of M - MDSCs than that of G - MDSCs in an in - vitro study

hyperactivity and impulsivity in mice via dopamine D2 receptor - mediated mechanismcauseshyperactivity and impulsivity in mice via dopamine D2 receptor - mediated mechanism

to the proper circadian oscillation of both transcriptscontributesto the proper circadian oscillation of both transcripts

to an increase in LPL , GLUT4 and FABPs at 4 days post - infection of miR-122 inhibitorleadsto an increase in LPL , GLUT4 and FABPs at 4 days post - infection of miR-122 inhibitor

to relaxation of opposing muscles during physical activityleadingto relaxation of opposing muscles during physical activity

to attenuated mTORC1 but not mTORC2 signaling , suggesting that mTORC2 / SGK1 and Rac1 / PAK1 pathways are independently responsible for mTORC1 activation in NF2-deficient meningiomasalso leadsto attenuated mTORC1 but not mTORC2 signaling , suggesting that mTORC2 / SGK1 and Rac1 / PAK1 pathways are independently responsible for mTORC1 activation in NF2-deficient meningiomas