cancer , infection , drugs(passive) caused byReactive thrombocytosis
wikipedia.org ) Insults to neurons in the central nervous system caused by infection , trauma , ischemia , stroke , autoimmune responses , or other neurodegenerative diseasesmay causereactive astrocytes
A skin infection such as impetigo or cellulitisalso can causereactive lymphadenopathy
Otrivin ... unlikelyto causereactive hyperaemia
various things - most commonly alcohol , NSAIDs and Helicobacter pylori infection(passive) can be caused byReactive gastropathy
a five - year look back period in placeto preventreactive divestitures
Infection James L. Chen , Khosrow Afsari , MD Infect Med(passive) Caused byReactive Thrombocytosis
Qualitative Turmeric paper Paper strip Coming into contact with bacteria and developing an infectioncan triggerreactive arthritis.[1
if the copper has corrodedcreatingreactive salts.[3
prolonged stimulation of the immune system by a number of agents(passive) is caused byReactive polyarthritis
iron deposition in the hippocampus of rats(passive) caused byreactive astrocytosis
an inflated cuff ( 250 mm Hg for 5 min(passive) created byreactive hyperemia
a downstream vasodilation(passive) is caused byReactive hyperemia
pathological conditionscausingreactive gliosis
Long - term use of NSAIDsmay causereactive gastropathy
the underlying mechanismscausingreactive gliosis
inflammation or prior fine needle aspiration(passive) can be caused byReactive atypia
Our rationale systemwill preventreactive
a role in man never passed through the hypothalamus and limbic corticesprovokedreactive gliosis
a rule in placeto preventreactive divestitures
that has been put into placeto preventreactive divestitures
the ITDC and autoregulatory vasodilationcausingreactive hyperemia
skin conditions in the vicinity(passive) can be caused byReactive lymphadenopathy
epilepsy ... not that they could be the cause(passive) were caused byreactive astrocytes
The intervention employed to induce acute systemic hypotension in this studycreatesreactive hyperemia
systemic administration of Stx2 and/or LPScausedreactive astrocytes
medications , ethanol , bile reflux or by vascular problems or vascular congestion(passive) is usually caused byReactive gastropathy
some metabolic disorders and some tumors as wellcan causereactive hypos
We assume that either a mutation occurred during the breeding process necessary to produce an inbred strain ( as is the case for BXD24 ) or the specific constellation of genes in these particular strainscausereactive gliosis
infections and inflammatory disorders Clonal thrombocytosis(passive) can be caused byReactive thrombocytosis
nerve damage or degenerative brain diseases(passive) caused byreactive gliosis
the diagnosis of familial autoinflammatory diseases ( FADscausingreactive amyloidosis
long - term contact with substances that irritate the stomach lining , most often nonsteroidal anti - inflammatory drugs ( NSAIDs ) , alcohol , and bile reflux , which is backward flow of bile from the small intestine to the stomach(passive) is caused byReactive gastropathy
the place by staphylococcus aureus , iliescu r.(passive) is influencedreactive histiocytosis
blood pressure cuff maneuver and arm exercise(passive) provoked byReactive hyperemia
a cuff inflated at suprasystolic pressure for 5 mins(passive) created byreactive hyperemia
in response to several CNS pathologies , such as trauma , stroke , and neurodegenerative diseases [ 77(passive) can be triggeredReactive gliosis
a heart attack , allergic reactions , exercise , cancer , vitamin deficiency , iron deficiency , infections such as tuberculosis , removal of the spleen , coronary artery bypass , and acute bleeding(passive) can be caused byReactive thrombocytosis
to the formation of the glial scar , but they might also have beneficial effects on axonal growthcontributeto the formation of the glial scar , but they might also have beneficial effects on axonal growth
to 28 neuronal damage as well as compromising transplantioncan contributeto 28 neuronal damage as well as compromising transplantion
in neuroinflammation , neurotoxic function and the formation of a glial scar ( 39resultin neuroinflammation , neurotoxic function and the formation of a glial scar ( 39
to impairment of axonal regeneration by the formation of glial scar [ 31contributeto impairment of axonal regeneration by the formation of glial scar [ 31
to the inhibition of glial scar formation in vitromay leadto the inhibition of glial scar formation in vitro
to the rapid formation of injectable hydrogels with improved modulusleadingto the rapid formation of injectable hydrogels with improved modulus
to hippocampal neuronal damage under hypoxic and ischemic conditionscan ... contributeto hippocampal neuronal damage under hypoxic and ischemic conditions
of Butyl Acrylate and Glycidyl ( Meth)AcrylateComposedof Butyl Acrylate and Glycidyl ( Meth)Acrylate
to the persistent and development of chronic pain ... 4contributeto the persistent and development of chronic pain ... 4
to chemotherapy - related thrombophilia in patients with lung cancermight contributeto chemotherapy - related thrombophilia in patients with lung cancer
a minor component to the cellularity presentcontributeda minor component to the cellularity present
to renal failure and can be prophylactically treated with colchicineleadsto renal failure and can be prophylactically treated with colchicine
to stroke recovery ... partly via the release of a nuclear protein called high - mobility group box 1may ... contributeto stroke recovery ... partly via the release of a nuclear protein called high - mobility group box 1
from tissue damage resulting from an injury , or of a surgical procedurecan resultfrom tissue damage resulting from an injury , or of a surgical procedure
to inflammation of the gallbladder wallleadsto inflammation of the gallbladder wall
to increased level of inflammationleadsto increased level of inflammation
to tissue damage in MS through impaired glutamate handling and redox homeostasislikely contributeto tissue damage in MS through impaired glutamate handling and redox homeostasis
to their beneficial effects at impairing inflammationmay contributeto their beneficial effects at impairing inflammation
scar formation without depleting reactive astrocytes from the lesion sitepreventsscar formation without depleting reactive astrocytes from the lesion site
TRPA1 in the cells that line the surface of the bodycan triggerTRPA1 in the cells that line the surface of the body
Symptoms of reactive thrombocytosis Recent questions on Reactive thrombocytosiscausesSymptoms of reactive thrombocytosis Recent questions on Reactive thrombocytosis
to functional recovery after stroke by using an in vivo imaging methodcontributeto functional recovery after stroke by using an in vivo imaging method
to cerebral microcirculatory impairments during the early stage of embolic stroke by constricting cerebral vesselsmay ... contributeto cerebral microcirculatory impairments during the early stage of embolic stroke by constricting cerebral vessels
largely to the NF - κB transcriptional signature ... and to pro - inflammatory cytokine expression ... in neuroinflammation - associated schizophrenia in the putative absence of microglial activationcontributelargely to the NF - κB transcriptional signature ... and to pro - inflammatory cytokine expression ... in neuroinflammation - associated schizophrenia in the putative absence of microglial activation
to regulation of CNS inflammation , tissue protection , tissue repair , axon regeneration and the maintenance or recovery of neurological function after diverse CNS insultscontributeto regulation of CNS inflammation , tissue protection , tissue repair , axon regeneration and the maintenance or recovery of neurological function after diverse CNS insults
degeneration of DA neurons in the SN during inflammation by producing NADPH oxidase - derived ROS.31 , 32 Many clinical and experimental data have demonstrated the existence of reactive microglia and increased ROS generation by activated NADPH oxidase originating from reactive microglia in the SN of PD patients33 , 34and in the SN of the MPTP mouse PD model.21 , 22can causedegeneration of DA neurons in the SN during inflammation by producing NADPH oxidase - derived ROS.31 , 32 Many clinical and experimental data have demonstrated the existence of reactive microglia and increased ROS generation by activated NADPH oxidase originating from reactive microglia in the SN of PD patients33 , 34and in the SN of the MPTP mouse PD model.21 , 22
the shear rate(passive) created bythe shear rate
to overestimation of the degree of intracranial stenosismay possibly leadto overestimation of the degree of intracranial stenosis