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PMID:19936862.(2010)Vang A , Mazer J , Casserly B , Choudhary G. Activation of endothelial BKCa channelscausespulmonary vasodilation
Vascular pharmacology Activation of endothelial BKCa channelscausespulmonary vasodilation
severe congenital diaphragmatic herniais causingpulmonary hypoplasia
McMurtry I. F. Endothelin 1causespulmonary vasodilation
Vang A , Mazer JM , Casserly B , Choudhary G. Activation of endothelial BKCa channelscausespulmonary vasodilation
University of Arizona Thiol oxidationcausespulmonary vasodilation
a history of severe and prolonged oligohydramnioscausingpulmonary hypoplasia
congenital diaphragmatic hernia — University of Texas Southwestern Medical Center(passive) caused bypulmonary hypoplasia
309441718.Platelet - activating factorcausespulmonary vasodilation
Publications ] M. Nakanishi : " Leukotoxin , 9,10-Epoxy-12-octadecenoatecausespulmonary vasodilation
2002 Dipyridamole , a cGMP phosphodiesterase inhibitorcausespulmonary vasodilation
Abman , Dipyridamole , a cGMP phosphodiesterase inhibitorcausespulmonary vasodilation
various etiologies such as oligohydramnios , congenital diaphragmatic hernia , pulmonary masses , chest wall deformities , and hydrothorax ( 2(passive) can be caused byPulmonary hypoplasia
1384 - 1392 Vascular endothelial growth factorcausespulmonary vasodilation
Inhalation of NO gascausespulmonary vasodilation
Inhaled nitric oxidecausespulmonary vasodilation
to rescue the fetus from an underlying conditioncausingpulmonary hypoplasia
Thio oxidationcausespulmonary vasodilation
Oxygen is knownto causepulmonary vasodilation
birth - related stimuli in the ovine fetus | Heart and Circulatory Physiology Skip to(passive) caused bypulmonary vasodilation
Thorax ... severe enoughto causepulmonary hypoplasia
Genetic defects that perturb the thoracic skeleton , or space - occupying lesions that compress the lungs , like congenital diaphragmatic herniacan causepulmonary hypoplasia
endothelium - dependent nitric oxide(passive) caused byPulmonary vasodilation
Nitric oxide ( NO ) is traditionally utilizedto causepulmonary vasodilation
narrow thorax [ 5(passive) caused bypulmonary hypoplasia
pharmacologic agentscausepulmonary vasodilation
A ] ZIEGLER JAMES W ET AL , " Dipyridamole , a cGMP phosphodiesterase inhibitorcausespulmonary vasodilation
McMurtry I.F. , Morris K.G. Platelet - activating factorcausespulmonary vasodilation
a diaphragmatic defect , or(passive) caused bypulmonary hypoplasia
no effect on the hypoxic pulmonary vasoconstrictor response ... insufficient nitric oxide escapes red cell scavengingto causepulmonary vasodilation
Three sets of data ... Hemodynamics following inhalation of 100 % FiO2causespulmonary vasodilation
The use of bronchodilatorscan causepulmonary vasodilation
Use of iNOcausespulmonary vasodilation
ROCHEFORT AND MICHELAKISAbstractFulltextPDFThiol oxidationcausespulmonary vasodilation
increased oxygen tension , nitric oxide , alkalosis , and shear stress(passive) caused bypulmonary vasodilation
An increase of oxygen in the alveolicausespulmonary vasodilation
c.╇ viagra on line orderpreventingpulmonary hypoplasia
birth - related stimuli in the ovine fetus American Journal of Physiology - Heart and Circulatory Physiology May 1997 , 272 ( 5(passive) caused bypulmonary vasodilation
KCNK9 ( TASK-3 ) in Ovarian CarcinomaA Critical Role for the Protein Apoptosis Repressor With Caspase Recruitment Domain in Hypoxia - Induced Pulmonary HypertensionThiol oxidationcausespulmonary vasodilation
some COPD patients ... agentscausepulmonary vasodilation
to increased pulmonary vascular resistance ( PVR ) , pulmonary arterial hypertension ( PAHleadingto increased pulmonary vascular resistance ( PVR ) , pulmonary arterial hypertension ( PAH
increased pulmonary vascular resistance ( PVR ... and decreased pulmonary blood flowcausesincreased pulmonary vascular resistance ( PVR ... and decreased pulmonary blood flow
Foetal or neonatal death(passive) is caused byFoetal or neonatal death
increased pulmonary vascular resistance ( PVR ) , adaptation of right ventricular ( RV ) pump functioncausingincreased pulmonary vascular resistance ( PVR ) , adaptation of right ventricular ( RV ) pump function
in respiratory insufficiency and pulmonary hypertensionresultsin respiratory insufficiency and pulmonary hypertension
in persistent pulmonary hypertension of neonates ( PPHN ) , which is the main contributor to both high mortality and morbidityresultingin persistent pulmonary hypertension of neonates ( PPHN ) , which is the main contributor to both high mortality and morbidity
to respiratory insufficiency and deathmost often leadsto respiratory insufficiency and death
to an increased ventilation - perfusion mismatch and hypoxiamay leadto an increased ventilation - perfusion mismatch and hypoxia
to respiratory distress after birthmay leadto respiratory distress after birth
to respiratory distress 1leadingto respiratory distress 1
to severe respiratory failureleadsto severe respiratory failure
Respiratory distress in the delivery room(passive) may be caused byRespiratory distress in the delivery room
respiratory failure [ 7,8may causerespiratory failure [ 7,8
from oligohydramnios or anhydramniosresultingfrom oligohydramnios or anhydramnios
to severe respiratory insufficiency at birthleadingto severe respiratory insufficiency at birth
in an increase in pulmonary vascular resistance causing pulmonary hypertension , hemolytic anemia , and DICresultingin an increase in pulmonary vascular resistance causing pulmonary hypertension , hemolytic anemia , and DIC
mortality rate(passive) caused bymortality rate
to increased pulmonary vascular resistance , right heart failure , and death in 30–60 % of PAH patients five years after diagnosisleadingto increased pulmonary vascular resistance , right heart failure , and death in 30–60 % of PAH patients five years after diagnosis
to increased pulmonary vascular resistance , right heart failure and death in 40 - 60 % of PAH patients five years after diagnosisleadingto increased pulmonary vascular resistance , right heart failure and death in 40 - 60 % of PAH patients five years after diagnosis
to a variety of ailments after birth , including pulmonary hypertension , or high blood pressure in the lungs , respiratory failure , feeding problems and developmental delayscan leadto a variety of ailments after birth , including pulmonary hypertension , or high blood pressure in the lungs , respiratory failure , feeding problems and developmental delays
to death in early neonatal lifeleadingto death in early neonatal life
in a perinatal mortality of about 20 % in ARPKDresultingin a perinatal mortality of about 20 % in ARPKD
from severe oligohydramnios in utero secondary to inadequate urinary outputresultingfrom severe oligohydramnios in utero secondary to inadequate urinary output
from oligohydramnios due to certain fetal urinary tract anomaliesresultsfrom oligohydramnios due to certain fetal urinary tract anomalies
from PROM associated with severe oligohydramnios of as short as 6 days durationcan resultfrom PROM associated with severe oligohydramnios of as short as 6 days duration
in an increase of pulmonary pressure and pulmonary vascular resistance that might lead to right ventricle failure and eventually death [ 1resultingin an increase of pulmonary pressure and pulmonary vascular resistance that might lead to right ventricle failure and eventually death [ 1
from less ramification , both bronchiolar and vascularresultsfrom less ramification , both bronchiolar and vascular
to a worsening ventilation - perfusion mismatchleadingto a worsening ventilation - perfusion mismatch
soft tissue calcificationcausessoft tissue calcification
from the discordant ventriculo - arterial connectionsresultingfrom the discordant ventriculo - arterial connections
to these abnormalitiesmay contributeto these abnormalities
systemic arterial blood from being oxygenated What are some signs of a bad pulmonary transition into postnatal lifepreventingsystemic arterial blood from being oxygenated What are some signs of a bad pulmonary transition into postnatal life
to higher oxygen requirementsleadingto higher oxygen requirements
to the narrowing of the lumenleadingto the narrowing of the lumen
from the lungs having to develop within the thoracic volume of half a conjoined body cavityostensibly resultsfrom the lungs having to develop within the thoracic volume of half a conjoined body cavity
pulmonary pressurespreventspulmonary pressures
when lungs are compressed by abnormally positioned abdominal viscera and can not develop normally or expand at birthresultswhen lungs are compressed by abnormally positioned abdominal viscera and can not develop normally or expand at birth
to a series of larger pulmonary veinsleadto a series of larger pulmonary veins
in increased pulmonary blood pressure and reduced delivery of oxygenated blood to the bodyresultingin increased pulmonary blood pressure and reduced delivery of oxygenated blood to the body
from the compression of the developing lungs by the herniated viscera ( 5 , 20resultingfrom the compression of the developing lungs by the herniated viscera ( 5 , 20