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Mitochondrial uncoupling proteins ( UCPscreateproton leaks
OB_005285 Mitochondrial uncoupling proteinscreateproton leaks
The T9101C mutationcausesproton leakage
the Nav1.5 / R219H mutationcreatesa proton leak
specific membrane proteins - uncouplers ( uncoupler proteins - UCPs(passive) is caused byProton leakage
a predominant obstaclepreventingproton leakage
The periplasmic gate is tightly sealedpreventingproton leakage
a change in inner membrane surface area ... by whichmay influenceproton leak
the proposed role of Glu286 ... a robust gating valvepreventsproton leakage
a waypreventsproton leakage
GCs influence ion transport of plasma cells via direct contact with the cell membrane , and direct contact with the mitochondrial membranescausesproton leakage
cooverproduction of MotA with the MotB - TetA chimera protein ( 3 , 36 , 45(passive) caused byproton leakage
The enzyme ANT2causesproton leakage
Thus , the water orientation is essentialto preventproton leakage
UCP4createsproton leaks
to site 1 in the oxidized state ( step II → Ito preventproton leakage
The positively charged , strictly conserved aArg239 in H5 separates the lumenal and matrix channelspreventingproton leakage
protons ... other wordsto createa proton leak
forming a sealto preventproton leakage
The latterresultedfrom a proton leak
Mitochondrial depolarizationcan originatefrom a proton leak
that the hermetic seal of the micro - chamber with a lipid bilayer was sufficiently tightto preventproton leakage
as ANT may also be expectedto contributeto the proton leak
the drugs or their combination(passive) caused byproton leak
This mechanismpreventsproton leak
more energy ... the reactionswould causeproton leakage
UCP-2 upregulationcontributingto proton leak
proton uncoupling in the mitochondrial inner membrane in brown fat and beige fat(passive) caused byproton leak
O2 consumptionresultingfrom proton leak
IM of mammals , including humans -These proteinscreatea “ proton leak
protein 1 ( UCP1causesmitochondrial proton leak
F. tularensis subsp(passive) triggered bythe proton leak
hypoxia +(passive) caused byproton leak
corresponding increases in OCRresultingfrom the proton leak
The maximum respiratory capacity ... the respirationresultsin proton leak
in decreased ATP production efficiency and decreased Complex IV capacityresultingin decreased ATP production efficiency and decreased Complex IV capacity
to instability of V(REST ... indirectly ... for instance ... by interfering with intracellular pH homeostasismay contributeto instability of V(REST ... indirectly ... for instance ... by interfering with intracellular pH homeostasis
in poor fuel conversion efficiencyresultingin poor fuel conversion efficiency
to less reactive oxygen species ( ROS ) productionleadsto less reactive oxygen species ( ROS ) production
to the reduction in mitochondrial potential and ATP productionleadingto the reduction in mitochondrial potential and ATP production
from a heat - dependent alteration of the permeability of the thylakoid membranesoriginatingfrom a heat - dependent alteration of the permeability of the thylakoid membranes
in heat generation ( Rial and Gonzalez - Barroso , 2001resultsin heat generation ( Rial and Gonzalez - Barroso , 2001
the losses(passive) caused bythe losses
to membrane depolarization ( mild uncouplingleadingto membrane depolarization ( mild uncoupling
in the depletion of membrane potential ( 18 , 46resultingin the depletion of membrane potential ( 18 , 46
to instability of VREST indirectly ... for instance ... by interfering with intracellular pH homeostasismay contributeto instability of VREST indirectly ... for instance ... by interfering with intracellular pH homeostasis
to instability of the resting membrane potential by interfering with pH balance inside the cellcould ... contributeto instability of the resting membrane potential by interfering with pH balance inside the cell
OCRATP and OCR(passive) caused byOCRATP and OCR
to diminished MMP , which in turn is an early event in the apoptotic processleadsto diminished MMP , which in turn is an early event in the apoptotic process
to reduction of ATP production [ 31leadingto reduction of ATP production [ 31
to heat production for endothermy in this species ... if the measured state 4 respiration rates and membrane potentials reflect the conditions under which the mitochondria operate in vivocontributesto heat production for endothermy in this species ... if the measured state 4 respiration rates and membrane potentials reflect the conditions under which the mitochondria operate in vivo
to energy dissipation ( 3leadingto energy dissipation ( 3
directly to the induction of apoptosis ( 47 , 48can contributedirectly to the induction of apoptosis ( 47 , 48
The oligomycin - insensitive OCR itself(passive) caused byThe oligomycin - insensitive OCR itself
the effective P / O ratio in hepatocytes to be substantially less than the theoretical maximum valuecausesthe effective P / O ratio in hepatocytes to be substantially less than the theoretical maximum value
to a partial compensation of the increase in V̇o2 caused by an elevated energy demandleadsto a partial compensation of the increase in V̇o2 caused by an elevated energy demand
to the energetics of the Mx2 cellscontributed significantlyto the energetics of the Mx2 cells
in an increase of mitochondrial couplingresultingin an increase of mitochondrial coupling
to zero , a stoichiometry increase from 3:2 to 3:1setto zero , a stoichiometry increase from 3:2 to 3:1
to ∼25 % of the resting oxygen consumption of mammalscontributesto ∼25 % of the resting oxygen consumption of mammals
an increase in mitochondrial energetic efficiency and an increase in ROS contentcausedan increase in mitochondrial energetic efficiency and an increase in ROS content
to approximately 25 % of the resting respiratory rate [ 24contributesto approximately 25 % of the resting respiratory rate [ 24