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that triggered STAT3 activationto causehRMVEC proliferation
fertilization from the egg cellsetsendosperm proliferation
membrane proteinscausingconstitutive proliferation
their abilityto causeendothelial proliferation
the key mechanismsprovokeglial proliferation
the production of cytokinesinfluenceosteoclast proliferation
likelyinfluenceglial proliferation
the proliferating cell nuclear antigen ( ... ) and anti - apoptotic B - cell lymphoma protein 2causingproliferation
Soma - germline interactionsinfluencegermline proliferation
To determinecan influenceglial proliferation
apoptotic or anti - apoptotic factors ... , and survival of the cell and its response to toxic agentsinfluencethe proliferation
This results in a new , activated , protein that produces new proteinsthereby causingproliferation
transcription factors , promoters , mitogenic agents , apoptotic or anti - apoptotic factors ... , and survival of the cell and its responseinfluencethe proliferation
transcription factors , promoters , mitogenic agents , apoptotic or anti - apoptotic factors ... , and survival of the cell and its response to toxic agentsinfluencethe proliferation
During the inflammation stage of the cycle , new cells are producedcausingproliferation
This cytokinecausesfibroblast proliferation
The MAPK Pathway Activation of MAPKcausesproliferation
a new , activated , protein that produces new proteinsthereby causingproliferation
which can be even an acute infectioncausinghistiocytic proliferation
These T effectorscausedfibroblast proliferation
non - hematopoietic cellscausingthe proliferation
channel block - induced mammalian neuroblastoma cell ... a possible mechanismto influenceproliferation
the addition of pharmacological agents to the culture medium ... , differentiation and/or survival of MSCsinfluencesthe proliferation
human bone marrow cells ... sufficientto causeproliferation
due to failure of the checkpoints during the cell cycle that ensure normal division of a cell in the body(passive) is causedProliferation
Genes and cancer : Genes and cancer Oncogenes ( tumor causing genesCauseproliferation
transcription factors , promoters , mitogenic agents , apoptotic or anti - apoptotic factors ... , and survival of the cell and its response to toxic agents and endogenous stressorsinfluencethe proliferation
by microenvironmental changes brought about by interactive signaling between BMPR2 mutant vascular cells and macrophages(passive) caused byproliferation
a robust Th1 antigen - specific cellular response(passive) caused byproliferation
pathway ... antigen - stimulated mature T lymphocytesto provokeproliferation
these pathway genescausingproliferation
the over - expression of p53(passive) caused byproliferation
by the over - expression of p53(passive) caused byproliferation
signaling mechanismsto causeproliferation
Variations in these pathway genescausingproliferation
Increased apoptosis and decreasedcontributeproliferation
Most functions of these proteins are connected to signal transmission across the membrane down to the level of gene expressionthereby influencingproliferation
the host immune responses and expression of viral genes(passive) is influenced byThe proliferation
that constitutive activation of PKC activates ERK1/2to causeproliferation
constitutive activation of PKC activates ERK1/2to causeproliferation
to a selectivity window of > 122-foldleadingto a selectivity window of > 122-fold
tractional [ [ retinal detachmentcan also causetractional [ [ retinal detachment
tractional retinal detachmentcan ... causetractional retinal detachment
in the formation of a second neointimaresultingin the formation of a second neointima
in arterial stenosisresultingin arterial stenosis
retinal detachmentcan also causeretinal detachment
to thickened pleura that can encase the lungleadingto thickened pleura that can encase the lung
to tractional retinal detachment and macular puckercan leadto tractional retinal detachment and macular pucker
tractional retinal detachment or macular puckercan causetractional retinal detachment or macular pucker
to narrowing of the vascular luminaleadingto narrowing of the vascular lumina
traction retinal detachmentcausingtraction retinal detachment
a tractional RDcan causea tractional RD
as a result of elevated PDGFcan causeas a result of elevated PDGF
the tractional detachmentcausingthe tractional detachment
in an approximate 3.5-fold total expansion of these cells within the infarction area compared with the uninjured heart and that this enhanced cell content was maintained long term within the scar without cell turnoverresultedin an approximate 3.5-fold total expansion of these cells within the infarction area compared with the uninjured heart and that this enhanced cell content was maintained long term within the scar without cell turnover
from the anterior retinaoriginatingfrom the anterior retina
to cellular expansion and accumulation of tissue massleadingto cellular expansion and accumulation of tissue mass
in new tissue growthresultingin new tissue growth
to an increase in tissue sizeleadsto an increase in tissue size
to the generation of complex fractionated electrograms in humans ( 40 ... and that fibroblast ion - channel remodeling might significantly alter the properties and persistence of AF ( 41may contributeto the generation of complex fractionated electrograms in humans ( 40 ... and that fibroblast ion - channel remodeling might significantly alter the properties and persistence of AF ( 41
to lesion formation and restenosisleadingto lesion formation and restenosis
to lower numbers of cellsleadingto lower numbers of cells
to apoptosis of neoplastic cellsleadingto apoptosis of neoplastic cells
to fibrosis and sclerosis with subsequent loss of organ functionoften leadingto fibrosis and sclerosis with subsequent loss of organ function
to an unnecessary increase in complexitywill leadto an unnecessary increase in complexity
to an expansion of immature cellsleadingto an expansion of immature cells
to malignant cancerleadsto malignant cancer
to reduced tumor cell growthleadingto reduced tumor cell growth
in a higher cell densityresultingin a higher cell density
significant diseasecausingsignificant disease
restenosis of the treated area ( closure of an artery through cellular proliferationmay causerestenosis of the treated area ( closure of an artery through cellular proliferation
in powerful cell regenerationresultingin powerful cell regeneration
growth of the ductal tree ... and inducing apoptosis , which is required for lumen formationcausesgrowth of the ductal tree ... and inducing apoptosis , which is required for lumen formation
to the formation of [ [ extracellular matrixleadsto the formation of [ [ extracellular matrix
to the reduction of tumorsleadingto the reduction of tumors
to cyst formationleadsto cyst formation
significant disease [ 3causingsignificant disease [ 3
to the loss of a cellular pattern over timecontributeto the loss of a cellular pattern over time
to a proliferative vascular remodelingleadingto a proliferative vascular remodeling
in the formation of aresultingin the formation of a