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Ejection Fraction Analysis of FactorsCan InfluenceEjection Fraction
The relatively low rates of risk factors , the small infarct sizecould ... have influencedthe preserved ejection fraction
If EDV , or preload , is increased , the Frank - Starling mechanism will increase stroke volume such that ESV remains the sameresultingin a preserved ejection fraction
I / R injury(passive) caused byejection fraction
transverse aortic constriction and deoxycorticosterone infusion(passive) created bya preserved ejection fraction
to 33 % , 45 % , or 60 %(passive) was setEjection fraction
impaired ischemic myocardial contractility(passive) caused byejection fraction
changes in loading conditions ( higher preload , lower afterload(passive) caused byejection fraction
heart ratesettingEjection fraction
4Factorsinfluencingejection fraction
adversely(passive) was influencedEjection fraction
respiratory - induced intrathoracic pressure changes during normal free breathing(passive) caused byejection fraction
Volle(passive) Created ByPreserved Ejection Fraction ( HFpEF
an identical cardiac myosin - binding protein C gene abnormality(passive) caused bypreserved LV ejection fraction
a reduction of systolic dimensions(passive) mainly caused byejection fraction
that some pioneering trials of autologous bone marrow stem cells have unexplained discrepancies that cast doubt on their validity.5 discrepancies was not possible to report this directly in the journals that published the trials.5 Discrepancies in reports have never been systematically explored as a possible explanatory variable for the effect size of autologous bone marrow stem cells on ejection fraction(passive) has recently been discoveredejection fraction
hardening of heart muscles(passive) caused bypreserved ejection fraction
a virus(passive) caused byejection fraction
to systolic heart failureleadsto systolic heart failure
pharmacologic stress on the heartCausespharmacologic stress on the heart
a 60 to 75 percentsettinga 60 to 75 percent
from diastolic function abnormalities in patients with left ventricular outflow tract obstruction.51resultingfrom diastolic function abnormalities in patients with left ventricular outflow tract obstruction.51
ischemiacauseischemia
the risk of rehospitalization ( P=0.035influencedthe risk of rehospitalization ( P=0.035
in pulmonary edema and abnormal cardiac screening testsresultingin pulmonary edema and abnormal cardiac screening tests
independentlyinfluencedindependently
treatment discontinuation in everolimus - treated patients and one in the placebo groupcausedtreatment discontinuation in everolimus - treated patients and one in the placebo group
from reduced LV cavity sizeresultingfrom reduced LV cavity size
to episodes of arrhythmia , progressive pump failureleadingto episodes of arrhythmia , progressive pump failure
in better survival rateresultingin better survival rate
to 33 , 45 , and 60 %setto 33 , 45 , and 60 %
in substantial morbidity and mortalityresultingin substantial morbidity and mortality
the hemodynamic consequences of arrhythmias , both supraventricular and ventricularmay influencethe hemodynamic consequences of arrhythmias , both supraventricular and ventricular
to decompensation , valvular damage , or worse arrhythmias.6leadingto decompensation , valvular damage , or worse arrhythmias.6
to trastuzumab interruptionleadingto trastuzumab interruption
from therapy with nonglycosidic positive inotropic agentsresultingfrom therapy with nonglycosidic positive inotropic agents
in alcoholic cardiomyopathyresultingin alcoholic cardiomyopathy
breathing problems and swelling in the feet and legscan causebreathing problems and swelling in the feet and legs
a significant misinterpretation of the actual pathophysiologycausesa significant misinterpretation of the actual pathophysiology
to a subgroup - treatment interaction in the main SOLVD publicationsledto a subgroup - treatment interaction in the main SOLVD publications
topamaxdoes ... causetopamax
patients with symptomatic HF(passive) caused bypatients with symptomatic HF