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Qaagi - Book of Why

Causes

Effects

the multiple pathwaysleadingto platelet activation

the signaling pathwaysleadto platelet activation

intracellular signal transduction pathwaysresultin platelet activation

many pathwaysleadingto platelet activation

intracellular pathwaysleadingto platelet activation

classical pathwaysleadingto platelet activation

several pathwaysleadingto platelet activation

different signalling pathwayscontributeto platelet activation

Many agonistsleadto platelet activation

the several signal transduction pathwaysleadingto platelet activation

the addition of brazilin or various agoniststo triggerplatelet activation

platelets ... adhesion receptorsleadto platelet activation

the specific downstream pathways of VPS34 signalingleadingto platelet activation

different agonists ( Figure 1C(passive) triggered byplatelet activation

interactions between platelets and collagenresultin platelet activation

an organized signaling cascadeleadsto platelet activation

agonists such as thrombin or by contact with the extracellular matrix(passive) caused byPlatelet activation

a sequence of eventsresultsin platelet activation

thromboxane A2-independent pathways , such as adenosine diphosphate or thrombin(passive) caused byplatelet activation

Simultaneous binding of these antibodies to Fc receptors on the platelet surfacecausesplatelet activation

vivo evidenceleadsto platelet activation

stimulating platelets in a platelet sampleto causeactivation of the platelets

the platelets ... in turnleadsto platelet activation

platelets and bacterialeadingto platelet activation

platelets ... the subendotheliumleadsto platelet activation

virions and plateletsleadingto platelet activation

A number of physiological agonistscan causeplatelet activation

either von Willebrand factor interactions with GP Ibα and GP IIb - IIIa[1–4(passive) triggered byplatelet activation

All the platelet agonists , either directly or indirectly , depend on G protein pathwaysto causeplatelet activation

alpha-6-fucosyltransferase ... thrombin or other agonistscauseplatelet activation

an intracellular signaling pathwayresultingin platelet activation

the intracellular signalscontributeto platelet activation

contact of collagen ( of the plaques extracellular matrix ... the bloodcan triggerplatelet activation

several agonists including ADP , thromboxane A2 , collagen , thrombin , epinephrine and others(passive) is caused byPlatelet activation

a broad variety of events(passive) is caused byPlatelet activation

that promotes thrombinsignaling leadingto platelet activation

intracellular processesleadingto platelet activation

Both processes are believedto contributeto platelet activation

When present , these antibodies bind to the heparin / PF4 complex on the surface of platelets and crosslink Fc receptors on the platelet surfacethereby causingplatelet activation

ADP and the von Willebrand factor(passive) is triggered byPlatelet activation

to platelet aggregation and clotleadingto platelet aggregation and clot

platelet aggregation and adhesion to the blood vessel wallpreventingplatelet aggregation and adhesion to the blood vessel wall

to optimal thrombus formationleadingto optimal thrombus formation

to arterial thrombus formationleadsto arterial thrombus formation

in decreased tendency of platelets to adhere to one another and to damaged blood vessels endothelium.[2 ] 4.1 Prevention and treatment of arterial thrombosis 5 Dental Management of Patients on Antiplatelet Drugs 5.1 Assessresultingin decreased tendency of platelets to adhere to one another and to damaged blood vessels endothelium.[2 ] 4.1 Prevention and treatment of arterial thrombosis 5 Dental Management of Patients on Antiplatelet Drugs 5.1 Assess

to whacking great platelet aggregationleadsto whacking great platelet aggregation

in platelet aggregation , Neuropathology , Supercentenarian , Tauresultedin platelet aggregation , Neuropathology , Supercentenarian , Tau

to platelet aggregation and formation of a primary hemostatic plugleadsto platelet aggregation and formation of a primary hemostatic plug

to clot formation and thrombosisleadingto clot formation and thrombosis

to the link between depression , thrombus formation , and haemostasismay contributeto the link between depression , thrombus formation , and haemostasis

to enhanced aggregation and thrombosis [ 7leadingto enhanced aggregation and thrombosis [ 7

to fibrinogen cross - linking of platelets and platelet aggregationleadingto fibrinogen cross - linking of platelets and platelet aggregation

to thrombus formation on medical device surfacesleadsto thrombus formation on medical device surfaces

to a conformational shape change of the platelet , thrombin generation , and platelet aggregationleadsto a conformational shape change of the platelet , thrombin generation , and platelet aggregation

to increased thrombosisleadingto increased thrombosis

to thrombosis and embolizationleadingto thrombosis and embolization

to change in platelet size , platelet shape , platelet aggregationleadsto change in platelet size , platelet shape , platelet aggregation

in platelet aggregation | Identification and Characterization of Inhibitors of Human Apurinicresultedin platelet aggregation | Identification and Characterization of Inhibitors of Human Apurinic

in platelet aggregation ... but it is blocked by anti - Fc receptor CD32resultedin platelet aggregation ... but it is blocked by anti - Fc receptor CD32

to their aggregation and clot formationleadsto their aggregation and clot formation

to secretion and supports platelet aggregation thereby protecting against bleeding during inflammationleadsto secretion and supports platelet aggregation thereby protecting against bleeding during inflammation

a cascade of further platelet activation eventually causing the formation of the thrombus.[1 ] This process is regulated through thromboregulationcausesa cascade of further platelet activation eventually causing the formation of the thrombus.[1 ] This process is regulated through thromboregulation

in less thrombus formation or clotting in catheter - based productsresultingin less thrombus formation or clotting in catheter - based products

to activation and propagation of the complement system | Journal ofleadsto activation and propagation of the complement system | Journal of

to the clinical manifestations of thrombocytopenia and thrombosisleadsto the clinical manifestations of thrombocytopenia and thrombosis

Particularly , arterial thrombosis is considered(passive) to be triggered byParticularly , arterial thrombosis is considered

p1442 Individuals with mechanical heart valve implants(passive) are ... caused byp1442 Individuals with mechanical heart valve implants

to conformational changes in the structure of integrin β3 , which enhances platelet aggregation [ 25leadsto conformational changes in the structure of integrin β3 , which enhances platelet aggregation [ 25

to conformational changes in the structure of integrin αⅡbβ3 , which enhances platelet aggregation[27leadsto conformational changes in the structure of integrin αⅡbβ3 , which enhances platelet aggregation[27

to up - regulation of PrPc not only on CD55 + but surprisingly also on CD55−plateletsledto up - regulation of PrPc not only on CD55 + but surprisingly also on CD55−platelets

to dangerous blood clottingcan leadto dangerous blood clotting

to activation and propagation of the complement system del Condeleadsto activation and propagation of the complement system del Conde

to a hypercoagulable stateleadsto a hypercoagulable state

1442 - 1450 Individuals with mechanical heart valve implants(passive) are ... caused by1442 - 1450 Individuals with mechanical heart valve implants

in the form of degranulation and adhesionleadsin the form of degranulation and adhesion

to thrombosis but not essential for hemostasiscontributingto thrombosis but not essential for hemostasis

in thrombosis [ 10 - 12resultingin thrombosis [ 10 - 12

in platelet aggregation | Comparison of the Cancer Gene Targeting and Biochemical Selectivities Supplementary MaterialsSupporting Informationresultedin platelet aggregation | Comparison of the Cancer Gene Targeting and Biochemical Selectivities Supplementary MaterialsSupporting Information

to activation and propagation of the complement system | JEM jem Homeleadsto activation and propagation of the complement system | JEM jem Home

in platelet degranulation and aggregationfinally resultingin platelet degranulation and aggregation

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