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cytokinescausevasoconstriction , platelet activation
the factcausesreduced platelet activation
The proposed mechanism for acute coronary syndrome following rituximab infusion ... cytokinescausevasoconstriction , platelet activation
The proposed mechanism for acute coronary syndrome following rituximab infusion ... cytokines , ... and/orcausevasoconstriction , platelet activation
The proposed mechanism of myocardial infarction for ACS following rituximab infusion ... cytokinescausevasoconstriction , platelet activation
The proposed mechanism of myocardial infarction for ACS following rituximab infusion ... cytokines , ... and/orcausevasoconstriction , platelet activation
by an overall inhibition in platelet response(passive) may be caused byimpaired platelet activation
PlateletsresultsVascular Platelet activation
9 healthy volunteerscausedenhanced platelet activation
Heparin - induced thrombocytopenia ( HIT ) is usually caused by anti - platelet factor 4 ( PF4)/heparin antibodiesleadingto intravascular platelet activation
the pathwaysleadingto platelet integrin activation
occurs when IgG antibodies bind to heparin - platelet factor 4 complexleadingto platelet activation
when IgG antibodies bind to heparin - platelet factor 4 complexleadingto platelet activation
a method for interfering in coagulation of blood comprising ... , expression of Tissue Factor , release of Tissue Factor , and/or activation of Tissue Factorinfluencingplatelet activation
This condition ... platelet factor 4 / heparin complexescan ... leadto platelet activation
a complex signaling cascaderesultsin platelet activation
platelets ... platelet receptorsleadsto platelet activation
a number of intracellular signaling eventsleadto platelet activation
interaction - induced signalingleadingto platelet activation
IgG antibodies ... the heparin - platelet factorleadsto platelet activation
cAMP levels in human plateletsthereby leadingto platelet activation
Moreover , LPS treated mice have increased thrombin activationleadingto platelet activation
heparin - dependent IgG ( HIT - IgG ) that recognizes a Complex of heparin and Platelet factor 4 ( PF4leadingto Platelet activation
ADP interacts with a family of ADP receptors ... plateletsleadsto platelet activation
Activation of coagulation , leading to generation of thrombincontributesto platelet activation
The ADP released following endothelial injury binds to purinergic receptors P2Y1 and P2Y12leadingto platelet activation
mitochondrial complex V inhibitioncontributesto platelet activation
This process activates the coagulation cascadecausingplatelet activation
platelet receptorscausingplatelet activation
platelet receptorsleadsto platelet activation
when specific heparin - dependent antibodies attach to platelet membranescausingplatelet activation
the signaling eventscontributeto platelet activation
abnormal platelet functionleadingto platelet activation
the presence of HIT antibody immune complexescauseplatelet activation
Fc - independent cross - linking of a novel platelet membrane protein by a monoclonal antibodycausesplatelet activation
. - Treatment of platelets with Aledto platelet activation
complement activationleadsto platelet activation
the mechanisms ... platelet apoptosisleadingto platelet activation
factors released during red cell destructionleadsto platelet activation
an interaction between platelets and collagenresultsin platelet activation
persistent mucosal inflammationmay causepersistent mucosal inflammation
to thrombosisleadingto thrombosis
to tumor disseminationcontributesto tumor dissemination
to deathleadingto death
to thrombus formationcould leadto thrombus formation
generation of procoagulant microparticles which is responsible for the formation of platelet - rich arterial as well as venous blood clotsalso causesgeneration of procoagulant microparticles which is responsible for the formation of platelet - rich arterial as well as venous blood clots
to the inflammatory recruitment ofcontributeto the inflammatory recruitment of
microthrombi formationcausingmicrothrombi formation
in subsequent platelet dysfunction following surgeryresultsin subsequent platelet dysfunction following surgery
to platelet degranulationleadsto platelet degranulation
alsoleadsalso
them changecausesthem change
them to transform formcausesthem to transform form
to a hypercoagulable stateleadsto a hypercoagulable state
finallyresultingfinally
a cascade of further platelet activation ... eventually causing the formation of the thrombuscausesa cascade of further platelet activation ... eventually causing the formation of the thrombus
the release of thromboxane A2 ( TXA2 ) and other mediators to allow recruitment at the vascular injury sitecausesthe release of thromboxane A2 ( TXA2 ) and other mediators to allow recruitment at the vascular injury site
howeverleadshowever
to inflammation blood clots forming [ 4leadto inflammation blood clots forming [ 4
inflammation or if inflammation causes platelet activationcausesinflammation or if inflammation causes platelet activation
if inflammation causes platelet activationcausesif inflammation causes platelet activation
in platelet aggregation migration and platelet activationresultedin platelet aggregation migration and platelet activation
to thrombosis and activation of the inflammatory systemcan leadto thrombosis and activation of the inflammatory system
a cascade of further platelet activationcausesa cascade of further platelet activation
to activation and propagation of the complement system Platelet activation leads to activation and propagation of the complement system leadsto activation and propagation of the complement system Platelet activation leads to activation and propagation of the complement system
to activation and propagation of the complement system Platelet activation leads to activation and propagation of the complement systemleadsto activation and propagation of the complement system Platelet activation leads to activation and propagation of the complement system
the release of proinflammatory mediators such as sphingosine-1-phosphate ( S1Pcausesthe release of proinflammatory mediators such as sphingosine-1-phosphate ( S1P
to the formation of potentially life - threatening blood clotscan leadto the formation of potentially life - threatening blood clots
to decreased ability of stored platelet to functioncontributesto decreased ability of stored platelet to function
to inflammatory pathways as well as activation of coagulationalso contributesto inflammatory pathways as well as activation of coagulation
to increased c - src kinase activity and association of c - srcleadsto increased c - src kinase activity and association of c - src
to activation and propagation of the off systemleadsto activation and propagation of the off system
to intravascular coagulation or changes in protein composition of plasmaleadingto intravascular coagulation or changes in protein composition of plasma
to rapid , small platelet clearancecontributingto rapid , small platelet clearance
to activation and propagation of the complement systemleadsto activation and propagation of the complement system
to activation and propagation of the complement system leadsto activation and propagation of the complement system
to activation and propagation of the complement systemleadsto activation and propagation of the complement system
to activation and propagation of the complement system leadsto activation and propagation of the complement system
to activation and propagation of the complement system leadsto activation and propagation of the complement system
to activation and propagation of the complement systemleadsto activation and propagation of the complement system