Loading ...
However , anythinginfluencesplasma protein level
diagnosis and subsequent problems with accurate measurement ... anythinginfluencesplasma protein level
Because patients with proteinuria ,resultingin the loss of plasma protein
massive proteinuriacauseslow plasma protein
excessive loss of protein in urine(proteinuria(passive) can be caused byDecreased plasma proteins
These changes in pressureresultfrom loss of plasma proteins
the kidneysresultsin loss of plasma protein
of several proteins that can be denatured by high temperatures ( > 200 � F.(passive) is composedPlasma protein
Premature platelet activation as well ascould resultplasma protein denaturation
Hypoalbuminaemiamay resultfrom loss of plasma protein
in turnpreventsplasma loss ( albumin
substance P in a vein or by trigeminal ganglion stimulation(passive) caused byplasma protein extravasation
kidney or liver disease(passive) caused bylow plasma protein (
severe malnutrition , including low protein intake and nutrition loss because of infectious diseases such as Dengue and malariacauseslow plasma protein
LC - MS / MS evaluation of SAMs = 3(passive) discovered byPlasma transmembrane protein
a combination of symptoms some time(passive) caused byplasma protein levels
variantsinfluencingplasma protein levels
the actual diseasecausinglow plasma protein
For instance , VEGF , also known as permeability factor , increases endothelial permeabilitycausingplasma protein extravasation
electrical stimulation of meninges [ 89 , 90(passive) caused byplasma protein extravasation
LC - MS / MS evaluation of SAMs Enrichment of tetraspanins in SAMs(passive) discovered byPlasma transmembrane protein
The construction toolsto causedamage the plasma protein
the xenobiotics(passive) caused byplasma protein carbonyls
the ability of PEGto preventplasma protein denaturation
electrical stimulation of the trigeminal ganglion in guinea pigs(passive) caused byplasma protein extravasation
regulation during human hemoglobin switching;and 2discoverplasma protein biomarkers
a syndrome caused by urine(passive) caused byplasma protein loss
to retinal edemacontributedto retinal edema
to edema , ascites ( pooling of fluid in the abdominal cavity ) , hypotension , and susceptibility to infectionleadsto edema , ascites ( pooling of fluid in the abdominal cavity ) , hypotension , and susceptibility to infection
to edema , ascites ( pooling of fluid in the abdominal cavity ) , hypotension , best binary optionleadsto edema , ascites ( pooling of fluid in the abdominal cavity ) , hypotension , best binary option
cirrhosis - Malnutrition edema(passive) caused bycirrhosis - Malnutrition edema
to fewer binding sitesleadto fewer binding sites
to edema , ascites , hypotension , and susceptibility to infection ( because of immunoglobulin lossleadsto edema , ascites , hypotension , and susceptibility to infection ( because of immunoglobulin loss
fluid to be pulled into the patient 's intracellular spaces in order for osmotic fluid pressures to become equalized and result in the edemawould causefluid to be pulled into the patient 's intracellular spaces in order for osmotic fluid pressures to become equalized and result in the edema
in more free drug circulatingresultin more free drug circulating
a decrease in oncotic ( colloid osmotic ) pressure which promotes further fluid movementcausesa decrease in oncotic ( colloid osmotic ) pressure which promotes further fluid movement
to reduced oncotic pressureleadsto reduced oncotic pressure
to decrease in blood volume ( hypovolemia ) andleadto decrease in blood volume ( hypovolemia ) and
proteinuria(passive) caused byproteinuria
to rapid white matter injury through inducing a cascade of acute inflammatory events including oxidative stress , proinflammatory cytokine gene expression , and DNA damage within 24 h after ICHledto rapid white matter injury through inducing a cascade of acute inflammatory events including oxidative stress , proinflammatory cytokine gene expression , and DNA damage within 24 h after ICH
to a hypercoagulable stateleadsto a hypercoagulable state
swelling and breakdown of certain blood cell nuclei in vitrocausesswelling and breakdown of certain blood cell nuclei in vitro
the hypo - osmolar state(passive) caused bythe hypo - osmolar state
the hypoosmolar state(passive) caused bythe hypoosmolar state
the treatment of hypoproteinaemia(passive) caused bythe treatment of hypoproteinaemia
rapid mitosis of the hepatocytes and actual growth of liver , which is coupled with increase in synthesis of plasma proteins until blood values are again normal · Transaminationcausesrapid mitosis of the hepatocytes and actual growth of liver , which is coupled with increase in synthesis of plasma proteins until blood values are again normal · Transamination
in binding to proteins over 90 %resultingin binding to proteins over 90 %
rapid mitosis of the hepatic cells and growth of the liver to a larger sizecausesrapid mitosis of the hepatic cells and growth of the liver to a larger size
strokes are removed postmortem , carrierscausesstrokes are removed postmortem , carriers
extensive foaming in the purge vessel and that this interferes with the chemical reduction of [ NOcausesextensive foaming in the purge vessel and that this interferes with the chemical reduction of [ NO
about 7.030(passive) more influenced byabout 7.030
the formation of antibodiesinfluencethe formation of antibodies
from the patientscreatedfrom the patients
mainlyresultsmainly
a milky white viscous gelto createa milky white viscous gel
from increased intraoperative bleedingresultingfrom increased intraoperative bleeding
from the inhibition of tachykinin releaseresultingfrom the inhibition of tachykinin release
the increase in the δγ - T lymphocytes in Peyer ’s patches and in lamina propria , which supports the hypothesis that a SDP - supplemented diet can play a role in the modulation of the immune responsepreventsthe increase in the δγ - T lymphocytes in Peyer ’s patches and in lamina propria , which supports the hypothesis that a SDP - supplemented diet can play a role in the modulation of the immune response
to blood clot formationcontributesto blood clot formation
to heart diseaseleadsto heart disease
plasma ( 1.025 - 1.030 )(passive) more influenced byplasma ( 1.025 - 1.030 )
cognitive and a thiazolidine ringwill causecognitive and a thiazolidine ring
drug distribution and the relationship between total drug concentrations and its pharmacological effectscan influencedrug distribution and the relationship between total drug concentrations and its pharmacological effects
the mechanical properties of the glycocalyx and suggest a possible origin for its stiffnessmay influencethe mechanical properties of the glycocalyx and suggest a possible origin for its stiffness
a large number of urinary protein(passive) caused bya large number of urinary protein
drug binding.89 Considering the concentration time courses of AEDscan influencedrug binding.89 Considering the concentration time courses of AEDs
The monomeric form of this acute phase(passive) is composedThe monomeric form of this acute phase