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Factors ... free drug concentrationinfluencedrug binding to plasma proteins
by a syndrome caused by the disease(passive) caused byplasma protein loss
hemorrhage(passive) can be caused byPlasma protein loss
36 Plasma proteinwill causebinding Extensive plasma protein binding
Iron deficiencyleadsto low plasma bound protein
7 Medicinal chemistry optimization of WM-8014designedto reduce plasma - protein binding
knownto influenceMPA plasma protein binding
the situationscausedaltered plasma protein binding
massive proteinuriacauseslow plasma protein
a large amount of proteinurialeadsto low plasma protein
A quantitative trait locusinfluencingfree plasma protein
the relationship between total and free MPA pharmacokinetics ... knownto influenceMPA plasma protein binding
likely ... the free concentrations of drugsto influenceplasma protein binding and
Factorsinfluencingplasma protein binding
componentsinfluencingplasma protein binding
1 mg of 2.5 mg per day or higherresultedin plasma protein binding
The distributionleadsto plasma protein binding
doses of 2.5 mg per day or higherresultedin plasma protein binding
30 mg doses of 2.5 mg per day or higherresultedin plasma protein binding
mg / ml doses of 2.5 mg per day or higherresultedin plasma protein binding
of two identical molecular halves each consisting of three non - identical A alpha- , B beta- and gamma - chain subunits held together by multiple disulfide bonds(passive) is composedFibrinogen , a 340-kDa plasma protein
70 mg uses doses of 2.5 mg per day or higherresultedin plasma protein binding
side effects of 2.5 mg per day or higherresultedin plasma protein binding
muscular doses of 2.5 mg per day or higherresultedin plasma protein binding
500 mg doses of 2.5 mg per day or higherresultedin plasma protein binding
Studies administering fibroblast growth factor 8 assay doses of 2.5 mg per day or higherresultedin plasma protein binding
hyperglycemiacausingnonenzymatic glycation of plasma proteins
orange pill of 2.5 mg per day or higherresultedin plasma protein binding
Substance Pcausesplasma protein extravasation
Oxidative stresscausesplasma protein modification
Oxidative stresscausesplasma protein modification
Studies administering how much does methotrexate pill cost doses of 2.5 mg per day or higherresultedin plasma protein binding
capillary permeabilitycausingextravasation of plasma proteins
a capillary leak syndromeresultingfrom extravasation of plasma proteins
Damage to the alveolar wallscausesextravasation of plasma proteins
Action on venulesleadsto extravasation of plasma proteins
Factors ... binding , such as acidosis , systemic diseases that alter protein production , or competition of other drugs for protein binding sitesinfluencingplasma protein
Factors ... binding , such as acidosis , systemic diseases which alter protein production , or competition of other drugs for protein binding sitesinfluencingplasma protein
Factors ... binding , such as acidosis , systemic diseases that alter protein production or competition with other drugs for protein binding sitesinfluencingplasma protein
Factors ... , such as acidosis , systemic diseases which alter protein production , or competition of other drugs for protein binding sitesinfluencingplasma protein
the distribution , clearance , and efficacy of a drugmay significantly influencethe distribution , clearance , and efficacy of a drug
efficacy , distribution and eliminationcan influenceefficacy , distribution and elimination
the compounds to have reduced distribution in the plasmamay causethe compounds to have reduced distribution in the plasma
the compounds to have reduced distribution in the plasmamay causethe compounds to have reduced distribution in the plasma
decreased amino acidscausesdecreased amino acids
Drug effectiveness(passive) might be influenced byDrug effectiveness
to swift excretionleadingto swift excretion
mGluR1s(passive) was ... caused bymGluR1s
in increased plasma clearanceresultedin increased plasma clearance
in higher levels of free circulating drugsresultsin higher levels of free circulating drugs
to increase in freeleadto increase in free
renal clearancecan influencerenal clearance
more drug to stay in thewill causemore drug to stay in the
alsoinfluencesalso
alsoinfluencesalso
your resultsinfluencingyour results
not only urate deposition but also the renal excretion of uric acidmay influencenot only urate deposition but also the renal excretion of uric acid
to an increase in free levels of these substancesleadsto an increase in free levels of these substances
to a higher free fraction of the drugleadingto a higher free fraction of the drug
in reduced pharmacologically active unbound drug and reduced insult to myocardial contractilityresultingin reduced pharmacologically active unbound drug and reduced insult to myocardial contractility
The stereoselective brain distribution indicated by the K(p ) of 0.22 and 0.04 for S- and R - CZE(passive) respectively ... was caused byThe stereoselective brain distribution indicated by the K(p ) of 0.22 and 0.04 for S- and R - CZE
to retinal edemacontributedto retinal edema
displacement interactions More than one drug can bind to the same site on albumincan causedisplacement interactions More than one drug can bind to the same site on albumin
the tissue distribution , clearance , and pharmacological effects of drugscan greatly influencethe tissue distribution , clearance , and pharmacological effects of drugs
betterpromptingbetter
side effectscausingside effects
The stereoselective brain distribution indicated by the Kp of 0.22 and 0.04 for S- and R - CZE , respectively(passive) was caused byThe stereoselective brain distribution indicated by the Kp of 0.22 and 0.04 for S- and R - CZE , respectively
displacement interactionscan causedisplacement interactions
alsoinfluencesalso
in preparationresultingin preparation
to a few clientsleadingto a few clients
to maximal plasma - free concentrations that are insufficient to inhibit OAT1leadsto maximal plasma - free concentrations that are insufficient to inhibit OAT1
in fibrinogen - mediated platelet aggregation and development of stable thrombusresultsin fibrinogen - mediated platelet aggregation and development of stable thrombus
blood clotscan causeblood clots
to heart diseaseleadsto heart disease
to increased levels of free concentrations of drugsleadingto increased levels of free concentrations of drugs
in excessive free quinine and perhaps quinine toxicityresultingin excessive free quinine and perhaps quinine toxicity
to the displacement of one drug by anotherleadingto the displacement of one drug by another
to about 50 %contributesto about 50 %
in a more predictable responseresultsin a more predictable response