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genetic mutations(passive) can be caused byPlasma exchange
as an outpatient(passive) can be createdPlasma exchange
the neurologistto setup a plasma exchange
liver IR(passive) caused byplasma MIP-2
if the torsion is the expression of the induced mesenchyme expression of(passive) can be preventedPlasma exchange
because many cardiovascular risk factors are knownto influenceplasma PAI-1
fluid volumes of 1.2 times that predicted by the Parkland formula(passive) was triggered byPlasma exchange
barotrauma(passive) can be caused byPlasma exchange
dehydration and tongue - biting ... scarringpreventingplasma haptoglobin
Escalating psychotic symptoms and behavioural disturbancepreventedplasma exchange
left loin mass index of malasezia furfur / pityrospo- rum orbiculare(passive) is caused byPlasma exchange
in marked clinical improvementresultedin marked clinical improvement
an interference with the function of the voltage - dependent potassium channels , one of the underlying issues of hyper - excitability in autoimmune Neuromyotonia.[8 ] Botox injections also provide short - term reliefcausesan interference with the function of the voltage - dependent potassium channels , one of the underlying issues of hyper - excitability in autoimmune Neuromyotonia.[8 ] Botox injections also provide short - term relief
to a higher rate of renal recovery than intravenous methylpredisolone in severe vasculitis Home » COMMENTARYledto a higher rate of renal recovery than intravenous methylpredisolone in severe vasculitis Home » COMMENTARY
in partial clinical improvementresultedin partial clinical improvement
in temporary clinical improvementresultedin temporary clinical improvement
in a further , significant clinical improvementresultedin a further , significant clinical improvement
to a higher rate of renal recovery than intravenous methylpredisolone in severe vasculitis ACP Journal Club;Jan /ledto a higher rate of renal recovery than intravenous methylpredisolone in severe vasculitis ACP Journal Club;Jan /
an interference with the function of the voltage - dependent potassium channels , one of the underlying issues of hyper - excitability in autoimmune neuromyotonia.[6causesan interference with the function of the voltage - dependent potassium channels , one of the underlying issues of hyper - excitability in autoimmune neuromyotonia.[6
to a dramatic improvementledto a dramatic improvement
to recovery from thrombocytopenia and improvement in renal functionledto recovery from thrombocytopenia and improvement in renal function
in an improvement in the treatment effectresultingin an improvement in the treatment effect
to a rapid improvement of weakness during exacerbationsleadto a rapid improvement of weakness during exacerbations
a rapid but temporary improvement in myastheniacan causea rapid but temporary improvement in myasthenia
in full recovery from a bedridden statusresultedin full recovery from a bedridden status
rapid improvement without new deteriorationcausedrapid improvement without new deterioration
in an impressive improvement in consciousness as well as the laboratory abnormalitiesresultedin an impressive improvement in consciousness as well as the laboratory abnormalities
bleeding and allergic reactions , and bleeding and allergic reactions can make your chance of getting an infection highercan causebleeding and allergic reactions , and bleeding and allergic reactions can make your chance of getting an infection higher
to recovery from an acute attack in people with MS who have not responded to standard steroid treatmentmight contributeto recovery from an acute attack in people with MS who have not responded to standard steroid treatment
to functionally important neurological recovery in an important proportion of severely disabled patients with acute attacks of idiopathic inflammatory demyelinating diseaseleadsto functionally important neurological recovery in an important proportion of severely disabled patients with acute attacks of idiopathic inflammatory demyelinating disease
in a consistent strong reduction of proteinuria for all of themresultingin a consistent strong reduction of proteinuria for all of them
in a rapid anti - AQP4 reactivity decline ( patient 9resultedin a rapid anti - AQP4 reactivity decline ( patient 9
to relapsesrepeatedly ledto relapses
up for her alongside consistent physical therapysetup for her alongside consistent physical therapy
immune complex - mediated tissue injurymay preventimmune complex - mediated tissue injury
the course of the diseaseinfluencedthe course of the disease
to stabilizationledto stabilization
the rebound phenomenon that may be observed after the aphaeresis discontinuationcan preventthe rebound phenomenon that may be observed after the aphaeresis discontinuation
signiicontributesignii
in an immediate decrease in total plasma bilirubin from 466 μmol / Lresultedin an immediate decrease in total plasma bilirubin from 466 μmol / L
in resolution of symptoms and laboratory abnormalitiesresultedin resolution of symptoms and laboratory abnormalities
injury mediated by sepsis or by an unknown immune mechanism in severe leptospirosispreventsinjury mediated by sepsis or by an unknown immune mechanism in severe leptospirosis
hepatitis C viremia(passive) caused byhepatitis C viremia
in significant reductions in plasma viscosityresultedin significant reductions in plasma viscosity
to catheter- and plasma - related complications ( 2323can leadto catheter- and plasma - related complications ( 2323
a drop in levels of calcium in the bloodmay causea drop in levels of calcium in the blood
in a rapid decrease in symptoms and is an important modality in the treatment of acute exacerbationscan resultin a rapid decrease in symptoms and is an important modality in the treatment of acute exacerbations
the bowel and warm them take parai lm of appropriate history doing the dialysisto causethe bowel and warm them take parai lm of appropriate history doing the dialysis
additionallycontributesadditionally
an inhibition with the function of the voltage - dependent potassium channels , one of the basic outcome of hyper - excitabilitycausesan inhibition with the function of the voltage - dependent potassium channels , one of the basic outcome of hyper - excitability
in an approximately 50 % decline in eculizumab concentrations following a 1 hour intervention and the elimination half - life of eculizumabresultedin an approximately 50 % decline in eculizumab concentrations following a 1 hour intervention and the elimination half - life of eculizumab