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Capacitive Ca2 + entry ( CCE ... depletion of intracellular Ca2 + storescausesthe activation of plasma membrane Ca2 + channels
Capacitive Ca(2 + ) entry ( CCE ... depletion of intracellular Ca(2 + ) storescausesthe activation of plasma membrane Ca(2 + ) channels
However , as INPP4B generates a membrane - bound pool of PtdIns3P , INPP4B was predictedto triggerSGK3 activation through hydrolysis of plasma membrane - bound PtdIns(3,4)P2
NOX2 activationresultingfrom Rac1 activation and p47phox translocation to the plasma
in guard cellsleadsto activation of plasma membrane Ca2 + channels
that this is reversed by TSAleadingto the activation of CIITA and class II in plasma cells
radicalsoriginatingfrom the silane / hydrogen plasma with surfaces of crystalline silicon substrates and a - Si
a meshwork that is kept hydrated and act as an anticlogging agentto preventhydrogen bonding and adsorption of anionic plasma proteins
Antigen stimulationcan causeplasma cell activation and producing corresponding Immue globulin
restoring cellular ATP / ADP to control levelsresultingin persistent plasma membrane KATP channel activation and vasodilation
in better bondabilitycan resultin better bondability
a rise in [ Ca2+]i to mediate this effecttriggereda rise in [ Ca2+]i to mediate this effect
to cellular responses such as proliferation , differentiation , or increased resistance to apoptosiscan leadto cellular responses such as proliferation , differentiation , or increased resistance to apoptosis
to significant changes in the surface properties ( polarity , chemical composition , morphology , and roughness ) of the substratesledto significant changes in the surface properties ( polarity , chemical composition , morphology , and roughness ) of the substrates
only to an apparent specificity for anionic - hydrophilic surfaces that is actually due to a relative diminution of the FXII→surfaceFXIIa reaction at hydrophobic surfacesleadsonly to an apparent specificity for anionic - hydrophilic surfaces that is actually due to a relative diminution of the FXII→surfaceFXIIa reaction at hydrophobic surfaces
an area of continuing research C. EGF signaling(passive) is set ... byan area of continuing research C. EGF signaling
to increased C5a generationledto increased C5a generation
to Rapid Ca2 Export - Plasma Membrane Calmodulin Mediated Activation of Plasma Membrane Ca2 ATPaseLeadsto Rapid Ca2 Export - Plasma Membrane Calmodulin Mediated Activation of Plasma Membrane Ca2 ATPase
to reduced high - molecular - weight vWF multimers and impairment of the vWF - platelet aggregation pathway during mechanical circulatory supportmay have contributedto reduced high - molecular - weight vWF multimers and impairment of the vWF - platelet aggregation pathway during mechanical circulatory support
plasma damage to surfaces of the substrates before bondingcan causeplasma damage to surfaces of the substrates before bonding