A defect in this enzymeleadsto deficient mitochondrial nicotinamide adenine dinucleotide phosphate levels
4 ) Renal failurecausesretention of phosphate ... leading to secondary hyperparathyroidism
malnutrition(passive) are caused byPhosphate medicine Low phosphate syndromes
However , some patients using these calcium - based binders develop hypercalcemia , high calcium levelspromptinga search for other types of phosphate binders
with the aim of altering physicochemical properties and improving BPQ skin permeation(passive) were designedLess lipophilic phosphate prodrugs
Loss of renal functioncan leadto reduced phosphate excretion , phosphate dysregulation and
a dialysis populationdesignedto compare the phosphate binder sevelamer ( Renagel ) with Ca - containing phosphate binders
Conditionsleadto abnormal breakdown of phosphate
NHD are effective in terms of phosphate removalresultingin fewer phosphate binders being required
Spaindiscoveredhigh grade phosphate deposits at Bu Craa
the largest ever conducted in a dialysis populationdesignedto compare the phosphate binder sevelamer ( Renagel ) with Ca - containing phosphate binders
a high affinity transportercontributing greatlyto phosphate uptake in phosphate deprived plants
a genetic mutationcausesexcessive amounts of phosphate to build up in glycogen
Nitrate enrichment of the watercausesphosphate deficiency in Symbiodinium
The renal dysfunctionalso leadsto impairment in phosphate reabsorption
its abilityto causeintracellular phosphate depletion
by a defect in phosphate uptake(passive) caused byinsufficient intracellular phosphate
single gene defectresultingin dysfunctional nicotinamide adenine dineucleotide phosphate
to vascular oxidative stress in human coronary artery diseasecontributesto vascular oxidative stress in human coronary artery disease
at timescausingat times
reduced form oxidaseleadsreduced form oxidase
joint pain in some interested peoplemay causejoint pain in some interested people
congenital disorder of glycosylation typecausescongenital disorder of glycosylation type
congenital disorder of glycosylation type Iecausescongenital disorder of glycosylation type Ie
The sides(passive) are composedThe sides
to frank hypercalcemia that persisted despite low calcium dialysateledto frank hypercalcemia that persisted despite low calcium dialysate
hypercalcemiacausinghypercalcemia
hypercalcemiacan causehypercalcemia
in mean serum aluminiumresultedin mean serum aluminium
for smaller operations or educational needsdesignedfor smaller operations or educational needs
constipationcan causeconstipation
constipation(passive) caused byconstipation
bruising or purple hemorrhagic spots on the skinmay causebruising or purple hemorrhagic spots on the skin
constipationcan causeconstipation
constipationcan causeconstipation
constipationcan causeconstipation
to aluminum toxicity patientsmay leadto aluminum toxicity patients
hypercalcemiacan causehypercalcemia
probablycausedprobably
squealing and/or shedding of magnetic material onto playback device headscausessquealing and/or shedding of magnetic material onto playback device heads
damage to blood vessels and soft tissues , skeletal changes , kidney function , and risks of cardiovascular disease , bone pain , fractures , and death that accompany worsening kidney failureinfluencedamage to blood vessels and soft tissues , skeletal changes , kidney function , and risks of cardiovascular disease , bone pain , fractures , and death that accompany worsening kidney failure
in an amplicon of approximately 1264bp , which upon RFLP with HhaI restriction enzyme yields the following patterns : 2 bands ( 447 + 817 ) , 3 bands ( 253 + 447 + 490 ) , or 4 bands ( 253 + 447 + 490 + 817resultsin an amplicon of approximately 1264bp , which upon RFLP with HhaI restriction enzyme yields the following patterns : 2 bands ( 447 + 817 ) , 3 bands ( 253 + 447 + 490 ) , or 4 bands ( 253 + 447 + 490 + 817