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Qaagi - Book of Why

Causes

Effects

transcript levels of genesinfluencemitochondrial function and structure

the capabilityto influenceneuronal structure and functions

the peptides presenceinfluencesneuronal structure and functionality

s ... likelyto influencemicrobial community structure and function

how these factors combine with species interactionsto influenceintertidal community structure and function

heterogeneous nodescontributedifferentially to network structure and function

these irreversible modificationsleadto the alteration of protein structure and function

These changescan resultin disturbed tissue structure and function

the total bilayers [ 73,74leadingto altered membrane structure and function

gradientscontributeto spatial heterogeneity in structure and function

the viral infection(passive) caused bygut mucosal structure and function

cell identity ... terminally differentiated cellscontributeto tissue structure and function

trophic interactionsinfluencepelagic ecosystem structure and function

altering the tubulin polymerization equilibriumresultingin altered microtubule structure and function

inherited Diseasesinfluencecollagen structure and function

regenerative processesleadingto the reestablishment of hepatic structure and function

the complex cellular pathwaysinfluencemitochondrial structure and function

These proteins are thoughtto influencechromatin structure and function

these losseswill influenceecosystem structure and function

genesinfluencingmitochondrial structure and function

the major factorsinfluencingecosystem structure and function

proteinscontributeto nucleoid structure and function

to investigateinfluenceneuronal structure and function

mechanical signalsinfluencemitochondrial structure and function

the biochemical mechanisms by whichinfluenceschromatin structure and function

a RNA dependent regulating mechanisminfluencingchromatin structure and function

the unique phospholipid composition of its membranes(passive) are influenced byMitochondrial structure and function

the neurochemi - cal and molecular mechanismscontributeto impaired structure and function

the non - conservative replacement of the acidic amino acid glutamine with the cyclic amino acid proline at position 384causesa pathogenic alteration in myotubularin structure and function

The many cellular proteinsinfluencetelomere structure , function and

other bite issuesmay have ledto diminished structure and function

by HAMLET(passive) caused byproteasome structure and function

by HAMLET in tumor cells(passive) caused byproteasome structure and function

HAMLET in tumor cells(passive) caused byproteasome structure and function

One of the principal signals to which the endothelium is exposed , and , consequentlyinfluencesendothelial structure and function

appearto contributeto ciliary structure and function

by maternal caloric restriction(passive) caused byhypothalamic structure and function

by maternal caloric restriction during gestation(passive) caused byhypothalamic structure and function

maternal caloric restriction during gestation(passive) caused byhypothalamic structure and function

moderate maternal caloric restriction during gestation(passive) caused byhypothalamic structure and function

immediate irritationcausingimmediate irritation

defective microfibril productioncausesdefective microfibril production

its designinfluenceits design

to cellular signalling events by activating small G proteinscontributeto cellular signalling events by activating small G proteins

to cellular signalling events by activating small G proteinscontributeto cellular signalling events by activating small G proteins

to cellular signalling events by activating small G proteinscontributeto cellular signalling events by activating small G proteins

to cellular signalling events by activating small G proteinscontributeto cellular signalling events by activating small G proteins

cell deathwill causecell death

cell deathwill causecell death

origin activityinfluencesorigin activity

to improved activity and participationleadingto improved activity and participation

to improved activity and participation ( 3leadingto improved activity and participation ( 3

to improved physical activity and participationleadingto improved physical activity and participation

from their cancer operationcan resultfrom their cancer operation

one anotherinfluenceone another

to respiratory disease ... with the end goal of developing novel interventions for asthmacontributeto respiratory disease ... with the end goal of developing novel interventions for asthma

to major ocular diseaseslikely contributeto major ocular diseases

to teeth agenesisleadingto teeth agenesis

to teeth agenesis in OIleadingto teeth agenesis in OI

to electron leakage that reacts with molecular oxygen forming superoxide radical [ 2,3leadsto electron leakage that reacts with molecular oxygen forming superoxide radical [ 2,3

to heart failureleadingto heart failure

from common symptoms of Rheumatoid Arthritisresultingfrom common symptoms of Rheumatoid Arthritis

in altered gene expression and changes in cell behaviorresultingin altered gene expression and changes in cell behavior

to the increased cardiovascular morbidity and mortality seen in patients with atrial fibrillationmay contributeto the increased cardiovascular morbidity and mortality seen in patients with atrial fibrillation

to healthcontributeto health

to altered venous hemodynamicsleadsto altered venous hemodynamics

to an AMI in Drosophilacontributeto an AMI in Drosophila

in chronic oto - sinopulmonary diseaseresultsin chronic oto - sinopulmonary disease

to advances in many fields , especially pharmaceutical designleadingto advances in many fields , especially pharmaceutical design

to impaired mucociliary clearanceleadingto impaired mucociliary clearance

to adult diseasecontributingto adult disease

from their cancer operationcan resultfrom their cancer operation

from two centuries of anthropogenic activitiesresultingfrom two centuries of anthropogenic activities

in " leakage " of proteins normally released into the acinar ductsresultingin " leakage " of proteins normally released into the acinar ducts

in cutaneous diseaseresultingin cutaneous disease

to [ [ inflammation ] ] and [ [ fibrosisleadingto [ [ inflammation ] ] and [ [ fibrosis

to symptomsleadingto symptoms

to [ [ inflammationleadingto [ [ inflammation

to improved fitnessleadto improved fitness

to cell deathcontributeto cell death

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