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Protein - Protein Networks that Exclude or Trap Signaling Molecules in T Cells SummaryComments(passive) Created byPlasma Membrane Microdomains
protein - protein networks that exclude or trap signaling molecules in T cells , Cell 121(passive) created byplasma membrane microdomains
of two large subunits of 91 - 107 kDa , one medium chain ( mu ) of 47 - 50 kDa and one small chain ( sigma ) of 17 - 19 kDa(passive) are composedplasma membrane
salt stress(passive) caused byplasma membrane
USA300 supernatant(passive) caused byplasma membrane permeability
of a bilayer of phospholipids containing proteins , carbohydrates , and cholesterol and functions to provide a semipermeable barrier around the cell(passive) is composedplasma membrane
penetrating the bacterial cell membranecausingplasma membrane disintegration
of lipid and protein molecules(passive) is composedPlasma membrane
Step 4 : further myosin II activity causes the ring to further condensethereby causingconstriction of the plasma membrane
USA300 ( Figure 2(passive) caused byPMN plasma membrane permeability
Hla in conjunction with Annexin V binding and ApoBrdU Tunel assays examining PBMCs(passive) caused byplasma membrane permeability
Protein - Protein Networks that Exclude or Trap Signaling Molecules in T CellsCell , Volume 121 , Issue 6(passive) Created byPlasma Membrane Microdomains
Supernatants from M31A , W32A , and F33A Mutant Strains ... Unableto CausePMN Plasma Membrane Permeability
distinct means , including high extracellular K+ , the lipophilic cation tetraphenylphosphonium , or the ionophore nystatin(passive) provoked byplasma membrane depolarization
oxidative damage to lipids and proteins(passive) caused byincreased plasma membrane permeability
Lipid raftscreateplasma membrane heterogeneity
The cell wall on around each plant cellpreventsconstriction of the plasma membrane
Such binding of Al3 + to the cell wall as well ascausedplasma membrane
of DHA ( 1(passive) is composedplasma membrane
of a lipid bilayer that is not as rigid as a bacterial cell wall(passive) is composedPlasma membrane
In return , Yrt counteracts aPKC functionsto preventapicalization of the plasma membrane
Full Text Polyaminescauseplasma membrane depolarization
electroporation that enables … ( More)116View PDFRelated PublicationsMore(passive) caused byincreased plasma membrane permeability
Substitution of aspartic acid or lysine residues with glycine residues or scrambling of the amino acid sequence of the seven - amino acid regioncausedmistargeting to the plasma membrane
pri- principal parts : marily of phospholipid and protein molecules(passive) is composedplasma ) membrane
Colloid Interface Sci , 2013 ; Pottosin , I. , Velarde - Buendia , AM . , Bose , J. , Fuglsang , AT . , Shabala , S. Polyaminescauseplasma membrane depolarization
of a lipid layer which is semipermeable(passive) is composedPlasma membrane
single , intense , submicrosecond square wave pulses(passive) caused byplasma membrane permeabilization
of a double phospholipid layer w/ proteins embedded in the phospholipid layer(passive) is composedThe plasma membrane ( plasmalemma
unit membranesoriginatingat the hyphal plasma membrane
which flow into the cellcausinghyperpolarisation of the plasma membrane
different factorsmight influenceplasma membrane
BzATPcausespermeabilization of the plasma membrane
channel formation ... potassiumcausesplasma membrane depolarization
the lipid content(passive) is ... influenced byPlasma membrane fluidity
proteases and lipasescausingpermeabilization of the plasma membrane
SMase treatment(passive) caused byplasma membrane internalization
the extracellular matrix Despite the limited overlap with the excitation spectrum of TB ( Fig . 1 C(passive) is influenced byQuenching of plasma membrane
This influx in anionscausesa depolarization of the plasma membrane
Conditionscausehyperpolarization of the plasma membrane
Macromolecules|macromolecules ] ] , bound to receptor proteins ... to enter the [ [ Cell|cellcausesMacromolecules|macromolecules ] ] , bound to receptor proteins ... to enter the [ [ Cell|cell
from crypt cells and from villous cellsoriginatingfrom crypt cells and from villous cells
to recruitment of TNF receptor - associated death domain ( TRADDleadsto recruitment of TNF receptor - associated death domain ( TRADD
the plasma membrane 's interaction with the Cdk inhibitor p27 and therefore nuclear translocation of the cyclin D1-CDK4-p27 complex regulating transition throughpreventingthe plasma membrane 's interaction with the Cdk inhibitor p27 and therefore nuclear translocation of the cyclin D1-CDK4-p27 complex regulating transition through
in necrotic cell deathresultingin necrotic cell death
The generation of an action potential(passive) is triggered byThe generation of an action potential
to the formation of TEMs [ 19contributeto the formation of TEMs [ 19
in a sustained influx of Ca2resultingin a sustained influx of Ca2
reversible dispersion of the H+‐symporters , not however of the Sur7 proteincausesreversible dispersion of the H+‐symporters , not however of the Sur7 protein
to an influx of calciumledto an influx of calcium
of phospholipids and proteinsComposedof phospholipids and proteins
of proteins in the bilayeris composedof proteins in the bilayer
to oncotic / necrotic death in cancer cells ( 47leadingto oncotic / necrotic death in cancer cells ( 47
the opening of intracellular channels and enables the introduction of electroporated assetscausesthe opening of intracellular channels and enables the introduction of electroporated assets
to rapid closure of KATP channels , depolarization and exocytosisleadsto rapid closure of KATP channels , depolarization and exocytosis
in a flux of calcium and potassiumresultingin a flux of calcium and potassium
to the shedding of the vesicles3,4leadsto the shedding of the vesicles3,4
reversible dispersion of the proteinscausesreversible dispersion of the proteins
of microtubules and motor proteins andcomposedof microtubules and motor proteins and
of connexin ( Cx ) proteinscomposedof connexin ( Cx ) proteins
reversible dispersion of the H+ symporters previously present in 300-nm patches ( Grossmann et al . , 2007causedreversible dispersion of the H+ symporters previously present in 300-nm patches ( Grossmann et al . , 2007
to cell apoptosis processleadsto cell apoptosis process
to oncotic / necrotic death in cancer cells in caspase - dependent and caspase - independent pathways14,15leadingto oncotic / necrotic death in cancer cells in caspase - dependent and caspase - independent pathways14,15
to their specific association with transmembrane-4leadsto their specific association with transmembrane-4
of phospholipids with embedded and attached proteinscomposedof phospholipids with embedded and attached proteins
skeletal muscle contraction by triggering Ca2 + release from an intracellular membrane network , the sarcoplasmic reticulumcausesskeletal muscle contraction by triggering Ca2 + release from an intracellular membrane network , the sarcoplasmic reticulum
of a lipid bilayer housing different freely diffusing membrane proteinsis composedof a lipid bilayer housing different freely diffusing membrane proteins
to the activation of the JNK pathway and subsequent cell death via caspase-12 activation [ 121leadsto the activation of the JNK pathway and subsequent cell death via caspase-12 activation [ 121
of a membranous component , flavocytochrome b558 , and cytosolic proteinscomposedof a membranous component , flavocytochrome b558 , and cytosolic proteins
to the formation of pre - autophagosomal structures Ravikumar , Brinda | Moreau , Kevin | Jahreiss , Luca | Puri , Claudia | Rubinsztein , David C Nature cell biologycontributesto the formation of pre - autophagosomal structures Ravikumar , Brinda | Moreau , Kevin | Jahreiss , Luca | Puri , Claudia | Rubinsztein , David C Nature cell biology
to calcium and proton influx that appears to be necessary and sufficient for the induction of H2O2leadingto calcium and proton influx that appears to be necessary and sufficient for the induction of H2O2
of short carbohydrate chains that attach to membrane proteins and phospholipid moleculescomposedof short carbohydrate chains that attach to membrane proteins and phospholipid molecules
to formation of early endosomes , which may include transfer of plasma membrane proteinsleadto formation of early endosomes , which may include transfer of plasma membrane proteins
a strong enhancement of calcium oscillations in β - cells and a pronounced potentiation of synaptic transmissiontriggersa strong enhancement of calcium oscillations in β - cells and a pronounced potentiation of synaptic transmission
to increased transmembrane conductance , calcium influx , cell swelling and cytolysis [ 5 , 6leadingto increased transmembrane conductance , calcium influx , cell swelling and cytolysis [ 5 , 6
in activation of calcium entry channels mediating crucial long - term calcium signalsresultin activation of calcium entry channels mediating crucial long - term calcium signals
to intracellular calcium ( Ca2 + ) mobilization and several important cellular functions such as cell adhesion , secretion , and proliferation ( 43leadingto intracellular calcium ( Ca2 + ) mobilization and several important cellular functions such as cell adhesion , secretion , and proliferation ( 43
5 ] and receptor activation may lead to release of Ca 2 + from endoplasmic reticulum ( ER ) stores [ 3 , 4leads5 ] and receptor activation may lead to release of Ca 2 + from endoplasmic reticulum ( ER ) stores [ 3 , 4
to an osmotic water inflow , which in turn leads to cell swelling and increases the hydration of membrane compartments ( fatty acids and proteins ) , which are usually separated from excess amount of waterleadsto an osmotic water inflow , which in turn leads to cell swelling and increases the hydration of membrane compartments ( fatty acids and proteins ) , which are usually separated from excess amount of water
cell volume changes due to the so- called colloid osmotic mechanism [ 47 , 50also triggerscell volume changes due to the so- called colloid osmotic mechanism [ 47 , 50