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hyperglycemiaresultingfrom impaired insulin secretion , defects in insulin action
foodcausesinsulin release ... leading to insulin resistance
a chronic hyperglycemic conditionresultingfrom defects in insulin secretion , insulin action
excess proteincausesspikes in insulin for insulin resistant people
too much of itcan provokepancreas to manage insulin properly
intracellular accumulation of ceramidescan ... resultin insulin resistance in insulin responsive tissues
other genescausesusceptibility to insulin - dependent diabetes mellitus
the matrix effect ... the kinetics of insulin releaseinfluencesinsulin - release mechanisms
the most common endocrine disordersresultingfrom dysfunctions in the normal insulin mechanism
also i areto leadavoidance with pattern in insulin
autoimmune destruction of pancreatic beta cells by T cells(passive) is usually caused byInsulin - dependent diabetes mellitus
a frequent findingresultingfrom inadaequate insulin dosing in T1D.
Australian researchersdiscoverinsulin binding mechanism
the increased production of pro - inflammatory cytokines by adipose tissueinfluencesinsulin signaling in insulin - responsive tissues
damage ... the pancreasresultsin insulin - dependent diabetes mellitus
low blood sugars ... pillscauseinsulin to be released from the pancreas ( Insulin Secretagogues
Long - termmay ... leadcompensating insulin secretion to insulin resistance
Reserpine , fibrates , and MAOIsmay causedrug interactions with insulin lispro protamine / insulin lispro
In direct contrast , activation mutations increase KATP channel activityresultingin impaired insulin secretion , NDM
factorsinfluenceadherence to insulin regimens
this genemight contributeto the impaired insulin secretion of NIDDM
of 3 segments , the A - chain , B - chain , and C - peptide(passive) is composedProinsulin , the precursor of insulin
blood sugar levels ... a mealcausespancreas to release insulin
hormonescauserelease of insulin from the islets of Langerhans
Incretins ... hormonescauserelease of insulin from the islets of Langerhans
This condition(passive) can be caused byThis condition
to a decrease in functionalleadsto a decrease in functional
in a reduced rate of overall symptomatic hypoglycemiaresultedin a reduced rate of overall symptomatic hypoglycemia
to the uptake of glucosecontributingto the uptake of glucose
to an impairment of insulin signalingledto an impairment of insulin signaling
in an increase in blood sugar ( hyperglycaemia ) and sugar appearing in the urine ( see GLYCOSURIAresultingin an increase in blood sugar ( hyperglycaemia ) and sugar appearing in the urine ( see GLYCOSURIA
to the enzymatic latencyledto the enzymatic latency
prolonged reduction of blood glucose levels in diabetic ratscausedprolonged reduction of blood glucose levels in diabetic rats
laminitiscan causelaminitis
to weight losscan leadto weight loss
to chronically elevated blood glucose and associated health consequencescan leadto chronically elevated blood glucose and associated health consequences
to many health issues including type II diabetesleadsto many health issues including type II diabetes
in improved glycemic index and lipid profileresultingin improved glycemic index and lipid profile
in lower rates of overall symptomatic hypoglycemic episodes and nocturnal symptomatic hypoglycemia in the 16-week maintenance period and a lower proportion experienced severe hypoglycemia during theresultedin lower rates of overall symptomatic hypoglycemic episodes and nocturnal symptomatic hypoglycemia in the 16-week maintenance period and a lower proportion experienced severe hypoglycemia during the
obesitycausesobesity
satiety in the short - termcausessatiety in the short - term
some unwanted effectsmay causesome unwanted effects
to increased blood glucose levels during stressleadsto increased blood glucose levels during stress
in lower rates of overall symptomatic hypoglycemic episodes and nocturnal symptomatic hypoglycemia in the 16-week maintenance period and a lower proportion experienced severe hypoglycemia during the maintenance period ( 10 percent vs 17 percentresultedin lower rates of overall symptomatic hypoglycemic episodes and nocturnal symptomatic hypoglycemia in the 16-week maintenance period and a lower proportion experienced severe hypoglycemia during the maintenance period ( 10 percent vs 17 percent
in increased tracer uptakeresultsin increased tracer uptake