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Qaagi - Book of Why

Causes

Effects

Stimulation of these receptorsleadsto increased parasympathetic outflow

causes binding of vesicles containing acetylcholine to the pre synaptic membranecausingrelease of acetylcholine

specific receptorscausingrelease of acetylcholine

Motilin receptor activationcausesrelease of acetylcholine

Arrival of nerve impulsecausesrelease of acetylcholine

Variability in lake levelhas causeddisruption of the outflow

reversible carboxylation of the acetylcholinesterase enzyme(passive) is caused byAcetylcholine accumulation

Menmay also causeparasympathetic dysfunction

irreversibly and non - competitively inhibit acetylcholinesterase , causing poisoning by phosphorylating the serine hydroxyl residue on AChE , which inactivates AChE. AChE is critical for nerve function ... this enzymecausesacetylcholine accumulation

ventricular tachycardiaoriginatingfrom the ventricular outflow

the right internal jugular veincausingvenous outflow

by a narrowing of the vein or partial thrombosis(passive) caused byvenous outflow

that depression , panic disorders and anxiety have negative impact on autonomic functiontypically causingdepletion of parasympathetic tonus

that depression , panic disorders and anxiety have negative impact on autonomic functiontypically causingdepletion of the parasympathetic tonus

Catecholamines and nerve stimulation have been shownto provokesecretion of prostaglandins

by factors such as the gravid uterus(passive) caused byvenous outflow

the arteriesmay leadto venous outflow

the craniosynostosis(passive) caused bythe venous outflow

Pain , fear and several other stressorscan causesecretion of epinephrine

the brain or pulmonary diseasescauseadh secretion

by cells(passive) caused byparasympathetic stimula- tion

the hormonecausesreleasing hypothalamus

aneurysmcausingobstructive outflow

cardiac pacing modemay influencesympathetic outflow

many similar or different mechanismscausingionospheric outflow

heating upto causehydrodynamic outflow

elevated venous pressure , such as in patients with Sturge - Weber syndrome(passive) caused byimpeded outflow

by butaprost(passive) caused byuveoscleral outflow

to decreased heart rateleadingto decreased heart rate

in a acute hydrocephalusresultingin a acute hydrocephalus

to obstruction ofleadingto obstruction of

to a decrease in the heart ratesleadto a decrease in the heart rates

to a decrease in the heart ratesleadto a decrease in the heart rates

in retrograde venous drainage and venous hypertension andresultsin retrograde venous drainage and venous hypertension and

to ischemia and necrosisleadingto ischemia and necrosis

to acute graft dysfunctionleadingto acute graft dysfunction

to increased icpleadingto increased icp

further swelling and increased pressurecausesfurther swelling and increased pressure

to ischemialeadingto ischemia

ischemiacausesischemia

an increase in the pressure inside the right ventricle , which in turn leads to right ventricular hypertrophy or enlargement ( RVH).causesan increase in the pressure inside the right ventricle , which in turn leads to right ventricular hypertrophy or enlargement ( RVH).

an increase in the pressure inside the right ventricle , which in turn leads to right ventricular hypertrophy or enlargement ( RVHcausesan increase in the pressure inside the right ventricle , which in turn leads to right ventricular hypertrophy or enlargement ( RVH

in flap failureresultingin flap failure

to excessive slowing of heart rateleadingto excessive slowing of heart rate

to hypoxialeadingto hypoxia

in increased pressureresultsin increased pressure

in the deep vascular complex ( DVCoriginatingin the deep vascular complex ( DVC

to wall thickeningleadingto wall thickening

constriction of the pupilcauseconstriction of the pupil

in congestion and further swellingresultingin congestion and further swelling

diseasecausingdisease

in decrease blood pressureresultingin decrease blood pressure

to decrease blood pressureleadingto decrease blood pressure

seriouscausingserious

in lower blood pressureresultingin lower blood pressure

from T1 to T4originatingfrom T1 to T4

alsocould ... causealso

to a decrease in the heart ratesleadto a decrease in the heart rates

in preganglionic cell bodies in the midbrain and medullaoriginatingin preganglionic cell bodies in the midbrain and medulla

in preganglionic cell bodies in the midbrain and the medullaoriginatingin preganglionic cell bodies in the midbrain and the medulla

an increase in the excitability of upper motor neuronescausingan increase in the excitability of upper motor neurones

bradycardia or occasional asystole , hypotension , and salivationcan causebradycardia or occasional asystole , hypotension , and salivation

to an automatic bladderleadsto an automatic bladder

her to sweat excessivelycausingher to sweat excessively

to pupillary constrictionleadsto pupillary constriction

their functioninfluencestheir function

to thepressor responsecontributesto thepressor response

to constriction of the bronchileadingto constriction of the bronchi

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