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Intracellular Ca2 + releasesparksatrial pacemaker activity
Calcium ion entry through L - type Ca2 + channels in the sinoatrial ( SA ) nodecontributesto pacemaker activity
modulation of IP3Rs by ET-1(passive) influenced bypacemaker activity
a less negative maximum diastolic potential ( MDP ) and diastolic depolarizationresultedin pacemaker activity
heart muscle activity(passive) is triggered bythe pacemaker activity
interact with the exchanger current and the non - specific Ca2 + -activated currentto causepacemaker activity
enzymecan influencepacemaker activity
L - type ( ICa , L ) and T - type ( ICA , Tcontributeto pacemaker activity
The latter effect ... the currentscontributeto pacemaker activity
ionic currentscontributeto pacemaker activity
According to these findings , activation of mGluRIII was expectedto influencepacemaker activity
that both I(f ) andcontributeto pacemaker activity
a positive feedback regulation of L - type channelscontributesto the pacemaker activity
Several classes of ion channels and proteins involved in intracellular Ca(2 + ) dynamicscontributeto pacemaker activity
In the SA node , the role of ICa , like that of the other time - and voltage - dependent membrane currents , isto contributeto pacemaker activity
authors Frank Müller Hyperpolarization - activated and cyclic nucleotide - gated ( HCN ) channelscontributeto pacemaker activity
age(passive) is influenced dramatically byPacemaker activity
A fourth mechanism by whichcould contributeto pacemaker activity
The second impactinfluencingpacemaker activity
unitary currentscould contributeto pacemaker activity
Higher 1-EBIO concentrations ( 0.5–2 mm ... tonic membrane hyperpolarizationspreventedpacemaker activity
current ... the only currentcontributesto pacemaker activity
another type of mechanistic redundancy ... unlikelyto preventpacemaker activity altogether
patient 's cardiac electrical activitytriggeredpatient 's cardiac electrical activity
from the PNE ( Figure 2B shows one exampleoriginatingfrom the PNE ( Figure 2B shows one example
the rhythm and rate of cardiac chamber contractionsetsthe rhythm and rate of cardiac chamber contraction
from STICsresultsfrom STICs
in the sinus nodeoriginatesin the sinus node
from the coupling of cardiomyocytes with non excitable cells like fibroblasts ... and Kryukov , et alcan resultfrom the coupling of cardiomyocytes with non excitable cells like fibroblasts ... and Kryukov , et al
within the svoriginatingwithin the sv
to a computer diagnosis of wide complex rhythm and intraventricular block ... occasionally left bundle branch blockcan leadto a computer diagnosis of wide complex rhythm and intraventricular block ... occasionally left bundle branch block
in repetitive coiling of the trunkresultingin repetitive coiling of the trunk
from the coupling with nonexcitable cellsresultingfrom the coupling with nonexcitable cells
to computer misinterpretation of the underlying rhythmcan leadto computer misinterpretation of the underlying rhythm
as a consequence of a preceding impulse or series of impulses , without which electrical quiescence occursresultsas a consequence of a preceding impulse or series of impulses , without which electrical quiescence occurs
to a diagnosis of sinus rhythmleadsto a diagnosis of sinus rhythm
from conduction failure in the SACPs ( SAN exit blockresultingfrom conduction failure in the SACPs ( SAN exit block
to false computer diagnosis of myocardial infarction and hypertrophy patterns , which mislead the electrocardiographercan leadto false computer diagnosis of myocardial infarction and hypertrophy patterns , which mislead the electrocardiographer
ventricular contraction before the underlying rhythm would normally depolarise ventriclescausingventricular contraction before the underlying rhythm would normally depolarise ventricles
from multiple ectopic foci within the atriaoriginatingfrom multiple ectopic foci within the atria
waves of mitochondrial reactive oxygen species that might generate damage ( Guzman et al . , 2010causeswaves of mitochondrial reactive oxygen species that might generate damage ( Guzman et al . , 2010