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Irradiation of breast cancer xenograftscauseda phenotype conversion of non - stem breast cancer cells into induced breast cancer - initiating cells
The combination of ribociclib and fulvestrantresultedin tumor growth inhibition in estrogen receptor - positive breast cancer models
These findings highlight the potential of stromal fibroblaststo contributeto chemoresistance in breast cancer cells in part through fibroblast - induced HMGB1 production
the workresultedin the development of lapatinib for the treatment of breast cancer
activation of the hdm2-P2 promoter through multiple transcription factor response elementsresultsin elevated hdm2 expression in estrogen receptor alpha - positive breast cancer cells
the eventsleadto increased Notch activity during EMT of breast cancer cells
doxorubicinmight causea PI3-K - dependent increase of Akt activity in breast cancer cells
signalcausestumors to develop Breast cancer breakthrough
the downstream pathwayleadsto Akt activation in breast cancer cells after treatment with doxorubicin
Factorsinfluencingsurvival among patients with HER2-positive metastatic breast cancer treated with trastuzumab
that is consideredto contribute substantiallyto the colonisation of breast cancer cells into metastatic site[22
that is consideredto contribute substantiallyto the colonisation of breast cancer cells into metastatic site[22].
the identification of pathwayscontributeto the survival of breast cancer cells following therapy
inhibits tumor proliferationresultingin a survival benefit for HER2-positive breast cancer patients treated with anthracyclines
newer treatmentsresultingin increases in survival from breast cancer
Disruption of these pathwaysleadsto a malignant phenotype observed in breast cancer cells
genesresultingin enhanced development of breast cancer cells
additional targetis contributingto the overexpression of EGFR in Akt1 impaired breast cancer cells
a processcontributesto FGFR signaling in metastatic breast cancer
ABT 888 has additionally been notedto causesenescence when along with radiation in breast cancer cells
let-7f reductionmay leadto the augment of TSP-1 expression in breast cancer cells
the breast to appear red or inflamedcausingthe breast to appear red or inflamed
The Axxent Electronic Brachytherapy System , Xoft 's first treatment system(passive) is designedThe Axxent Electronic Brachytherapy System , Xoft 's first treatment system
a dose - dependent decrease in cell proliferationcauseda dose - dependent decrease in cell proliferation
the cells to be more susceptible to radiation treatmentcausedthe cells to be more susceptible to radiation treatment
in increased E - CADHERIN expression and a parallel reduction in their invasive capacityresultedin increased E - CADHERIN expression and a parallel reduction in their invasive capacity
to increase of cell invasionleadingto increase of cell invasion
in inhibition of apoptosis and acceleration of mitotic transitionsresultsin inhibition of apoptosis and acceleration of mitotic transitions
cancer cells to divide symmetrically , expanding the tumorcausescancer cells to divide symmetrically , expanding the tumor
to growth inhibition and induction of apoptosisleadsto growth inhibition and induction of apoptosis
a decrease in metastatic properties and the reemergence of normal cell propertiescausesa decrease in metastatic properties and the reemergence of normal cell properties
in selective target gene activationresultsin selective target gene activation
in selective target gene activationresultsin selective target gene activation
any excess mortality(passive) caused byany excess mortality
in significant apoptosisresultedin significant apoptosis
to a fundamental reduction of the risk of local relapsecontributesto a fundamental reduction of the risk of local relapse
to an inhibition in cancer progressionleadingto an inhibition in cancer progression
to the down - regulation of tumor aggressivenessleadingto the down - regulation of tumor aggressiveness
in an increased association of ERalpha with AIB1 as confirmed by co - immunoprecipitation assays from cell lysatesresultsin an increased association of ERalpha with AIB1 as confirmed by co - immunoprecipitation assays from cell lysates
to decreased proliferation and invasiveness in cell cultureledto decreased proliferation and invasiveness in cell culture
in an increased level of FGFR2resultedin an increased level of FGFR2
in destabilization of EZH2 , inhibition of cell proliferation and migration , and induction of cell deathresultedin destabilization of EZH2 , inhibition of cell proliferation and migration , and induction of cell death
in a POL3-driven increase in RN7SL1resultsin a POL3-driven increase in RN7SL1
to reduced tumour growth in distant organsleadingto reduced tumour growth in distant organs