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being recruited toward the neuronal promoterscausingneuronal differentiation.[5
certain two - pore domain potassium channels , which usually leak potassium out of the cell and help control the resting membrane potential ... acidcausesneuronal depolarization
the transmitter ... extracellular fluidwould causeneuronal depolarization
short term REM deprivationinfluencedbrainstem neuronal excitability
Our findings revealedcausedneuronal cytotoxicity
tDCSdoes ... causeneuronal depolarization
in turncausesupsurge in neuronal excitability
tACS phaseinfluencescortical neural excitability
several voltage - gated sodium channels ... likelyto contribute significantlyto neuronal excitability
although both activation and inactivation of voltage - gated Na+ channels are knownto contributeto neuronal excitability
Alzheimer 's disease progressioninfluencingneuronal proteolysis
While sodium channels are knownto influenceneuronal excitability
many of the voltage - gated sodium , potassium , and calcium channelsinfluencingneuronal excitability
calcium channelsinfluencingneuronal excitability
a reduction in neuronal inhibitioncan leadto neuronal excitability
both synaptic transmission and neuronal intrinsic properties(passive) is influenced byNeuronal excitability
gated M - channelinfluencesneuronal excitability
Na+ and K+ channels ... modulation of whichcan influenceneuronal excitability
Brain metabolismcan profoundly influenceneuronal excitability
Brain metabolismcan profoundly influenceneuronal excitability
This combinationcausesneuronal hyperexcitation
Calcium ( Ca2 +influencesneuronal excitability
the extracellular concentration of potassiuminfluencesneuronal excitability
Many of these mutationsshould leadto neuronal excitability
The membrane ion channelsinfluenceneuronal excitability
M - channelinfluencesneuronal excitability
other known mechanismscan influenceneuronal excitability
other known mechanisms ... , including the effects of transporters , peptide neurotransmitterscan influenceneuronal excitability
tonic inhibitioninfluencesneuronal excitability
the NMDA receptorcontributesto neuronal excitability
Astrocytic changes are knownto ... influenceneuronal excitability
in particular the extracellular concentration of potassiuminfluencesneuronal excitability
persistent inward currents , such as persistent sodium currentsinfluenceneuronal excitability
Kv7.2 ( KCNQ2 ... gated M - channelinfluencesneuronal excitability
that has been shownto influenceneuronal excitability
in turninfluencesneuronal excitability
Glial cellsinfluenceneuronal excitability
proteinsinfluenceneuronal excitability
factorscontributingto neuronal excitability
ion channelsinfluenceneuronal excitability
to neuronal discharge and the release of neurotransmittersleadsto neuronal discharge and the release of neurotransmitters
to enhanced glutamate release from the presynaptic cellcan leadto enhanced glutamate release from the presynaptic cell
in activation of Ca2resultedin activation of Ca2
to neurodegenerative diseasescontributingto neurodegenerative diseases
in a large influx of ionsresultingin a large influx of ions
to calcium influx that binds to calmodulinleadsto calcium influx that binds to calmodulin
to bradycardia and hypotensionleadingto bradycardia and hypotension
to intracellularleadsto intracellular
to the onset of neurological diseasescontributeto the onset of neurological diseases
to seizure - like behaviorleadingto seizure - like behavior
to seizureleadingto seizure
seizurescausingseizures
to behavioral deficitsleadingto behavioral deficits
in abnormal discharges and central sensitizationresultingin abnormal discharges and central sensitization
to the skinleadingto the skin
to inflammationleadingto inflammation
in thinning of cortical grey matterresultingin thinning of cortical grey matter
to transient change in blood flow in region of activation which results in differential oxygenated - to - deoxygenated hemoglobin concentrationleadsto transient change in blood flow in region of activation which results in differential oxygenated - to - deoxygenated hemoglobin concentration
in neurodegenerative disordersresultingin neurodegenerative disorders
at the synapseoriginateat the synapse
to altered communication at these connectionswill leadto altered communication at these connections
to synaptic plasticitycontributeto synaptic plasticity
at the synapse yetoriginateat the synapse yet
to synaptic plasticity or neuronal toxicitycontributeto synaptic plasticity or neuronal toxicity
to CNS or peripheral nerve damageleadingto CNS or peripheral nerve damage
in an increase in seizure activity.(5resultingin an increase in seizure activity.(5
in seizure activityresultingin seizure activity
to seizure activityleadingto seizure activity
to the generation of seizurescontributesto the generation of seizures
in an increase in seizureresultingin an increase in seizure
to sustained action potential firingleadingto sustained action potential firing
to spontaneous recurrent seizuresleadingto spontaneous recurrent seizures
to epileptic output , or seizure activitycan leadto epileptic output , or seizure activity
neurodegeneration(passive) caused byneurodegeneration
to seizuresleadsto seizures
to enhanced seizure susceptibilityleadsto enhanced seizure susceptibility
in seizure actionresultingin seizure action
to seizure actionleadingto seizure action
in abnormal spontaneous dischargeresultsin abnormal spontaneous discharge
in epilepsyresultingin epilepsy