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PubMed ABSTRACT Two - pore potassium channelscan influenceneuronal excitability
Full AccessRestricted Access A cytoplasmic intron - containing mRNA encoding a potassium channel subunitinfluencesneuronal excitability
Satellite glial cellsinfluenceneuronal excitability
Axon initial segment ( AIS ) geometrycritically influencesneuronal excitability
increased autophagy , apoptosis and proteasome activity Adropin acts in the rat paraventricular nucleusto influenceneuronal excitability
a multitude of neuroactive molecules ... in turnwill influenceneuronal excitability
authors Gary Yellen Brain metabolismcan profoundly influenceneuronal excitability
abnormal expression and function of membrane ion channels(passive) caused byneuronal excitability ,
the targeting of Nav β4 to the axon initial segmentshould ... influenceneuronal excitability
K+ channelsinfluenceneuronal excitability
Fitzgerald and Fulton 1992).One family of ion channelscan ... influenceneuronal excitability
10.7554 / eLife.32721 Brain metabolismcan ... influenceneuronal excitability
the activation of potassium channels and inhibition of voltage - gated calcium channels(passive) caused byneuronal excitability
many of the volt - age - gated sodiuan , potassium , and calcium channelsinfluencingneuronal excitability
Kv7.2 ( KCNQ2 ... gated M - channelinfluencesneuronal excitability
that both dopamine D1- and D2-receptor - expressing MSNs ( D - MSNs ) additionally harbor extrasynaptic GABAARs incorporating α4 , β , and δ subunits that mediate tonic inhibitionthereby influencingneuronal excitability
While sodium channels are knownto influenceneuronal excitability
Sally N. Lawson Ihinfluencesneuronal excitability
QxMD https://www.readbyqxmd.com/read/28100478/adropin-acts-in-the-rat-paraventricular-nucleus-to-influence-neuronal-excitability Adropin acts in the rat paraventricular nucleusto influenceneuronal excitability
a multitude of neuroactive molecules such as glutamate , ATP , nitric oxide ( NO ... in turninfluencesneuronal excitability
Inhibition of the Müller cell KIR channels by the neurotransmitter glutamate ... in turninfluencesneuronal excitability
authors G LeesBrain research2003Anandamide ... diffusible lipid moleculesinfluencesneuronal excitability
authors G. J. Lees Anandamide ... diffusible lipid moleculesinfluencesneuronal excitability
a variety of neuroactive molecules including glutamate , ATP , nitric oxide , prostaglandins , atrial natriuretic peptide ( ANP ) , and d - serine ... in turninfluencesneuronal excitability
the fraction of sodium channels that are available to open at subthreshold membrane potentials(passive) can also be influenced byNeuronal excitability
More ) Cristian G. Acosta , Simon McMullan , +5 authors Sally N. Lawson I(hinfluencesneuronal excitability
opioid posts outside the kind of brain nerve pathway activation emitted from the rod to the spinal cord(passive) caused byneuronal excitability
Their roles in dorsal root ganglion ( DRG ) neurons normally , and in … ( More ) Cristian Acosta , Simon McMullan , +5 authors Sally N. Lawson I(hinfluencesneuronal excitability
In both neocortical and amygdalar neurons , α - DTX - sensitive channels are strategically located at the soma ( Bekkers and Delaney 2001 ) or the primary apical dendrite ( Faber and Sah 2004to influenceneuronal excitability
an increase in threshold levels requiredto triggerneuronal excitability
extracellular pH.(passive) can be influenced byneuronal excitability
Regulation of extracellular levels of ions and neuro - transmitters , especially K+ and glutamatestrongly influencesneuronal excitability
the plateau potential(passive) caused byneuronal excitability
neuroactive steroidsmay influenceneuronal excitability
aspartate and glutamatecausingneuronal excitability
peptide hormonescan influenceneuronal excitability
noradrenergic projections from the midbraincan influenceneuronal excitability
acting on glia and neuronsinfluenceneuronal excitability
persistent form of inhibitioninfluencesneuronal excitability
changes in corticosterone , glutamate , or GABA rather than stress - induced dendritic growth in BLA neurons(passive) caused byneuronal excitability
to pain or seizuresleadsto pain or seizures
to enhanced brain activity before clinical manifestations of the diseaseleadingto enhanced brain activity before clinical manifestations of the disease
to the pain processmay contributeto the pain process
to the generation of seizurescontributesto the generation of seizures
to epileptic seizurescould ... contributeto epileptic seizures
to seizures ... 17leadsto seizures ... 17
to seizures ( 139leadingto seizures ( 139
to neurological disorders accompanied by inflammatory responseleadingto neurological disorders accompanied by inflammatory response
to seizure generation ( Frankenhaeuser & Hodgkin 1956leadingto seizure generation ( Frankenhaeuser & Hodgkin 1956
to increased pain perceptionleadingto increased pain perception
to seizures ( of which migraine is a part ofleadingto seizures ( of which migraine is a part of
to sedative effect of Pramipexoleleadingto sedative effect of Pramipexole
to sedative and sublingual tabletsleadingto sedative and sublingual tablets
in phase opposition with the glial membrane potential ... establishing that only the first 30 % of the neuronal depolarization is efficient for synaptic volleys within cortical neuronal networksresultedin phase opposition with the glial membrane potential ... establishing that only the first 30 % of the neuronal depolarization is efficient for synaptic volleys within cortical neuronal networks
to seizures , Brain , ( 2009 ) in pressleadingto seizures , Brain , ( 2009 ) in press
to sedative effect of Hypnotics ( Nonbenzodiazepineleadingto sedative effect of Hypnotics ( Nonbenzodiazepine
in devastating medical conditions such as chronic painresultingin devastating medical conditions such as chronic pain
to chronic pain and mechanical hypersensitivity.15leadsto chronic pain and mechanical hypersensitivity.15
an exaggerated pain response in the absence of ongoing measurable tissue damagecausingan exaggerated pain response in the absence of ongoing measurable tissue damage
to the repetitive generation of action potentials that can occur during spontaneous seizures in epilepsymay contributeto the repetitive generation of action potentials that can occur during spontaneous seizures in epilepsy
to sedative effect of MetyroSINE . Monitor therapy Magnesium Sulfateleadingto sedative effect of MetyroSINE . Monitor therapy Magnesium Sulfate
to the onset of sporadic spontaneous seizures accompanied by changes in extracellular amino acid concentration in the hippocampusleadingto the onset of sporadic spontaneous seizures accompanied by changes in extracellular amino acid concentration in the hippocampus
in rare small fiber neuropathic pain conditions including primary erythromelalgia ( iEM ) and paroxysmal extreme pain disorder ( PEPDresultingin rare small fiber neuropathic pain conditions including primary erythromelalgia ( iEM ) and paroxysmal extreme pain disorder ( PEPD
to neuroprotectionleadingto neuroprotection
to altered brain functionleadingto altered brain function
the headache and extreme sensitivity to light , sound , smells , and motioncausesthe headache and extreme sensitivity to light , sound , smells , and motion
in enhanced firing probability following an excitatory stimulus , such as kainate - induced depolarizationcould resultin enhanced firing probability following an excitatory stimulus , such as kainate - induced depolarization
in stress and tensionresultingin stress and tension
the brain from becoming hyperactivepreventsthe brain from becoming hyperactive
in an increase in seizure activity.(5resultingin an increase in seizure activity.(5
further mast cell activationcausingfurther mast cell activation
hyperalgesia and allodyniacausinghyperalgesia and allodynia
in a greater state of calmresultingin a greater state of calm
to increased periods of wakefulnessleadsto increased periods of wakefulness
to hypersensitivity in painful processesleadingto hypersensitivity in painful processes
to trigeminal inflammatory hyperalgesiathereby contributingto trigeminal inflammatory hyperalgesia
spontaneous recurrent seizure formation in adult rats with pilocarpine - induced status epilepticuscould preventspontaneous recurrent seizure formation in adult rats with pilocarpine - induced status epilepticus
to the generation and conduction of action potentialsleadingto the generation and conduction of action potentials
from altered biophysical properties in the hNav1.7-A1632 G mutant channelresultingfrom altered biophysical properties in the hNav1.7-A1632 G mutant channel
in a calming effectresultingin a calming effect