Loading ...
Anti nuclear factorcausesneuronal apoptosis ( cell death
Also , anti nuclear factor , NF - kappa - beta is stimulatedcausesneuronal apoptosis ( cell death
deprivation of glutamate stimulation in the immature mammalian braincausesneuronal cell death ( apoptosis
anti nuclear factor ... NF - kappa - betacausesneuronal apoptosis ( cell death
reactive oxygen speciescan causeneuronal cell death ( neurodegeneration).
toxins ( reactive oxygen species ... the rest of the neuroncan causeneuronal cell death ( neurodegeneration
accumulationcausesapoptotic neuronal cell death
A ? accumulationcausesapoptotic neuronal cell death
drugscauseapoptotic neuronal cell death
Acute cerebral ischemiacauseshypoxic neuronal cell death
excitotoxic injurycausesapoptotic neuronal cell death
Glutamate , a major excitatory amino acid neurotransmittercausesapoptotic neuronal cell death
by withdrawal of trophic support(passive) caused byapoptotic neuronal cell death
by overactivation of glutamate receptors(passive) caused byExcitotoxic neuronal cell death
the accumulation of AGE proteins resulting from altered protein homeostasis(passive) can be caused byNeuronal cell death
exerts toxic effects in neuronal cells through multiple mechanisms , such as altering membrane permeability and causing neuronal cell dysfunctioncausingneuronal cell death
Excessive glutamate receptor activity is knownto causeneuronal cell death
Abnormal accumulation of the microtubule - associated protein tau is thoughtto causeneuronal cell death
excessive ROS , mitochondrial dysfunction and caspase activation(passive) may be caused byNeuronal cell death
by excessive activation of the glutamate receptors(passive) caused byneuronal cell death
by excessive activation of glutamate receptors(passive) caused byneuronal cell death
20 ] Epileptic seizures results in free radical production and oxidative damage to cellular proteins , lipids and DNAresultingneuronal cell death
In addition to A peptide - induced ROS mediated neurotoxicity , A peptide has been shown ... to causeneuronal cell death
by over - activation of glutamate receptors(passive) caused byneuronal cell death
The channel activation was lethal to the cellscausingneuronal cell death
Excessive stimulation of NMDA receptors is knownto causeneuronal cell death
abnormal intracellular huntingtin protein aggregatescauseneuronal cell death
by toxic levels of glutamate(passive) caused byneuronal cell death
by toxic levels of glutamate which is associated with Alzheimer 's disease(passive) caused byneuronal cell death
by glutamate - induced toxicity(passive) caused byNeuronal cell death
brain neuronscausingneuronal cell death
by excessive release of glutamate following TBI(passive) is causedneuronal cell death
by agents that disrupt lysosome function and by over - expression of wild - type human(passive) caused byneuronal cell death
by over - stimulation of the glutamate receptors(passive) caused byneuronal cell death
by a toxic level of glutamate(passive) caused byneuronal cell death
the known pathwayscan causeneuronal cell death
the toxic action of glutamatecausedneuronal cell death
leading to the formation of ROSto causeneuronal cell death
the virus and viral proteinscausingneuronal cell death
the virus and viral proteinscausingthe neuronal cell death
in a hypoxic brain injuryresultingin a hypoxic brain injury
to the observed symptomsleadingto the observed symptoms
from primary neuronal lesionsresultingfrom primary neuronal lesions
in a brain injuryresultsin a brain injury
from acute cellular injuryresultsfrom acute cellular injury
neurological changes irreversiblecan causeneurological changes irreversible
in irreversible neurological changesmay resultin irreversible neurological changes
to the development of mitochondria - directed drugs designed to trigger apoptosis in cancer cellshas ledto the development of mitochondria - directed drugs designed to trigger apoptosis in cancer cells
the fat inside these dead and broken cells to release the lipids inside themcausesthe fat inside these dead and broken cells to release the lipids inside them
from acute tissue injuryresultsfrom acute tissue injury
to Alzheimer 's disease ( ADleadsto Alzheimer 's disease ( AD
the reduced number of EGFP+ neural cells(passive) was caused bythe reduced number of EGFP+ neural cells
to mental retardationleadingto mental retardation
to the dysfunction seen in patients with neurodegenerative diseasesleadingto the dysfunction seen in patients with neurodegenerative diseases
to the development of ADleadsto the development of AD
to the development of ADcan leadto the development of AD
to the development ofleadsto the development of
to neurodegenerative diseases such as Parkinson 's disease and Alzheimer 's diseaseleadingto neurodegenerative diseases such as Parkinson 's disease and Alzheimer 's disease
from stroke - induced ischemiaresultsfrom stroke - induced ischemia
to the progressive mental decline in both Down syndrome and Alzheimer diseasecontributesto the progressive mental decline in both Down syndrome and Alzheimer disease
Alzheimer 's disease ( AD(passive) is caused by Alzheimer 's disease ( AD
to Alzheimer 's disease ( ADleadsto Alzheimer 's disease ( AD
to an irreversible decrease in visual functionleadsto an irreversible decrease in visual function
to devastating human pathologies that are associated with severe cognitive and motor deficitscan leadto devastating human pathologies that are associated with severe cognitive and motor deficits
to dementialeadingto dementia
neurodegenerative diseasescould causeneurodegenerative diseases
in neurodegenerationresultingin neurodegeneration
to the resolution of inflammationmay contributeto the resolution of inflammation
to PDleadingto PD
to a variety ofcan leadto a variety of
to Alzheimer 's diseaseleadingto Alzheimer 's disease
from this pathwayresultingfrom this pathway
to several neurological disordersleadsto several neurological disorders
to brain damageleadingto brain damage
to the formation of NETsleadsto the formation of NETs
to neurodegeneration16,17,18,19,20leadingto neurodegeneration16,17,18,19,20
to a variety of symptomscan leadto a variety of symptoms
from Sec24c deficiencyresultingfrom Sec24c deficiency
to reactive microgliosisleadsto reactive microgliosis
to either the induction or the inhibition of an inflammatory responsemay leadto either the induction or the inhibition of an inflammatory response