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Qaagi - Book of Why

Causes

Effects

the increased intake of phosphorus in higher quantitiescausesinhibition of 1?-hydroxylase

GABA Acauseshyperpolarization ( inhibition

calcium and potassium influxcausesneuronal inhibition

the synapsecausingneuronal inhibition

TMScausesneuronal inhibition

a reduction in the probability of action potential initiationcausingneuronal inhibition

A reduction in the probability of action potential generationcausesneuronal inhibition

Activation of GIRK by mGluRsshould causeneuronal inhibition

DNA methylationcausestranscriptional inhibition

GSK3-mediated phosphorylationscausetranscriptional inhibition

retrograde signal from metabotropic glutamate to cannabinoid receptors(passive) caused byPresynaptic inhibition

Activation of GABAB receptorscausespresynaptic inhibition

malayamycin treatment(passive) caused bysporulation inhibition

stroke(passive) caused bytranscallosal inhibition

by rapamycin(passive) caused bymTOR inhibition

MG132 treatment increased levels of high molecular weight polyubiquitinated proteins after 4 and 16 h(passive) caused byproteasome inhibition

the CLAVATA1 receptorultimately causingWUSCHEL inhibition

that has previously been shownto causepresynaptic inhibition

MG132(passive) caused byproteasome inhibition

by retrograde signal from metabotropic glutamate(passive) caused byPresynaptic inhibition

by retrograde signal(passive) caused byPresynaptic inhibition

such effectsare resultingfrom myostatin inhibition

Nutrient insufficiency ... in whichcausesmTOR inhibition

This primary afferent depolarizationcausespresynaptic inhibition

not such resultsare resultingfrom myostatin inhibition

such resultsare resultingfrom myostatin inhibition

endogenous adenosine(passive) caused bypresynaptic inhibition

JNKleadingto myostatin inhibition

by noradrenaline(passive) caused bypresynaptic inhibition

Enkephalincausespresynaptic inhibition

Histoneleadsdeacetylase inhibition

a sudden shock ... orcausingvagal inhibition

a sudden shockcausingvagal inhibition

female onescausingestrus inhibition

a resonance - suppported positive charge or radical(passive) may be influenced byinhibition of cox-2

By selectively exciting A - beta nerve fibres in the skin with TENS , the amount of painful stimulation being transmitted by smaller diameter A - delta and C - fibres can be reducedcausingsegmental inhibition

the mechanism of the P450-catalyzed conversion of NMH ... a nitrosomethane intermediatecausesP450 inhibition

lot of things that physical therapists , physicians , strength and conditioning experts , and coaches docauseneuromuscular inhibition

to increased skeletal muscle mass and strengthledto increased skeletal muscle mass and strength

muscle hyperplasia and hypertrophycausesmuscle hyperplasia and hypertrophy

to muscle hyperplasia and hypertrophyleadsto muscle hyperplasia and hypertrophy

synthetic lethalitycausessynthetic lethality

to increased skeletal muscle mass in mammalsleadsto increased skeletal muscle mass in mammals

to increased lifespanleadsto increased lifespan

in increased brain levels of 5-HTresultingin increased brain levels of 5-HT

the increase in muscle mass(passive) caused bythe increase in muscle mass

in increased muscle mass by preventing degradationwill ... resultin increased muscle mass by preventing degradation

in increased muscle masswill ... resultin increased muscle mass

in more muscle fiber buildingresultsin more muscle fiber building

to muscle enlargementcould leadto muscle enlargement

to physiological and behavioral phenotypesleadingto physiological and behavioral phenotypes

to physiological and behavioral phenotypes leadingto physiological and behavioral phenotypes

in the late 1950sdiscoveredin the late 1950s

to tremendous increases in muscle masscan leadto tremendous increases in muscle mass

ADAMTS13 activity(passive) caused byADAMTS13 activity

opposite effectscausedopposite effects

in downregulation of TopBP1resultsin downregulation of TopBP1

to enhanced stem cell production in vitroleadsto enhanced stem cell production in vitro

to increased bioavailability of the parent compound normally subject to extensive first - pass elimination orcan leadto increased bioavailability of the parent compound normally subject to extensive first - pass elimination or

mitochondrial movement(passive) caused bymitochondrial movement

to autophagyleadsto autophagy

to decreased energy metabolism ... and cell death by autophagy by inhibition of m - TOR pathway via AMPK / ACC and AMPK / raptor / ULK1leadsto decreased energy metabolism ... and cell death by autophagy by inhibition of m - TOR pathway via AMPK / ACC and AMPK / raptor / ULK1

in loss of lipid droplet formation ( Figresultedin loss of lipid droplet formation ( Fig

to enhanced tubulin acetylationleadingto enhanced tubulin acetylation

to an important increase in infection and whether this risk is confined to a definable group of patientsleadsto an important increase in infection and whether this risk is confined to a definable group of patients

thereforecould ... influencetherefore

muscle adaptations that would contribute to impaired endurance capacity , such as a shift toward a less oxidative muscle phenotype and lower capillary density32,33causesmuscle adaptations that would contribute to impaired endurance capacity , such as a shift toward a less oxidative muscle phenotype and lower capillary density32,33

to telomere alterations even in experiments conducted in artificial cellular systems where TERT and TERC are exogenously overexpressedleadsto telomere alterations even in experiments conducted in artificial cellular systems where TERT and TERC are exogenously overexpressed

the integration of virtually all types of inputs that CA3 PCs receivewill influencethe integration of virtually all types of inputs that CA3 PCs receive

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