skeletal muscle hypertrophy PMC4572457 10.1152 / ajpendo.00098.2015 Muscle hypertrophy(passive) caused byskeletal muscle hypertrophy PMC4572457 10.1152 / ajpendo.00098.2015 Muscle hypertrophy

skeletal muscle pathophysiology in Huntington 's disease micepreventsskeletal muscle pathophysiology in Huntington 's disease mice

muscle hyperplasia and hypertrophycausesmuscle hyperplasia and hypertrophy

in increased muscle mass by preventing degradationwill ... resultin increased muscle mass by preventing degradation

cancer - related muscle loss in both lung and skin cancer ( melanomamay preventcancer - related muscle loss in both lung and skin cancer ( melanoma

a chain of events including the reduction of factors ( tgf - beta ) that contribute to fibrosis and cartilage /tendon repaircausesa chain of events including the reduction of factors ( tgf - beta ) that contribute to fibrosis and cartilage /tendon repair

to an increase in skeletal muscle mass and fibre size ( 1leadsto an increase in skeletal muscle mass and fibre size ( 1

to abdominal muscle growthmay leadto abdominal muscle growth

to decreased energy metabolism ... and cell death by autophagy by inhibition of m - TOR pathway via AMPK / ACC and AMPK / raptor / ULK1leadsto decreased energy metabolism ... and cell death by autophagy by inhibition of m - TOR pathway via AMPK / ACC and AMPK / raptor / ULK1

to helping people with muscle weakness but it is n’t easymight leadto helping people with muscle weakness but it is n’t easy

aggresome formation [ 28].8preventedaggresome formation [ 28].8

loss of muscle mass in micepreventsloss of muscle mass in mice

in a dramatic increase in muscle massresultingin a dramatic increase in muscle mass

to tremendous increases in muscle masscan leadto tremendous increases in muscle mass

to an increase in active uPA levels ( Fig . 6A and Bledto an increase in active uPA levels ( Fig . 6A and B

to the E2F1- and myc - mediated transcriptional activation of the microRNAledto the E2F1- and myc - mediated transcriptional activation of the microRNA

to muscle growth in normal and dystrophic miceleadsto muscle growth in normal and dystrophic mice

in a substantial increase in total muscle masscan resultin a substantial increase in total muscle mass

to increased skeletal muscle mass in mammalsleadsto increased skeletal muscle mass in mammals

muscle growth and regeneration and reduction in fat and fibrosiscausesmuscle growth and regeneration and reduction in fat and fibrosis

microgravity - induced loss of skeletal muscle mass and strengthpreventsmicrogravity - induced loss of skeletal muscle mass and strength

to increased fiber size , muscle mass , and absolute forceledto increased fiber size , muscle mass , and absolute force

diet - induced adipocyte hypertrophy ( 4 ) , accumulation of triacylglycerols ( 5 ) and infiltration of macrophages ( 6 , illustrated in a ) in adipose tissue(passive) may be prevented bydiet - induced adipocyte hypertrophy ( 4 ) , accumulation of triacylglycerols ( 5 ) and infiltration of macrophages ( 6 , illustrated in a ) in adipose tissue

TNF - α - induced damage to endothelial cell - cell junctionscould preventTNF - α - induced damage to endothelial cell - cell junctions

TNF - α - induced caspase 3 activation in lung endothelial cellpreventsTNF - α - induced caspase 3 activation in lung endothelial cell

the tumour - promoting effect(passive) caused bythe tumour - promoting effect

but also reverse this processcould ... preventbut also reverse this process

muscle loss that happens as a result of other diseasescan ... preventmuscle loss that happens as a result of other diseases

to an alteration in the MHC content of postnatal skeletal muscleleadingto an alteration in the MHC content of postnatal skeletal muscle

disuse atrophy but not muscle atrophy caused by denervationcan preventdisuse atrophy but not muscle atrophy caused by denervation

to expansion of muscle stem cells ( satellite cellswill leadto expansion of muscle stem cells ( satellite cells

to autophagy through activation of raptor - ULK1-LC3 pathwayleadsto autophagy through activation of raptor - ULK1-LC3 pathway

cell proliferation and EMT ... and exerts an antitumor effect in RCC by activating the NF - κB - NLRP3-caspase-1 pyroptosis signaling pathwaypreventscell proliferation and EMT ... and exerts an antitumor effect in RCC by activating the NF - κB - NLRP3-caspase-1 pyroptosis signaling pathway

smooth muscle cell dedifferentiation in vitro and reduces neointima and restenosis in vivopreventssmooth muscle cell dedifferentiation in vitro and reduces neointima and restenosis in vivo

hyperplasia ( increase in the number of muscle fibres ) , essentially increasing the ceiling for muscle developmentcauseshyperplasia ( increase in the number of muscle fibres ) , essentially increasing the ceiling for muscle development

to less VE - cadherin production in cells and affects the trafficking of VE - cadherin to and conceivably also from the plasma membraneleadsto less VE - cadherin production in cells and affects the trafficking of VE - cadherin to and conceivably also from the plasma membrane

muscle weakness and muscle loss in mice with Huntington ’s Disease , an inherited genetic disorder that causes muscles to degenerate over time [ 17preventedmuscle weakness and muscle loss in mice with Huntington ’s Disease , an inherited genetic disorder that causes muscles to degenerate over time [ 17

muscle weakness and muscle loss in mice with Huntington ’s Disease , an inherited genetic disorder that causes muscles to degenerate over time [ 16preventedmuscle weakness and muscle loss in mice with Huntington ’s Disease , an inherited genetic disorder that causes muscles to degenerate over time [ 16

muscle weakness and muscle loss in mice with Huntington ’s Disease , an inherited genetic disorder that causes muscles to degenerate over time [ Rpreventedmuscle weakness and muscle loss in mice with Huntington ’s Disease , an inherited genetic disorder that causes muscles to degenerate over time [ R

cisplatin - induced mechanical allodynia ... and also completely reversed already existing cisplatin - induced mechanical allodynia , spontaneous pain , and numbnesspreventedcisplatin - induced mechanical allodynia ... and also completely reversed already existing cisplatin - induced mechanical allodynia , spontaneous pain , and numbness