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Qaagi - Book of Why

Causes

Effects

interruption of the blood supply or oxygen(passive) is caused byInfarction of the brain

thrombosis of a cerebral artery Medulloblastoma(passive) caused byAtherothrombotic brain infarction infarction

the ischemia - reperfusion injurycausesmyocardial infarction in the heart

a rapid increase in pressureprovokesmyocardial infarction and stroke of the brain

Scholar Plum , F. , 1983causesinfarction in ischemic brain

2 ) Articlescausesinfarction in ischemic brain

Ischemic Brain Simon provides a clear and well - focused discussion on the questioncausesinfarction in ischemic brain

1983).PubMedCrossRefGoogle Scholar F. Plumcausesinfarction in ischemic brain

the formation of atherosclerotic plaque in the micecan causemyocardial infarction and brain haemorrhaging

lifestyle - related diseases like diabetes , arteriosclerosis , hyperlipidemiacan causemyocardial infarction and brain infarction

in congestive heart failureresultingin congestive heart failure

ischemic - reperfusion damage(passive) caused byischemic - reperfusion damage

posttraumatic stress symptoms(passive) caused byposttraumatic stress symptoms

posttraumatic stress disorder(passive) caused byposttraumatic stress disorder

in permanent damage to the heart muscleresultingin permanent damage to the heart muscle

brain damage in many casescould causebrain damage in many cases

the patient 's heart failure(passive) caused bythe patient 's heart failure

a heart attack Heart disease health centrePreventinga heart attack Heart disease health centre

the ischemia - reperfusion sequence(passive) caused bythe ischemia - reperfusion sequence

from ischemia - reperfusion injuryresultingfrom ischemia - reperfusion injury

disease and/or deathcausesdisease and/or death

viagra Sciencedaily mortality(passive) caused byviagra Sciencedaily mortality

13 percent of deathscauses13 percent of deaths

Almost all of the sudden deaths(passive) were caused byAlmost all of the sudden deaths

a stroke , or the lungs , causing a pulmonary embolismcausinga stroke , or the lungs , causing a pulmonary embolism

damage to the heart muscle ... and the heart is then not strong enough to pump out blood to the bodycausesdamage to the heart muscle ... and the heart is then not strong enough to pump out blood to the body

a high rate of morbidity and mortalitycausesa high rate of morbidity and mortality

about 200,000 deaths a yearcausesabout 200,000 deaths a year

The children 's deaths are thought(passive) to have been caused byThe children 's deaths are thought

in worsening heart failure Oghenerukevwe Odiete , Ewa A Konik , Douglas Brian Sawyerresultingin worsening heart failure Oghenerukevwe Odiete , Ewa A Konik , Douglas Brian Sawyer

SIRS and that inflammation plays an important part in the development and persistence of cardiogenic shock , contributing to myocardial dysfunction and vasodilationmay causeSIRS and that inflammation plays an important part in the development and persistence of cardiogenic shock , contributing to myocardial dysfunction and vasodilation

arrhythmias and sudden cardiac deathcan triggerarrhythmias and sudden cardiac death

5 minutes of clinical death(passive) caused by5 minutes of clinical death

an increase in cardiac sympathetic tone and reduces mortalitypreventsan increase in cardiac sympathetic tone and reduces mortality

to limit the zone of ischemia of the heart muscle and reduces the risk of developing fatal arrhythmiascontributesto limit the zone of ischemia of the heart muscle and reduces the risk of developing fatal arrhythmias

from thrombotic occlusion of an epicardial vessel or stroke resulting from carotid artery plaque formationresultingfrom thrombotic occlusion of an epicardial vessel or stroke resulting from carotid artery plaque formation

stroke ... and can delay senility and retard the aging process , thus confirming the powerful antioxidant quality of jiaogulancausesstroke ... and can delay senility and retard the aging process , thus confirming the powerful antioxidant quality of jiaogulan

to improved myocardial performance , improved ejection fraction , and decreased mortality compared to patients treated with placeboleadsto improved myocardial performance , improved ejection fraction , and decreased mortality compared to patients treated with placebo

focal heart muscle necrosis , the death of heart muscle cells , and a loss of con- tractilitycausesfocal heart muscle necrosis , the death of heart muscle cells , and a loss of con- tractility

to an enhanced release of endocannabinoids and a massive accumulation of neutrophils and monocytes within the ischemic myocardiumleadsto an enhanced release of endocannabinoids and a massive accumulation of neutrophils and monocytes within the ischemic myocardium

scar tissue(passive) caused byscar tissue

to the loss of a huge number of cardiomyocytes and the reparatory response to this phenomenonleadsto the loss of a huge number of cardiomyocytes and the reparatory response to this phenomenon

a profound influx of neutrophils and monocytes on days 1 to 6triggersa profound influx of neutrophils and monocytes on days 1 to 6

to decreased inflammatory monocytes in myocardiumledto decreased inflammatory monocytes in myocardium

inflammation and leukocyte recruitment at remote sites in the myocardium and in the renal glomeruluscausesinflammation and leukocyte recruitment at remote sites in the myocardium and in the renal glomerulus

from oxygen deprivation due to poor circulationresultedfrom oxygen deprivation due to poor circulation

the staff pulmonary edema(passive) caused bythe staff pulmonary edema

to loss of cells in the heart and that enhanced proliferation of cells in p27-deficient miceleadsto loss of cells in the heart and that enhanced proliferation of cells in p27-deficient mice

in robust engraftment of transplanted cells , which exhibited colocalization with markers of cardiomyocyte ( CMC ) , EC , and smooth muscle cell ( SMC ) identity , consistent with differentiation of hBMSCs into multiple lineages in vivoresultedin robust engraftment of transplanted cells , which exhibited colocalization with markers of cardiomyocyte ( CMC ) , EC , and smooth muscle cell ( SMC ) identity , consistent with differentiation of hBMSCs into multiple lineages in vivo

crushing or squeezing pain and/or tightness in the chestcausescrushing or squeezing pain and/or tightness in the chest

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