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In a large number or cancer patients hyperactive or reactivated telomerase was notedto result partlyfrom mutations in the telomerase promoter
the mutagenesis procedurecould have createdpromoter mutations
two - round PCR(passive) were created byPromoter mutations
a two - stage PCR protocol using plasmid L4P 26018 - 26098Luc as the template(passive) were created byMutations in the core promoter
purple cauliflowerresultingfrom mutation in the BoMYB2 promoter
in solid and hematological malignancies , where they reflect TERT activation and cell - cycle progression(passive) have been discoveredpromoter mutations
A Quikchange site - directed mutagenesis kit ( Stratagene ) was usedto createmutations in the Ω4400 promoter region
the yeast retrotransposon(passive) caused bypromoter insertion mutations
as follows(passive) were createdThe ERG5 promoter mutations
β - lactamresultingfrom promoter mutations
binding of RNA polymerase to act in trans on another copy of the operon on a plasmid in the cell or only in cis on the copy immediately adjacent to the mutated sitepreventbinding of RNA polymerase to act in trans on another copy of the operon on a plasmid in the cell or only in cis on the copy immediately adjacent to the mutated site
binding of RNA polymerase to act in trans on another copy of the operon on a plasmid in the cell or only in cis on the copy immediately adjacent to the home forex ru sitepreventbinding of RNA polymerase to act in trans on another copy of the operon on a plasmid in the cell or only in cis on the copy immediately adjacent to the home forex ru site
Pelizaeus - Merzbacher - like disease Mol Genet MetabcausingPelizaeus - Merzbacher - like disease Mol Genet Metab
to increased expression of the phu systemleadingto increased expression of the phu system
to reduce or eliminate negative regulationdesignedto reduce or eliminate negative regulation
Pelizaeus - Merzbacher - like disease Psychogenic non - epileptic seizures in IrancausingPelizaeus - Merzbacher - like disease Psychogenic non - epileptic seizures in Iran
in ahpC overexpressionresultingin ahpC overexpression
in new phenotypes or diseasescan resultin new phenotypes or diseases
binding of RNA polymerase to act in trans on another copy of the operon on a plasmid in the cell binary option robot review only in cis on the copypreventbinding of RNA polymerase to act in trans on another copy of the operon on a plasmid in the cell binary option robot review only in cis on the copy
expression of the AhpC protein(passive) was caused byexpression of the AhpC protein
changes in the −35 region produce a stronger promoter that is capable of increased transcription and ... as a result ... increased expression of ClfB.causechanges in the −35 region produce a stronger promoter that is capable of increased transcription and ... as a result ... increased expression of ClfB.
to partial or complete inhibition of AOX1a gene expression [ 95ledto partial or complete inhibition of AOX1a gene expression [ 95
in over - expression of ahpC , a putative virulence gene involved in cell protection from oxidative stress 4resultingin over - expression of ahpC , a putative virulence gene involved in cell protection from oxidative stress 4
to enhanced gene expression like TERT gene , and virus genome integration that leads to disturbance of endogenous regulatory genes ( 15leadingto enhanced gene expression like TERT gene , and virus genome integration that leads to disturbance of endogenous regulatory genes ( 15
to decreased expressionleadingto decreased expression
operon expressionwould preventoperon expression
the pathogen - induced increase in OsSWEET11 expressiontherefore preventsthe pathogen - induced increase in OsSWEET11 expression
binding of RNA polymerase to act in trans on another copy of the operon binary options trading freepreventbinding of RNA polymerase to act in trans on another copy of the operon binary options trading free
the promoter to become nonfunctionalwill causethe promoter to become nonfunctional
novel ETS binding sites , which contributes increased telomerase activity in cancer cellscreatednovel ETS binding sites , which contributes increased telomerase activity in cancer cells
this locus codes for a negative repressor of cell division , and(passive) are caused bythis locus codes for a negative repressor of cell division , and
the antisense RNA synthesispreventsthe antisense RNA synthesis
de novo transcription factor binding sitescreatingde novo transcription factor binding sites
CEPcould causeCEP
in higher level nearly 2 fold of inducible expression for a marker gene ... howeverresultedin higher level nearly 2 fold of inducible expression for a marker gene ... however
C228 T and C250 TcausedC228 T and C250 T
upregulation of csgD expression or the mutations in genes , such as rpoS , mlrA and rcsBcausingupregulation of csgD expression or the mutations in genes , such as rpoS , mlrA and rcsB
changes of transcription factor binding sitescan causechanges of transcription factor binding sites
misregulation and disease / phenotypecan causemisregulation and disease / phenotype
to a reduction of mgrB expression or to mutations in other loci leading to upregulation of the Pmr system responsible for modification of the lipopolysaccharide polymyxin targetleadingto a reduction of mgrB expression or to mutations in other loci leading to upregulation of the Pmr system responsible for modification of the lipopolysaccharide polymyxin target
the rate of transcriptionmay influencethe rate of transcription
to the development of T2D and related metabolic traits [ 45might contributeto the development of T2D and related metabolic traits [ 45
in inefficient transcription initiationcan resultin inefficient transcription initiation
in altered cell morphology and reduced sporulation efficiencyresultin altered cell morphology and reduced sporulation efficiency
significant changes in the pattern ... binary / graded ) of the gene response to a signalmay causesignificant changes in the pattern ... binary / graded ) of the gene response to a signal
We have found(passive) is caused byWe have found
to increased transcription levelsleadingto increased transcription levels
in binding of the GABP transcription factorresultin binding of the GABP transcription factor
in aberrant generesultingin aberrant gene
binding sites for ETS transcription factorscreatebinding sites for ETS transcription factors