Restoration of blood supply to ischemic tissues ... free radicals and reactive oxygen speciescausesreperfusion injury
skin tissuescausingreperfusion injury
There has been concern about the potential of the formerto causeneurologic injury
some with enough G - forceto causeneurologic injury
to be lowto causeneurologic injury
factorsleadto mucosal injury
the pointcould causeneurologic injury
the timecould causereperfusion injury
also(passive) could be ... influencedReperfusion injury
also(passive) is ... causedReperfusion injury
a burst fracturecausingneurologic injury
a potential factorcontributingto mucosal injury
copper deficiencycould causeneurologic injury
enough G forcesto causeneurologic injury
by exposure of the esophagus(passive) caused bymucosal injury
productsleadingto mucosal injury
Gastric inflammation ... several processesleadsto mucosal injury
by asphyxia without motor dysfunction(passive) caused byneurologic injury
Surgeoncausesneurologic injury
Cerebral edemacan causeneurologic injury
by the formation of free radicals(passive) caused byreperfusion injury
reactive oxygen species ( ROS ) , which are knownto causereperfusion injury
cellsleadingto mucosal injury
neededto causeneurologic injury
by oxygen radicals(passive) was ... caused byreperfusion injury
oxygen radicals(passive) was ... caused byreperfusion injury
its activationleadsto mucosal injury
The mechanismsleadto mucosal injury
the mechanisms by whichleadto mucosal injury
by free radical release(passive) caused byReperfusion injury
the most important mechanismcausingmucosal injury
in case the seizure attack continuous for more than 30 minuteswill causeneurologic injury
bacterial by - productsleadingto mucosal injury
a serious problem from significant soft tissue necrosiscan causea serious problem from significant soft tissue necrosis
loss of sensation and movement from lower half of the body ( including legscausesloss of sensation and movement from lower half of the body ( including legs
loss of sensation and movementcausesloss of sensation and movement
to up to 50 % of the total myocardial damagecontributesto up to 50 % of the total myocardial damage
to damage of the lungcan also leadto damage of the lung
to endothelial injuryleadsto endothelial injury
damage(passive) caused bydamage
the damage(passive) caused bythe damage
from the rapid restoration of full blood flow and pressure to ischemic tissuecan resultfrom the rapid restoration of full blood flow and pressure to ischemic tissue
to necrosis and apoptosisleadsto necrosis and apoptosis
significant swelling and compartment syndromecan ... causesignificant swelling and compartment syndrome
myocardial cell deathmay causemyocardial cell death
alsomay ... leadalso
alsomay ... resultalso
alsomay ... influencealso
alsocan ... resultalso
alsocan causealso
Multiple organ dysfunction syndrome(passive) caused byMultiple organ dysfunction syndrome
from ischemiaresultingfrom ischemia
significant mortality after lung transplantationcausesignificant mortality after lung transplantation
significant mortality after lung transplantationDoes ... still causesignificant mortality after lung transplantation
to decreasedleadingto decreased
necrosis 4causesnecrosis 4
to an increased Infarct Size Impaired microvascular perfusion with subsequent large final infarct sizecontributesto an increased Infarct Size Impaired microvascular perfusion with subsequent large final infarct size
scarringcan causescarring
from ischemia , which may lead to endothelial cell dysfunctionresultingfrom ischemia , which may lead to endothelial cell dysfunction
in multi - organ failure and deathcan resultin multi - organ failure and death
release of oxygen free radicals , toxic byproducts of ischemic injury and neutrophil activationcausesrelease of oxygen free radicals , toxic byproducts of ischemic injury and neutrophil activation
to severe compartment syndromecan leadto severe compartment syndrome
an infarct size of 50%,causedan infarct size of 50%,
the fibrosismay causethe fibrosis
stillDoes ... causestill
to damage to the hemodynamic and functional parameters of the kidneyleadsto damage to the hemodynamic and functional parameters of the kidney
to stunning , necrosisleadingto stunning , necrosis
a significant amountmay contributea significant amount
to release of inflammatory mediators and reactive oxygen speciesleadingto release of inflammatory mediators and reactive oxygen species
from ischemia , which may lead to endothelial cellresultingfrom ischemia , which may lead to endothelial cell
mitochondria damage by the same NO - superoxide mechanismcan ... causemitochondria damage by the same NO - superoxide mechanism