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Smart Reasoning:

C&E

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Qaagi - Book of Why

Causes

Effects

The specific mechanismscausechanges in Per gene expression and rhythm in cancer

The specific mechanisms ... ; iiicausechanges in Per gene expression and rhythm in cancer

DNA methylationmay leadto changes in gene expression and loss of gene function

other cellsleadingto changes in cellular biochemistry and potentially gene expression

role in histone modificationleadsto epigenetic change in gene expression and tumorigenesis

signals from these receptorsleadingto changes in cell morphology and gene expression

these receptorsleadingto changes in cell morphology and gene expression

because it triggers the interferon responsecausingwidespread changes in gene expression and apoptosis

genetic and environmental factorsmight resultin epigenetic alterations of gene expression and other molecular path

a chemical cascade in the cellresultingin changes in gene expression and in cell response

changes in leaf topography into ion fluxesleadto changes in gene expression and appresorium formation.[4].

an Iyengar yoga intervention designed for fatigued breast cancer survivorswould leadto decreases in inflammation - related gene expression and

the stretching of the cell membrane caused by changes in leaf topography into ion fluxesleadto changes in gene expression and appresorium formation.[4].

rather are believedto leadto genetic variability in gene expression and splicing

These interactions or disturbancescan resultin heritable and non - heritable changes in gene expression and function

decreased glucose and insulin levelscausingdecreased leptin gene expression and synthesis

disrupted in many human cancers downstream of common oncogenic alterationsleadingto altered oscillation of gene expression and metabolism

complex signaling pathwaysleadto changes in gene expression and cellular function

the signal transduction pathwaysleadingto gene expression and biochemical changes

Secondly we define a previously unknown role for the transcription factor GRHL3 in the recruitment of a trxG complex to promoters of genesleadingto increased H3K4 methylation and gene expression

epigenetic events that can initiate the expansion of altered cells during the early stages of tumorigenesis(passive) caused bychanges in gene expression and transcriptional inactivation

insulinmay causevariation in gene expression of heparanase , syndecan and perlecan

natural variationcontributingto changes in gene expression and phenotype

aberrations in said mechanisms(passive) caused bychanges in gene expression and protein kinetics

their activitiescan contributeto gene expression and phenotypic variability

Epigenetic mechanismsleadto changes in gene expression and function

here showcausesdramatic changes in physiology and gene expression in Cand

subsequent signal transductionleadsto alterations in gene expression and morphology

several signaling pathwaysleadto changes in gene expression and metabolism

the genomeresultedin changes in gene expression and metabolism

Kindling effectsresultfrom epigenetics , gene - expression and neuroplasticity

aberrant proliferation as well as a significant increase in GATA-2 expression , consistent with the hypothesis ... overexpression of GATA-2contributesto altered gene expression and hyperproliferation of megakaryoblasts

GA biosynthesis , perception , and subsequent signal transductionleadsto alterations in gene expression and morphology

experimentally induced oxidative stress and ROS related diseases(passive) caused byalterations of gene expression and protein levels

genetic instabilityresultingin dramatic changes in gene expression and energy metabolism

This study explored in vitro and in vivoinfluencesgene expression and molecular mechanisms in keratinocytes

Vitamin D ... nuclear transcription factorsleadto changes in gene expression and cell function

enhancer modificationsresultingin changes in gene expression and morphology

disease progressionto influencedisease progression

cell growth and apoptotic deathinfluencecell growth and apoptotic death

to the activation of multiple signalling pathwaysleadto the activation of multiple signalling pathways

from the disease mutationresultfrom the disease mutation

This inhibition(passive) was caused byThis inhibition

to tumour growthcan contributeto tumour growth

to variations of thermo - sensitivity in natural Arabidopsis accessionscontributeto variations of thermo - sensitivity in natural Arabidopsis accessions

in cellular damageresultin cellular damage

to cancerleadto cancer

eventuallyresulteventually

to catabolism of fuel storesleadto catabolism of fuel stores

brain and nerve regenerationmay causebrain and nerve regeneration

The syndrome(passive) is caused byThe syndrome

to the diversity of visual systems in African cichlid fishledto the diversity of visual systems in African cichlid fish

to the diversity of visual systems in African cichlid fishledto the diversity of visual systems in African cichlid fish

to apoptosis and with stimulating lipolysis and inhibiting lipogenesiscan leadto apoptosis and with stimulating lipolysis and inhibiting lipogenesis

to the tragic eventleadto the tragic event

to HIV-1-associated dementiacontributeto HIV-1-associated dementia

to HIV-1-associated dementiacontributeto HIV-1-associated dementia

in disease susceptibility or resistanceresultingin disease susceptibility or resistance

to the penetrance of SIODcontributeto the penetrance of SIOD

at least partly to the aetiology of various pathologies , especially neurological disorders ( see Table 1may contributeat least partly to the aetiology of various pathologies , especially neurological disorders ( see Table 1

in cellular damageresultin cellular damage

to the onsetcontributeto the onset

malignancycausemalignancy

skeletal muscle wasting and weakness in ICU patients with CIMinfluencingskeletal muscle wasting and weakness in ICU patients with CIM

gain or loss of gene function(passive) caused bygain or loss of gene function

the fates of cells infected with human immunodeficiency virus ( HIVcan influencethe fates of cells infected with human immunodeficiency virus ( HIV

to the onset and the progression of breast cancercontributeto the onset and the progression of breast cancer

the fates of cells infected with human immunodeficiency virus ( HIV ) far more than previously thoughtcan influencethe fates of cells infected with human immunodeficiency virus ( HIV ) far more than previously thought

to the onsetcontributeto the onset

to T2D and obesitymay contributeto T2D and obesity

to adaptability reconstruction of structures and contractile characters of the muscular cells , which are closely related with activation of Na+ , K+-ATPase , and influences the distribution and functional activity of Na+ , K+- ATPase on the surface of muscular cell membraneleadingto adaptability reconstruction of structures and contractile characters of the muscular cells , which are closely related with activation of Na+ , K+-ATPase , and influences the distribution and functional activity of Na+ , K+- ATPase on the surface of muscular cell membrane

our experienceinfluencesour experience

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Smart Reasoning:

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