The specific mechanismscausechanges in Per gene expression and rhythm in cancer
The specific mechanisms ... ; iiicausechanges in Per gene expression and rhythm in cancer
DNA methylationmay leadto changes in gene expression and loss of gene function
other cellsleadingto changes in cellular biochemistry and potentially gene expression
role in histone modificationleadsto epigenetic change in gene expression and tumorigenesis
signals from these receptorsleadingto changes in cell morphology and gene expression
these receptorsleadingto changes in cell morphology and gene expression
because it triggers the interferon responsecausingwidespread changes in gene expression and apoptosis
genetic and environmental factorsmight resultin epigenetic alterations of gene expression and other molecular path
a chemical cascade in the cellresultingin changes in gene expression and in cell response
changes in leaf topography into ion fluxesleadto changes in gene expression and appresorium formation.[4].
an Iyengar yoga intervention designed for fatigued breast cancer survivorswould leadto decreases in inflammation - related gene expression and
the stretching of the cell membrane caused by changes in leaf topography into ion fluxesleadto changes in gene expression and appresorium formation.[4].
rather are believedto leadto genetic variability in gene expression and splicing
These interactions or disturbancescan resultin heritable and non - heritable changes in gene expression and function
decreased glucose and insulin levelscausingdecreased leptin gene expression and synthesis
disrupted in many human cancers downstream of common oncogenic alterationsleadingto altered oscillation of gene expression and metabolism
complex signaling pathwaysleadto changes in gene expression and cellular function
the signal transduction pathwaysleadingto gene expression and biochemical changes
Secondly we define a previously unknown role for the transcription factor GRHL3 in the recruitment of a trxG complex to promoters of genesleadingto increased H3K4 methylation and gene expression
epigenetic events that can initiate the expansion of altered cells during the early stages of tumorigenesis(passive) caused bychanges in gene expression and transcriptional inactivation
insulinmay causevariation in gene expression of heparanase , syndecan and perlecan
natural variationcontributingto changes in gene expression and phenotype
aberrations in said mechanisms(passive) caused bychanges in gene expression and protein kinetics
their activitiescan contributeto gene expression and phenotypic variability
Epigenetic mechanismsleadto changes in gene expression and function
here showcausesdramatic changes in physiology and gene expression in Cand
subsequent signal transductionleadsto alterations in gene expression and morphology
several signaling pathwaysleadto changes in gene expression and metabolism
the genomeresultedin changes in gene expression and metabolism
Kindling effectsresultfrom epigenetics , gene - expression and neuroplasticity
aberrant proliferation as well as a significant increase in GATA-2 expression , consistent with the hypothesis ... overexpression of GATA-2contributesto altered gene expression and hyperproliferation of megakaryoblasts
GA biosynthesis , perception , and subsequent signal transductionleadsto alterations in gene expression and morphology
experimentally induced oxidative stress and ROS related diseases(passive) caused byalterations of gene expression and protein levels
genetic instabilityresultingin dramatic changes in gene expression and energy metabolism
This study explored in vitro and in vivoinfluencesgene expression and molecular mechanisms in keratinocytes
Vitamin D ... nuclear transcription factorsleadto changes in gene expression and cell function
enhancer modificationsresultingin changes in gene expression and morphology
cell growth and apoptotic deathinfluencecell growth and apoptotic death
to the activation of multiple signalling pathwaysleadto the activation of multiple signalling pathways
from the disease mutationresultfrom the disease mutation
This inhibition(passive) was caused byThis inhibition
to tumour growthcan contributeto tumour growth
to variations of thermo - sensitivity in natural Arabidopsis accessionscontributeto variations of thermo - sensitivity in natural Arabidopsis accessions
in cellular damageresultin cellular damage
to cancerleadto cancer
eventuallyresulteventually
to catabolism of fuel storesleadto catabolism of fuel stores
brain and nerve regenerationmay causebrain and nerve regeneration
The syndrome(passive) is caused byThe syndrome
to the diversity of visual systems in African cichlid fishledto the diversity of visual systems in African cichlid fish
to the diversity of visual systems in African cichlid fishledto the diversity of visual systems in African cichlid fish
to apoptosis and with stimulating lipolysis and inhibiting lipogenesiscan leadto apoptosis and with stimulating lipolysis and inhibiting lipogenesis
to the tragic eventleadto the tragic event
to HIV-1-associated dementiacontributeto HIV-1-associated dementia
to HIV-1-associated dementiacontributeto HIV-1-associated dementia
in disease susceptibility or resistanceresultingin disease susceptibility or resistance
to the penetrance of SIODcontributeto the penetrance of SIOD
at least partly to the aetiology of various pathologies , especially neurological disorders ( see Table 1may contributeat least partly to the aetiology of various pathologies , especially neurological disorders ( see Table 1
in cellular damageresultin cellular damage
to the onsetcontributeto the onset
malignancycausemalignancy
skeletal muscle wasting and weakness in ICU patients with CIMinfluencingskeletal muscle wasting and weakness in ICU patients with CIM
gain or loss of gene function(passive) caused bygain or loss of gene function
the fates of cells infected with human immunodeficiency virus ( HIVcan influencethe fates of cells infected with human immunodeficiency virus ( HIV
to the onset and the progression of breast cancercontributeto the onset and the progression of breast cancer
the fates of cells infected with human immunodeficiency virus ( HIV ) far more than previously thoughtcan influencethe fates of cells infected with human immunodeficiency virus ( HIV ) far more than previously thought
to the onsetcontributeto the onset
to T2D and obesitymay contributeto T2D and obesity
to adaptability reconstruction of structures and contractile characters of the muscular cells , which are closely related with activation of Na+ , K+-ATPase , and influences the distribution and functional activity of Na+ , K+- ATPase on the surface of muscular cell membraneleadingto adaptability reconstruction of structures and contractile characters of the muscular cells , which are closely related with activation of Na+ , K+-ATPase , and influences the distribution and functional activity of Na+ , K+- ATPase on the surface of muscular cell membrane