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Laboratory reportactivity 9 : regulation of pulmonary ventilation1 hypercapnia ( high arterial carbon dioxide partial pressurecausesminute ventilation to2
Activity 9 : regulation of pulmonary ventilation name : instructor : date : predictions 1 hypercapnia ( high arterial carbon dioxide partial pressurecausesminute ventilation
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initially(passive) was ... setMinute ventilation ( V̇e
to 6 L / min(passive) was setThe minute ventilation
at 4 L / min(passive) was setMinute ventilation
at approximately 9 l / min(passive) was setMinute ventilation
to 6.2 L / min(passive) was setThe minute ventilation
at a rate of 110 l / min(passive) was setMinute ventilation
the reduction in mandatory breath frequency from 14 to 5 breaths / min(passive) caused byminute ventilation
apnea.• Prevents hypercapnea(passive) caused byminute ventilation
Tidal volume and respiratory ratesetthe minute ventilation
the cool dry air(passive) caused byminute ventilation
A lower operatorset% -minute ventilation with ASV
at 100 mL(passive) was initially setMinute ventilation
forth ... way of example only and that any number of different physiologic parameters indicative of metabolic demand may be employed , including but not limited to QT interval , respiration rate , venous oxygen saturation , stroke volume , PEI , dv / dt and venous blood temperature(passive) is set ... byminute ventilation
in that case(passive) may ... be setThe minute ventilation
the clinician(passive) set bythe minute ventilation
an increase in tidal volume increases from 500ml to 700ml(passive) caused byminute ventilation
to maintain an end - tidal carbon dioxide pressure of 3.5–4.5 kPa(passive) was setMinute ventilation
hypoxia(passive) triggered byminute ventilation
trigger failures and hyperinflation(passive) caused byminute ventilation
removal of the hypoxic stimulus [ 56 , 57(passive) caused byminute ventilation
compliance changes but guaranteessetminute ventilation
at 100 % of the theoretical value based on predicted body weight(passive) was setMinute ventilation
ATVs deliversetminute ventilation
according to the end - tidal carbon dioxide ( ETCO2 ) value(passive) was setMinute ventilation
S - nitroso - L - cysteine administration to the nucleus(passive) caused byminute ventilation
the following graduation(passive) is composed byMinute Ventilation
PaCO(2(passive) is influenced byMinute ventilation
from modifications in ventilator settingsresultingfrom modifications in ventilator settings
on the ventilator to achieve my goalseton the ventilator to achieve my goal
the blood CO2 levels and pH balanceinfluencesthe blood CO2 levels and pH balance
to faster and deeper breathingleadingto faster and deeper breathing
in the volume preset ventilator control panel and minute ventilation prescribed for the patient in 157 patients on volume preset ventilation and ( B ) inspiratory pressure in pressure preset ventilators in 137 patientssetin the volume preset ventilator control panel and minute ventilation prescribed for the patient in 157 patients on volume preset ventilation and ( B ) inspiratory pressure in pressure preset ventilators in 137 patients
Vmn , the minimum tidal volume VT min , and the maximum airway pressure PAWmax to satisfy the equationsettingVmn , the minimum tidal volume VT min , and the maximum airway pressure PAWmax to satisfy the equation
at 8 , 10 or 12 L / minsetat 8 , 10 or 12 L / min
in hypoventilationresultin hypoventilation
to compensate for the respiratory acidosissetto compensate for the respiratory acidosis
in a reduction in the actual durationresultingin a reduction in the actual duration
a marked reductioncausesa marked reduction
to several symptoms characteristic of hyperventilationleadsto several symptoms characteristic of hyperventilation
above 100 %settingabove 100 %
to decreased Pa CO2 and alkalosisleadsto decreased Pa CO2 and alkalosis
The inhaled dose of air pollutants of different groups of commuters(passive) is influenced byThe inhaled dose of air pollutants of different groups of commuters
in decreased PETCO2 valuesresultsin decreased PETCO2 values
severe acidosisto preventsevere acidosis
Intake of air pollutants(passive) is influenced byIntake of air pollutants
up sensibly for me about four weeks post implantsetup sensibly for me about four weeks post implant
in a reduction of ETCO 2 to 16resultingin a reduction of ETCO 2 to 16
in pulmoruuy vasoconstriction and a and bmay resultin pulmoruuy vasoconstriction and a and b
in increased arterial carbon dioxide ( CO2 ) that leads to hypoxemiaresultingin increased arterial carbon dioxide ( CO2 ) that leads to hypoxemia
from an extension ofThighresultingfrom an extension ofThigh
a target tidal volume and a rateto seta target tidal volume and a rate
in an increase in alveolar and arterial PO2resultingin an increase in alveolar and arterial PO2
from an increased tidal volumeprimarily resultingfrom an increased tidal volume
in an increase in PaCO2resultsin an increase in PaCO2
initially for most drugs other than factors VIII and XIII , fibrinogen , and fibronectin at concentrations over the counter prednisone cream as low as 6–7 mg/ dL. Hyperventilation often provokes attacksis setinitially for most drugs other than factors VIII and XIII , fibrinogen , and fibronectin at concentrations over the counter prednisone cream as low as 6–7 mg/ dL. Hyperventilation often provokes attacks
lung injury Patients who failed had a higher mortality than predicted by APACHE scorecausinglung injury Patients who failed had a higher mortality than predicted by APACHE score
change in PETCO2 and PaCO2to preventchange in PETCO2 and PaCO2
to increased PaCO2 [ 63can contributeto increased PaCO2 [ 63
to hypocapnialeadsto hypocapnia
in a more controlled increase of the rate as the rate rises above the ADL boundaryresultsin a more controlled increase of the rate as the rate rises above the ADL boundary