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Research Areas Translation regulatory factor RBM3 ... a proto - oncogenepreventsmitotic catastrophe
cumulative DNA damage(passive) caused bymitotic catastrophe
Sureban SM , Ramalingam S , Natarajan G , May R , Subramaniam D , Bishnupuri KS , Morrison AR , Dieckgraefe BK , Brackett DJ , Postier RG , Houchen CW , Anant S. Translation regulatory factor RBM3 ... a proto - oncogenepreventsmitotic catastrophe
1125016413.Replication checkpointpreventingmitotic catastrophe
the collision of an asteroid with the Earth． men(passive) has yet been caused byC)No catastrophe
the DNA damage - induced , Chk1-dependent checkpoint ( 30(passive) can also be triggered byMitotic catastrophe
that the CUL4A / B deficiency allows for entering mitosis despite irreparable DNA damagethereby causingmitotic catastrophe
Cdk1 is sequestered in the cytoplasm upon DNA damage in a 14 - 3 - 3-dependent mannerto preventmitotic catastrophe
Oct 2.PMID : 11593014 Free PMC ArticleSimilar articles Select item 112501642.Replication checkpointpreventingmitotic catastrophe
in G1 and subsequent G1 arrest associated with senescence or ( ii ) premature mitotic entry in the absence of p53 / p21WAF1causingmitotic catastrophe
Combining PARP inhibitor Olaparib with the BET inhibitor , we observed a synergistic increase in DNA damage and checkpoint defects , which allowed cells to enter mitosis despite the accumulation of DNA damageultimately causingmitotic catastrophe
agents that damage microtubules and disrupt the mitotic spindle(passive) is also caused byMitotic catastrophe
the space occupied by Kinesin‐7to triggermicrotubule catastrophe
drugs that target microtubules or affect the progression of mitosis(passive) could be caused byMitotic catastrophe
agents that affect the stability of microtubules , leading to defects in the assembly of the mitotic spindle ( 160(passive) can be triggered byMitotic catastrophe
agents that affect the stability of microtubules as well as by mitotic failure that results from impaired cell cycle checkpoints(passive) is also triggered byMitotic catastrophe
premature exit from mitosis(passive) is ... caused bymitotic catastrophe
knockdown of Bub1b promotes endoreduplicationcausingmitotic catastrophe
rapid progression(passive) can be caused byMitotic catastrophe
in addition to inducing apoptosismay causemitotic catastrophe
the effects of these motors on stabilized microtubules as models of the GTP cap , which was thought to be at the end of growing microtubules and whose loss was thoughtto causemicrotubule catastrophe
Radiationcausesmitotic catastrophe
mitotic errors leading to widespread chromosomal rearrangements [ 14(passive) caused byMitotic catastrophe
PC1 knockdowncausesmitotic catastrophe
microtubule instability leading to an abnormal mitotic checkpoint ( 19(passive) can also be triggered byMitotic catastrophe
lower dosescausedmitotic catastrophe
aberrant mitosis and executed either during mitosis or in the subsequent interphase.5 , 8(passive) are triggered bymitotic catastrophe
The dashed arrows point to cells that underwent endoreduplicationeventually causingmitotic catastrophe
key cell - cycle and mitosis genescausingmitotic catastrophe
Pololike kinase 1 inhibitor BI2536causesmitotic catastrophe
irreparable DNA lesionsto causemitotic catastrophe
chewablw locating the tracheostomy at the level of the second and Ssle tracheal rings(passive) is prevented byпThis catastrophe
Constitutive SOCEcausesmitotic catastrophe
Polo - like kinase 1 inhibitor BI2536causesmitotic catastrophe
the presence of neither p21WAF1 nor 14 - 3 - 3σ ... sufficientto preventmitotic catastrophe
to “ facilitate ” the reentry of cells into mitosisthus causingmitotic catastrophe
Kalanchoe tubiflora and opens avenues for further research by presenting comparable results obtained so far(passive) is triggered bymitotic catastrophe
premature mitotic entrance with either unrepaired DNA harm or(passive) caused bymitotic catastrophe
BKM120-dependent activation of the CDK1 / Cyclin B1 complex and concomitant upregulation of Cyclin B1 accompanied by a strong mitotic arrest(passive) is triggered byMitotic catastrophe
the dam rupturing of the mining company Vale in Brumadinho ( MG(passive) caused bySocioenvironmental catastrophe
to different types of cell deathcan leadto different types of cell death
to cell death in intestinal stem cellscontributesto cell death in intestinal stem cells
a P73-dependent apoptotic cell death in chronic myelogenous leukemia cells ( K562(passive) was triggered bya P73-dependent apoptotic cell death in chronic myelogenous leukemia cells ( K562
to cellular necrosisleadingto cellular necrosis
in fatal dilated cardiomyopathy , " Circulation Researchresultingin fatal dilated cardiomyopathy , " Circulation Research
either to apoptotic morphology or to necrosiscan leadeither to apoptotic morphology or to necrosis
in primary necrosis in mouse hepatocytesresultingin primary necrosis in mouse hepatocytes
in fatal dilated cardiomyopathy — Penn State Rationaleresultingin fatal dilated cardiomyopathy — Penn State Rationale
to apoptosis or necrosis in p53-deficient cellsleadingto apoptosis or necrosis in p53-deficient cells
in the activation of three distinct oncosuppressive mechanisms , i.e. , apoptosis , necrosis and senescence , and cancer cellscan resultin the activation of three distinct oncosuppressive mechanisms , i.e. , apoptosis , necrosis and senescence , and cancer cells
in fatal dilated cardiomyopathy AU - Ahmad , Firdos AU - Parikh , Shan AU - Hoffman , Nichole E. AU - Verma , Vipin K. AU - Yuan , Ancai AU - Guo , Yuanjun AU - Lal , Hind N2 - Rationaleresultingin fatal dilated cardiomyopathy AU - Ahmad , Firdos AU - Parikh , Shan AU - Hoffman , Nichole E. AU - Verma , Vipin K. AU - Yuan , Ancai AU - Guo , Yuanjun AU - Lal , Hind N2 - Rationale
in fatal dilated cardiomyopathy Zhou , J. , Ahmad , F. , Parikh , S. , Hoffman , N. E. , Rajan , S. , Verma , V. K. , Song , J. , Yuan , A. , Shanmughapriya , S. , Guo , Y. , Gao , E. , Koch , W. , Woodgett , J. R. , Madesh , M. , Kishore , R. , Lal , H. & Force , T. , Apr 15 2016resultingin fatal dilated cardiomyopathy Zhou , J. , Ahmad , F. , Parikh , S. , Hoffman , N. E. , Rajan , S. , Verma , V. K. , Song , J. , Yuan , A. , Shanmughapriya , S. , Guo , Y. , Gao , E. , Koch , W. , Woodgett , J. R. , Madesh , M. , Kishore , R. , Lal , H. & Force , T. , Apr 15 2016
in a loss of functional cardiac myocytesresultingin a loss of functional cardiac myocytes
in cell fusion , polyploidy , forex in india 2017resultingin cell fusion , polyploidy , forex in india 2017
normallycausednormally
cardiogenic shockcausingcardiogenic shock
endopolyploidy , giant cell formationprovokesendopolyploidy , giant cell formation
in remote desktop , polyploidy , or failure to identify cytokinesisresultingin remote desktop , polyploidy , or failure to identify cytokinesis
Many different cell fates(passive) could be caused byMany different cell fates
to apoptosis and senescence in TNBCleadsto apoptosis and senescence in TNBC
in delayed apoptosisresultingin delayed apoptosis
in apoptosis by downregulating CDC20 in glioma cellsresultsin apoptosis by downregulating CDC20 in glioma cells
in caspase dependent apoptosisresultin caspase dependent apoptosis
This man – made(passive) can be preventedThis man – made
from a combination of deficient cell - cycle checkpoints , in particular DNA structure checkpoints and the spindle assembly checkpoint , and cellular damage [ 1 ] Defective cell cycle checkpointmay resultfrom a combination of deficient cell - cycle checkpoints , in particular DNA structure checkpoints and the spindle assembly checkpoint , and cellular damage [ 1 ] Defective cell cycle checkpoint
stronglycontributesstrongly
in difference interval , timer , or gradient to develop cytokinesisresultingin difference interval , timer , or gradient to develop cytokinesis
in the ultimate development of fatal heart failureresultingin the ultimate development of fatal heart failure
survival of cells with extensively damaged chromosomes ( Figure 1preventingsurvival of cells with extensively damaged chromosomes ( Figure 1
from ATP depletion ( Edinger and Thompson , 2004resultingfrom ATP depletion ( Edinger and Thompson , 2004
to apoptosis followed by PARP and Laminleadsto apoptosis followed by PARP and Lamin
abnormal mitosis(passive) caused byabnormal mitosis
to p53-independent apoptosis or necrotic death.37 Lethal hematopoietic syndrome resulting from an acute p53 responseleadingto p53-independent apoptosis or necrotic death.37 Lethal hematopoietic syndrome resulting from an acute p53 response
starvation among wildlife , livestock , and even people in some parts of Kenyacausedstarvation among wildlife , livestock , and even people in some parts of Kenya
in the protagonist 's well - being Frenchresultsin the protagonist 's well - being French
lasting damage to the Weavecausedlasting damage to the Weave
to arrest in mitosis or , alternatively , to the formation of cells with multiple nuclei or a single giant nucleus ( 12 , 13leadsto arrest in mitosis or , alternatively , to the formation of cells with multiple nuclei or a single giant nucleus ( 12 , 13
to arrest in mitosis , or alternatively to the formation of cells with multiple nuclei or a single giant nucleus ( Gisselsson et al .leadsto arrest in mitosis , or alternatively to the formation of cells with multiple nuclei or a single giant nucleus ( Gisselsson et al .
our cancersmay have causedour cancers
in senescence , or irreversible cell cycle arrest precluding generation of genomic unstable cellsresultingin senescence , or irreversible cell cycle arrest precluding generation of genomic unstable cells